Shaunrick Stoll

436 total citations
11 papers, 315 citations indexed

About

Shaunrick Stoll is a scholar working on Cardiology and Cardiovascular Medicine, Physiology and Molecular Biology. According to data from OpenAlex, Shaunrick Stoll has authored 11 papers receiving a total of 315 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Cardiology and Cardiovascular Medicine, 6 papers in Physiology and 5 papers in Molecular Biology. Recurrent topics in Shaunrick Stoll's work include Endoplasmic Reticulum Stress and Disease (4 papers), Adipose Tissue and Metabolism (4 papers) and Cardiovascular Disease and Adiposity (3 papers). Shaunrick Stoll is often cited by papers focused on Endoplasmic Reticulum Stress and Disease (4 papers), Adipose Tissue and Metabolism (4 papers) and Cardiovascular Disease and Adiposity (3 papers). Shaunrick Stoll collaborates with scholars based in United States and China. Shaunrick Stoll's co-authors include Hongyu Qiu, Charles Wang, Ben Ma, Ning Zhou, Kevin D. Costa, Jia‐Jye Lee, Tristan T. Hays, Gangjian Qin, Jing Xi and Christiana Leimena and has published in prestigious journals such as Circulation Research, Scientific Reports and International Journal of Molecular Sciences.

In The Last Decade

Shaunrick Stoll

11 papers receiving 310 citations

Peers

Shaunrick Stoll

MB

CCM

PHYSI

CB

CR

Ben Ma United States

Brooke Henderson United States

Gang Cheng China

Yonglun Kong China

John C. Kostyak United States

Veronica Larcher Italy

Molly Enrick United States

Sandra J. Walchak United States

Martha Hinton Canada

I‐Fen Cheng Germany

Ben Ma United States

Shaunrick Stoll

150 ×0.9

MB

101 ×1.1

CCM

60 ×1.0

PHYSI

43 ×1.0

CB

28 ×0.8

CR

Citations per year, relative to Shaunrick Stoll Shaunrick Stoll (= 1×) peers Ben Ma

Countries citing papers authored by Shaunrick Stoll

Since Specialization

Citations

This map shows the geographic impact of Shaunrick Stoll's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Shaunrick Stoll with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Shaunrick Stoll more than expected).

Fields of papers citing papers by Shaunrick Stoll

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Shaunrick Stoll. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Shaunrick Stoll. The network helps show where Shaunrick Stoll may publish in the future.

Co-authorship network of co-authors of Shaunrick Stoll

This figure shows the co-authorship network connecting the top 25 collaborators of Shaunrick Stoll. A scholar is included among the top collaborators of Shaunrick Stoll based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Shaunrick Stoll. Shaunrick Stoll is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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11 of 11 papers shown

1.

Sun, Xiaonan, Ning Zhou, Ben Ma, et al.. (2021). Functional Inhibition of Valosin-Containing Protein Induces Cardiac Dilation and Dysfunction in a New Dominant-Negative Transgenic Mouse Model. Cells. 10(11). 2891–2891. 8 indexed citations

2.

Zhou, Ning, Xin Chen, Jing Xi, et al.. (2020). Genomic characterization reveals novel mechanisms underlying the valosin-containing protein-mediated cardiac protection against heart failure. Redox Biology. 36. 101662–101662. 13 indexed citations

3.

Zhou, Ning, Xin Chen, Jing Xi, et al.. (2020). Novel genomic targets of valosin-containing protein in protecting pathological cardiac hypertrophy. Scientific Reports. 10(1). 18098–18098. 8 indexed citations

4.

Stoll, Shaunrick, et al.. (2019). Abstract 882: The Valosin-containing Protein Resists Pathological Cardiac Calcium Overload via Inhibiting Mitochondrial Calcium Uptake. Circulation Research. 125(Suppl_1). 1 indexed citations

5.

Stoll, Shaunrick, Jing Xi, Ben Ma, et al.. (2019). The Valosin-Containing Protein Protects the Heart Against Pathological Ca2+ Overload by Modulating Ca2+ Uptake Proteins. Toxicological Sciences. 171(2). 473–484. 25 indexed citations

6.

Stoll, Shaunrick, Charles Wang, & Hongyu Qiu. (2018). DNA Methylation and Histone Modification in Hypertension. International Journal of Molecular Sciences. 19(4). 1174–1174. 94 indexed citations

7.

Hays, Tristan T., Ben Ma, Ning Zhou, et al.. (2018). Vascular smooth muscle cells direct extracellular dysregulation in aortic stiffening of hypertensive rats. Aging Cell. 17(3). e12748–e12748. 32 indexed citations

8.

Zhou, Ning, Jia‐Jye Lee, Shaunrick Stoll, et al.. (2017). Rho Kinase Regulates Aortic Vascular Smooth Muscle Cell Stiffness Via Actin/SRF/Myocardin in Hypertension. Cellular Physiology and Biochemistry. 44(2). 701–715. 43 indexed citations

9.

Lizano, Paulo, Eman Rashed, Shaunrick Stoll, et al.. (2017). The valosin-containing protein is a novel mediator of mitochondrial respiration and cell survival in the heart in vivo. Scientific Reports. 7(1). 46324–46324. 21 indexed citations

10.

Zhou, Ning, Ben Ma, Shaunrick Stoll, Tristan T. Hays, & Hongyu Qiu. (2017). The valosin‐containing protein is a novel repressor of cardiomyocyte hypertrophy induced by pressure overload. Aging Cell. 16(5). 1168–1179. 20 indexed citations

11.

Zhou, Ning, Jia‐Jye Lee, Shaunrick Stoll, et al.. (2016). Inhibition of SRF/myocardin reduces aortic stiffness by targeting vascular smooth muscle cell stiffening in hypertension. Cardiovascular Research. 113(2). 171–182. 50 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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