Sergiy V. Korol

870 total citations
27 papers, 586 citations indexed

About

Sergiy V. Korol is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Genetics. According to data from OpenAlex, Sergiy V. Korol has authored 27 papers receiving a total of 586 indexed citations (citations by other indexed papers that have themselves been cited), including 14 papers in Molecular Biology, 13 papers in Cellular and Molecular Neuroscience and 7 papers in Genetics. Recurrent topics in Sergiy V. Korol's work include Neuroscience and Neuropharmacology Research (12 papers), Receptor Mechanisms and Signaling (5 papers) and Neuroinflammation and Neurodegeneration Mechanisms (4 papers). Sergiy V. Korol is often cited by papers focused on Neuroscience and Neuropharmacology Research (12 papers), Receptor Mechanisms and Signaling (5 papers) and Neuroinflammation and Neurodegeneration Mechanisms (4 papers). Sergiy V. Korol collaborates with scholars based in Sweden, Ukraine and France. Sergiy V. Korol's co-authors include Zhe Jin, Bryndis Birnir, Amol K. Bhandage, Daniel Espes, Masood Kamali‐Moghaddam, Qiujin Shen, Per‐Ola Carlsson, Qiaolin Deng, Yu Pei and Lisa Ekselius and has published in prestigious journals such as PLoS ONE, Diabetes and International Journal of Molecular Sciences.

In The Last Decade

Sergiy V. Korol

26 papers receiving 575 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Sergiy V. Korol Sweden 15 181 178 116 94 92 27 586
Jin‐Xia Zhu China 16 241 1.3× 116 0.7× 74 0.6× 151 1.6× 42 0.5× 42 713
Bogna M. Ignatowska‐Jankowska United States 18 254 1.4× 513 2.9× 141 1.2× 97 1.0× 33 0.4× 37 1.1k
Jan Detka Poland 13 177 1.0× 103 0.6× 120 1.0× 31 0.3× 32 0.3× 24 585
Jennifer O’Connell United States 13 265 1.5× 95 0.5× 226 1.9× 96 1.0× 36 0.4× 23 752
Deyin Lu United States 15 177 1.0× 91 0.5× 108 0.9× 42 0.4× 74 0.8× 18 558
Kim Eerola Finland 14 135 0.7× 136 0.8× 159 1.4× 56 0.6× 28 0.3× 21 599
Jordi Serrats United States 15 271 1.5× 297 1.7× 120 1.0× 29 0.3× 31 0.3× 27 1.0k
José Marques-Lopes United States 14 300 1.7× 126 0.7× 182 1.6× 28 0.3× 39 0.4× 20 684
Míriam Hernangómez Spain 16 193 1.1× 306 1.7× 123 1.1× 69 0.7× 22 0.2× 18 1.1k

Countries citing papers authored by Sergiy V. Korol

Since Specialization
Citations

This map shows the geographic impact of Sergiy V. Korol's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Sergiy V. Korol with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Sergiy V. Korol more than expected).

Fields of papers citing papers by Sergiy V. Korol

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Sergiy V. Korol. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Sergiy V. Korol. The network helps show where Sergiy V. Korol may publish in the future.

Co-authorship network of co-authors of Sergiy V. Korol

This figure shows the co-authorship network connecting the top 25 collaborators of Sergiy V. Korol. A scholar is included among the top collaborators of Sergiy V. Korol based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Sergiy V. Korol. Sergiy V. Korol is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
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Jin, Zhe, Amol K. Bhandage, Sergiy V. Korol, et al.. (2024). GABA-mediated inhibition of human CD4+ T cell functions is enhanced by insulin but impaired by high glucose levels. EBioMedicine. 105. 105217–105217. 7 indexed citations
4.
Jin, Zhe & Sergiy V. Korol. (2023). GABA signalling in human pancreatic islets. Frontiers in Endocrinology. 14. 1059110–1059110. 11 indexed citations
5.
Schuster, Jens, Joakim Klar, Ambrin Fatima, et al.. (2022). ZEB2 haploinsufficient Mowat-Wilson syndrome induced pluripotent stem cells show disrupted GABAergic transcriptional regulation and function. Frontiers in Molecular Neuroscience. 15. 988993–988993. 1 indexed citations
6.
Bhandage, Amol K., Janet L. Cunningham, Zhe Jin, et al.. (2019). Depression, GABA, and Age Correlate with Plasma Levels of Inflammatory Markers. International Journal of Molecular Sciences. 20(24). 6172–6172. 19 indexed citations
7.
Korol, Sergiy V., et al.. (2019). Interferon-γ potentiates GABAA receptor-mediated inhibitory currents in rat hippocampal CA1 pyramidal neurons. Journal of Neuroimmunology. 337. 577050–577050. 34 indexed citations
8.
Schuster, Jens, Joakim Klar, Zhe Jin, et al.. (2019). Transcriptomes of Dravet syndrome iPSC derived GABAergic cells reveal dysregulated pathways for chromatin remodeling and neurodevelopment. Neurobiology of Disease. 132. 104583–104583. 33 indexed citations
9.
Moslem, Mohsen, Julien Bryois, Harriet Rönnholm, et al.. (2019). Single cell analysis of autism patient with bi-allelic NRXN1-alpha deletion reveals skewed fate choice in neural progenitors and impaired neuronal functionality. Experimental Cell Research. 383(1). 111469–111469. 38 indexed citations
10.
Bhandage, Amol K., Zhe Jin, Sergiy V. Korol, et al.. (2018). GABA Regulates Release of Inflammatory Cytokines From Peripheral Blood Mononuclear Cells and CD4+ T Cells and Is Immunosuppressive in Type 1 Diabetes. EBioMedicine. 30. 283–294. 121 indexed citations
11.
Korol, Sergiy V., et al.. (2018). Insulin enhances GABAA receptor-mediated inhibitory currents in rat central amygdala neurons. Neuroscience Letters. 671. 76–81. 16 indexed citations
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Bhandage, Amol K., Zhe Jin, Sergiy V. Korol, et al.. (2018). Expression of calcium release-activated and voltage-gated calcium channels genes in peripheral blood mononuclear cells is altered in pregnancy and in type 1 diabetes. PLoS ONE. 13(12). e0208981–e0208981. 7 indexed citations
13.
Korol, Sergiy V., Zhe Jin, Jin Yang, et al.. (2018). Functional Characterization of Native, High-Affinity GABAA Receptors in Human Pancreatic β Cells. EBioMedicine. 30. 273–282. 51 indexed citations
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Korol, Sergiy V., et al.. (2017). Liraglutide modulates GABAergic signaling in rat hippocampal CA3 pyramidal neurons predominantly by presynaptic mechanism. BMC Pharmacology and Toxicology. 18(1). 83–83. 17 indexed citations
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Korol, Sergiy V., Zhe Jin, & Bryndis Birnir. (2015). The GLP-1 Receptor Agonist Exendin-4 and Diazepam Differentially Regulate GABAA Receptor-Mediated Tonic Currents in Rat Hippocampal CA3 Pyramidal Neurons. PLoS ONE. 10(4). e0124765–e0124765. 23 indexed citations
18.
Jin, Zhe, Amol K. Bhandage, Sergiy V. Korol, et al.. (2014). Etomidate, propofol and diazepam potentiate GABA-evoked GABAA currents in a cell line derived from human glioblastoma. European Journal of Pharmacology. 748. 101–107. 18 indexed citations
19.
Yang, Jin, Sergiy V. Korol, Zhe Jin, Sebastian Barg, & Bryndis Birnir. (2013). In Intact Islets Interstitial GABA Activates GABAA Receptors That Generate Tonic Currents in α-Cells. PLoS ONE. 8(6). e67228–e67228. 19 indexed citations
20.
Korol, Sergiy V., et al.. (2010). Alteration of calcium signaling as one of the mechanisms of Alzheimer's disease and diabetic polyneuropathy. Fìzìologìčnij žurnal. 56(4). 130–138. 3 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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