Shareefa Dalvie

4.8k citations
42 papers · 680 · h-index 15

Impact in

Papers in

    • Genetic Associations and Epidemiology 15
    • Genomics and Rare Diseases 3
    • Genetics and Neurodevelopmental Disorders 2
    • Child Abuse and Trauma 3

Shareefa Dalvie

41 papers receiving 671 citations

Peers

Shareefa Dalvie
Comparison fields: 5 of 100
  • Biological Psychiatry 32
  • Behavioral Neuroscience 43
  • Clinical Psychology 162
  • Genetics 148
  • Psychiatry and Mental health 68
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Citations per field
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Citations per year

Countries citing papers authored by Shareefa Dalvie

Since Specialization
Citations

This map shows the geographic impact of Shareefa Dalvie's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Shareefa Dalvie with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Shareefa Dalvie more than expected).

Fields of papers citing papers by Shareefa Dalvie

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Shareefa Dalvie. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Shareefa Dalvie. The network helps show where Shareefa Dalvie may publish in the future.

Co-authors

The 25 scholars most cited alongside Shareefa Dalvie, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Shareefa Dalvie Line = papers co-authored together Shareefa Dalvie links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 42 papers — load more, or switch the sort, to bring in the rest.

#Work
1 2021120
2 201975
3 201963
4 201759
5 201434
6 201734
7 201834
8 201826
9 201825
10 201024
11 201816
12 201616
13 202115
14 202014
15 201814
16 201413
17 202112
18 201911
19 20238
20 20228

About Shareefa Dalvie

Shareefa Dalvie is a scholar working on Genetics, Clinical Psychology, Experimental and Cognitive Psychology, Molecular Biology and Epidemiology, having authored 42 papers that have together received 680 indexed citations. Recurring topics across this work include Genetic Associations and Epidemiology (15 papers), Cognitive Abilities and Testing (4 papers), Child Abuse and Trauma (3 papers), Genomics and Rare Diseases (3 papers), Alcohol Consumption and Health Effects (3 papers), Stress Responses and Cortisol (3 papers), Genetics and Neurodevelopmental Disorders (2 papers) and Substance Abuse Treatment and Outcomes (2 papers). The work is most often cited by research in Biological Psychiatry (32 citations), Behavioral Neuroscience (43 citations), Clinical Psychology (162 citations), Genetics (148 citations) and Psychiatry and Mental health (68 citations). Shareefa Dalvie has collaborated with scholars based in South Africa, United States and Norway. Frequent co-authors include Dan J. Stein, Raj Ramesar, Samantha J. Brooks, Mary S. Mufford, Nynke A. Groenewold, Caroline M. Nievergelt, Alicia R. Martin, Jantina de Vries, Emile R. Chimusa and Anne Uhlmann. Their work appears in journals such as Biological Psychiatry, Journal of Affective Disorders, European Neuropsychopharmacology, The World Journal of Biological Psychiatry and Frontiers in Genetics.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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