Shenfeng Qiu

3.1k total citations
57 papers, 2.3k citations indexed

About

Shenfeng Qiu is a scholar working on Cellular and Molecular Neuroscience, Molecular Biology and Genetics. According to data from OpenAlex, Shenfeng Qiu has authored 57 papers receiving a total of 2.3k indexed citations (citations by other indexed papers that have themselves been cited), including 30 papers in Cellular and Molecular Neuroscience, 23 papers in Molecular Biology and 17 papers in Genetics. Recurrent topics in Shenfeng Qiu's work include Neuroscience and Neuropharmacology Research (27 papers), Genetics and Neurodevelopmental Disorders (16 papers) and Neurogenesis and neuroplasticity mechanisms (16 papers). Shenfeng Qiu is often cited by papers focused on Neuroscience and Neuropharmacology Research (27 papers), Genetics and Neurodevelopmental Disorders (16 papers) and Neurogenesis and neuroplasticity mechanisms (16 papers). Shenfeng Qiu collaborates with scholars based in United States, China and Netherlands. Shenfeng Qiu's co-authors include Edwin J. Weeber, Pat Levitt, Kimberly Korwek, Zhongming Lu, Xiaokuang Ma, Giselind Adelmann, Robert E. Hammer, J. David Sweatt, Andre Durudas and Wei-Ping Li and has published in prestigious journals such as Journal of Biological Chemistry, Nature Communications and Neuron.

In The Last Decade

Shenfeng Qiu

56 papers receiving 2.3k citations

Peers

Shenfeng Qiu
Delia M. Talos United States
Daniel Vogt United States
Marlen Knobloch Switzerland
Steve C. Danzer United States
Cristina A. Ghiani United States
H. Jürgen Wenzel United States
Shenfeng Qiu
Citations per year, relative to Shenfeng Qiu Shenfeng Qiu (= 1×) peers Pablo Méndez

Countries citing papers authored by Shenfeng Qiu

Since Specialization
Citations

This map shows the geographic impact of Shenfeng Qiu's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Shenfeng Qiu with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Shenfeng Qiu more than expected).

Fields of papers citing papers by Shenfeng Qiu

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Shenfeng Qiu. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Shenfeng Qiu. The network helps show where Shenfeng Qiu may publish in the future.

Co-authorship network of co-authors of Shenfeng Qiu

This figure shows the co-authorship network connecting the top 25 collaborators of Shenfeng Qiu. A scholar is included among the top collaborators of Shenfeng Qiu based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Shenfeng Qiu. Shenfeng Qiu is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Wei, Jing, Xiaokuang Ma, Ross A. Johnson, et al.. (2024). SIRT1 Coordinates Transcriptional Regulation of Neural Activity and Modulates Depression-Like Behaviors in the Nucleus Accumbens. Biological Psychiatry. 96(6). 495–505. 13 indexed citations
2.
Liu, Bin, Dan Yi, Xiaokuang Ma, et al.. (2024). A Novel Animal Model for Pulmonary Hypertension: Lung Endothelial-Specific Deletion of Egln1 in Mice. PubMed. 1(2). 10007–10007.
3.
Saber, Maha, J. Bryce Ortiz, Xiaokuang Ma, et al.. (2021). Mice Born to Mothers with Gravida Traumatic Brain Injury Have Distorted Brain Circuitry and Altered Immune Responses. Journal of Neurotrauma. 38(20). 2862–2880. 8 indexed citations
4.
Xia, Baomei, Jing Wei, Xiaokuang Ma, et al.. (2021). Conditional knockout of MET receptor tyrosine kinase in cortical excitatory neurons leads to enhanced learning and memory in young adult mice but early cognitive decline in older adult mice. Neurobiology of Learning and Memory. 179. 107397–107397. 9 indexed citations
5.
Chen, Ke, Xiaokuang Ma, Jing Wei, et al.. (2020). Time-delimited signaling of MET receptor tyrosine kinase regulates cortical circuit development and critical period plasticity. Molecular Psychiatry. 26(8). 3723–3736. 12 indexed citations
6.
Ma, Zegang, Fenfei Gao, Ming Gao, et al.. (2019). Mechanisms of Cannabinoid CB2 Receptor-Mediated Reduction of Dopamine Neuronal Excitability in Mouse Ventral Tegmental Area. SSRN Electronic Journal. 3 indexed citations
7.
Gao, Fenfei, Ming Gao, Xiaokuang Ma, et al.. (2019). Mechanisms of cannabinoid CB2 receptor-mediated reduction of dopamine neuronal excitability in mouse ventral tegmental area. EBioMedicine. 42. 225–237. 44 indexed citations
8.
Huang, Guanqun, Shuting Chen, Xiaoxia Chen, et al.. (2019). Uncovering the Functional Link Between SHANK3 Deletions and Deficiency in Neurodevelopment Using iPSC-Derived Human Neurons. Frontiers in Neuroanatomy. 13. 23–23. 36 indexed citations
9.
Ma, Xiaokuang, et al.. (2018). Distinct Circuits for Recovery of Eye Dominance and Acuity in Murine Amblyopia. Current Biology. 28(12). 1914–1923.e5. 31 indexed citations
10.
Sun, Guozhu, Xiaokuang Ma, Shuangtao Li, et al.. (2017). Hippocampal synaptic and neural network deficits in young mice carrying the humanAPOE4gene. CNS Neuroscience & Therapeutics. 23(9). 748–758. 34 indexed citations
11.
Peng, Yun, Zhengqi Lu, Guohui Li, et al.. (2016). The autism-associated MET receptor tyrosine kinase engages early neuronal growth mechanism and controls glutamatergic circuits development in the forebrain. Molecular Psychiatry. 21(7). 925–935. 39 indexed citations
12.
Peng, Yun, Matthew J. Huentelman, Christopher J. Smith, & Shenfeng Qiu. (2013). MET Receptor Tyrosine Kinase as an Autism Genetic Risk Factor. International review of neurobiology. 113. 135–165. 36 indexed citations
13.
Qiu, Shenfeng, Shujun Luo, Oleg V. Evgrafov, et al.. (2012). Single-neuron RNA-Seq: technical feasibility and reproducibility. Frontiers in Genetics. 3. 124–124. 47 indexed citations
14.
Qiu, Shenfeng, Charles T. Anderson, Pat Levitt, & Gordon M. Shepherd. (2011). Circuit-Specific Intracortical Hyperconnectivity in Mice with Deletion of the Autism-Associated Met Receptor Tyrosine Kinase. Journal of Neuroscience. 31(15). 5855–5864. 93 indexed citations
15.
Aldinger, Kimberly A., Jasmine Plummer, Shenfeng Qiu, & Pat Levitt. (2011). SnapShot: Genetics of Autism. Neuron. 72(2). 418–418.e1. 25 indexed citations
16.
Qiu, Shenfeng, Danielle L. Champagne, Melinda Peters, et al.. (2010). Loss of Limbic System-Associated Membrane Protein Leads to Reduced Hippocampal Mineralocorticoid Receptor Expression, Impaired Synaptic Plasticity, and Spatial Memory Deficit. Biological Psychiatry. 68(2). 197–204. 43 indexed citations
17.
18.
Woerden, Geeske M. van, Mohammad Reza Hojjati, Richard M. Gustin, et al.. (2007). Rescue of neurological deficits in a mouse model for Angelman syndrome by reduction of αCaMKII inhibitory phosphorylation. Nature Neuroscience. 10(3). 280–282. 220 indexed citations
19.
Qiu, Shenfeng, et al.. (2006). Differential Reelin-Induced Enhancement of NMDA and AMPA Receptor Activity in the Adult Hippocampus. Journal of Neuroscience. 26(50). 12943–12955. 143 indexed citations
20.
Beffert, Uwe, Edwin J. Weeber, Andre Durudas, et al.. (2005). Modulation of Synaptic Plasticity and Memory by Reelin Involves Differential Splicing of the Lipoprotein Receptor Apoer2. Neuron. 47(4). 567–579. 368 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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