Shane R. Solst

432 total citations
12 papers, 337 citations indexed

About

Shane R. Solst is a scholar working on Molecular Biology, Biochemistry and Cancer Research. According to data from OpenAlex, Shane R. Solst has authored 12 papers receiving a total of 337 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Molecular Biology, 3 papers in Biochemistry and 3 papers in Cancer Research. Recurrent topics in Shane R. Solst's work include Cancer, Hypoxia, and Metabolism (3 papers), Redox biology and oxidative stress (3 papers) and Glutathione Transferases and Polymorphisms (2 papers). Shane R. Solst is often cited by papers focused on Cancer, Hypoxia, and Metabolism (3 papers), Redox biology and oxidative stress (3 papers) and Glutathione Transferases and Polymorphisms (2 papers). Shane R. Solst collaborates with scholars based in United States, Italy and Germany. Shane R. Solst's co-authors include Douglas R. Spitz, Samuel N. Rodman, Cristina M. Furdui, Ravi Singh, George L. Donati, Melissa A. Fath, Bryan G. Allen, Xiangming Guan, Yueming Zhu and Arpit Sharma and has published in prestigious journals such as Genetics, Free Radical Biology and Medicine and Science Advances.

In The Last Decade

Shane R. Solst

11 papers receiving 334 citations

Peers

Shane R. Solst
Karthikeyan Subburayan United Arab Emirates
Laura M. Bystrom United States
Ok Kim South Korea
Katherine M. Weh United States
William Wolter United States
Shane R. Solst
Citations per year, relative to Shane R. Solst Shane R. Solst (= 1×) peers Shuaijun Lu

Countries citing papers authored by Shane R. Solst

Since Specialization
Citations

This map shows the geographic impact of Shane R. Solst's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Shane R. Solst with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Shane R. Solst more than expected).

Fields of papers citing papers by Shane R. Solst

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Shane R. Solst. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Shane R. Solst. The network helps show where Shane R. Solst may publish in the future.

Co-authorship network of co-authors of Shane R. Solst

This figure shows the co-authorship network connecting the top 25 collaborators of Shane R. Solst. A scholar is included among the top collaborators of Shane R. Solst based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Shane R. Solst. Shane R. Solst is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

12 of 12 papers shown
1.
Borcherding, Nicholas, Adam J. Rauckhorst, Wade R. Gutierrez, et al.. (2025). Somatic CRISPR tumorigenesis and multiomic analysis reveal a pentose phosphate pathway disruption vulnerability in MPNSTs. Science Advances. 11(33). eadu2906–eadu2906.
2.
Pulliam, Casey, Melissa A. Fath, Susan T. Johnson, et al.. (2025). Pharmacological ascorbate combined with rucosopasem selectively radio-chemo-sensitizes NSCLC via generation of H2O2. Redox Biology. 80. 103505–103505. 2 indexed citations
3.
Liu, Dijie, Casey Pulliam, Brian Wels, et al.. (2024). Auranofin inhibition of thioredoxin reductase sensitizes lung neuroendocrine tumor cells (NETs) and small cell lung cancer (SCLC) cells to sorafenib as well as inhibiting SCLC xenograft growth. Cancer Biology & Therapy. 25(1). 2382524–2382524. 6 indexed citations
4.
Solst, Shane R., Lisa B. Fiore, Ann Tomanek‐Chalkley, et al.. (2024). Additive Effects of Cu-ATSM and Radiation on Survival of Diffuse Intrinsic Pontine Glioma Cells. Radiation Research. 203(1). 10–17. 1 indexed citations
5.
Solst, Shane R., et al.. (2023). Rapid Peroxide Removal Limits the Radiosensitization of Diffuse Intrinsic Pontine Glioma (DIPG) Cells by Pharmacologic Ascorbate. Radiation Research. 200(5). 456–461. 1 indexed citations
6.
Solst, Shane R., et al.. (2021). The mechanism of cell death induced by silver nanoparticles is distinct from silver cations. Particle and Fibre Toxicology. 18(1). 37–37. 108 indexed citations
7.
Li, Mengshi, Jeffrey M. Stolwijk, Samuel N. Rodman, et al.. (2020). Disulfiram causes selective hypoxic cancer cell toxicity and radio-chemo-sensitization via redox cycling of copper. Free Radical Biology and Medicine. 150. 1–11. 36 indexed citations
8.
Jiménez‐García, Brian, et al.. (2019). How Boundaries Form: Linked Nonautonomous Feedback Loops Regulate Pattern Formation in Yeast Colonies. Genetics. 213(4). 1373–1386. 1 indexed citations
9.
Sheldon, Ryan D., Adam J. Rauckhorst, Shane R. Solst, et al.. (2019). Disrupting Mitochondrial Pyruvate Uptake Directs Glutamine into the TCA Cycle away from Glutathione Synthesis and Impairs Hepatocellular Tumorigenesis. Cell Reports. 28(10). 2608–2619.e6. 61 indexed citations
11.
Solst, Shane R., Samuel N. Rodman, Melissa A. Fath, Eric B. Taylor, & Douglas R. Spitz. (2017). Inhibition of Mitochondrial Pyruvate Transport Selectively Sensitizes Cancer Cells to Metabolic Oxidative Stress. Free Radical Biology and Medicine. 112. 102–102. 1 indexed citations
12.
Rodman, Samuel N., Yueming Zhu, Shane R. Solst, et al.. (2016). Enhancement of Radiation Response in Breast Cancer Stem Cells by Inhibition of Thioredoxin- and Glutathione-Dependent Metabolism. Radiation Research. 186(4). 385–385. 83 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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