José Marques-Lopes

896 total citations
20 papers, 684 citations indexed

About

José Marques-Lopes is a scholar working on Social Psychology, Physiology and Cellular and Molecular Neuroscience. According to data from OpenAlex, José Marques-Lopes has authored 20 papers receiving a total of 684 indexed citations (citations by other indexed papers that have themselves been cited), including 9 papers in Social Psychology, 9 papers in Physiology and 8 papers in Cellular and Molecular Neuroscience. Recurrent topics in José Marques-Lopes's work include Neuroendocrine regulation and behavior (9 papers), Neuropeptides and Animal Physiology (8 papers) and Stress Responses and Cortisol (5 papers). José Marques-Lopes is often cited by papers focused on Neuroendocrine regulation and behavior (9 papers), Neuropeptides and Animal Physiology (8 papers) and Stress Responses and Cortisol (5 papers). José Marques-Lopes collaborates with scholars based in United States, Portugal and Canada. José Marques-Lopes's co-authors include Wing‐Yiu Choy, Rachel E. Lackie, Marco A. M. Prado, Valeriy G. Ostapchenko, Teresa A. Milner, Martin L. Duennwald, Andrzej Maciejewski, Vânia F. Prado, Costantino Iadecola and Virginia M. Pickel and has published in prestigious journals such as Journal of Neuroscience, The Journal of Comparative Neurology and Brain Research.

In The Last Decade

José Marques-Lopes

20 papers receiving 682 citations

Peers

Countries citing papers authored by José Marques-Lopes

Since Specialization

Citations

This map shows the geographic impact of José Marques-Lopes's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by José Marques-Lopes with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites José Marques-Lopes more than expected).

Fields of papers citing papers by José Marques-Lopes

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by José Marques-Lopes. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by José Marques-Lopes. The network helps show where José Marques-Lopes may publish in the future.

Co-authorship network of co-authors of José Marques-Lopes

This figure shows the co-authorship network connecting the top 25 collaborators of José Marques-Lopes. A scholar is included among the top collaborators of José Marques-Lopes based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with José Marques-Lopes. José Marques-Lopes is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

#	Title	Journal	Authors	Indexed citations
1	Estrogen Receptor Beta Agonist Influences Presynaptic NMDA Receptor Distribution in the Paraventricular Hypothalamic Nucleus Following Hypertension in a Mouse Model of Perimenopause	Biology	G. Sommer, José Marques-Lopes et al.	1
2	Angiotensin II differentially affects hippocampal glial inflammatory markers in young adult male and female mice	Learning & Memory	Teresa A. Milner, José Marques-Lopes et al.	4
3	Tumor Necrosis Factor α Receptor Type 1 Activation in the Hypothalamic Paraventricular Nucleus Contributes to Glutamate Signaling and Angiotensin II-Dependent Hypertension	Journal of Neuroscience	José Marques-Lopes, Teresa A. Milner et al.	24
4	Increased levels of Stress-inducible phosphoprotein-1 accelerates amyloid-β deposition in a mouse model of Alzheimer’s disease	Acta Neuropathologica Communications	Rachel E. Lackie, José Marques-Lopes et al.	20
5	Sex and age differentially affect GABAergic neurons in the mouse prefrontal cortex and hippocampus following chronic intermittent hypoxia	Experimental Neurology	Teresa A. Milner, Virginia M. Pickel et al.	13
6	Plasma Membrane Affiliated AMPA GluA1 in Estrogen Receptor β-containing Paraventricular Hypothalamic Neurons Increases Following Hypertension in a Mouse Model of Post-menopause	Neuroscience	José Marques-Lopes, Tracey A. Van Kempen et al.	7
7	The Hsp70/Hsp90 Chaperone Machinery in Neurodegenerative Diseases	Frontiers in Neuroscience	Rachel E. Lackie, Andrzej Maciejewski et al.	261
8	Redistribution of NMDA Receptors in Estrogen-Receptor-β-Containing Paraventricular Hypothalamic Neurons following Slow-Pressor Angiotensin II Hypertension in Female Mice with Accelerated Ovarian Failure	Neuroendocrinology	José Marques-Lopes, Tracey A. Van Kempen et al.	24
9	Sex differences in NMDA GluN1 plasticity in rostral ventrolateral medulla neurons containing corticotropin-releasing factor type 1 receptor following slow-pressor angiotensin II hypertension	Neuroscience	Tracey A. Van Kempen, José Marques-Lopes et al.	19
10	Alterations in the subcellular distribution of NADPH oxidase p47^phoxin hypothalamic paraventricular neurons following slow‐pressor angiotensin II hypertension in female mice with accelerated ovarian failure	The Journal of Comparative Neurology	Tracey A. Van Kempen, Ankita Narayan et al.	11
11	Female protection from slow‐pressor effects of angiotensin II involves prevention of ROS production independent of NMDA receptor trafficking in hypothalamic neurons expressing angiotensin 1A receptors	Synapse	José Marques-Lopes, Tracey A. Van Kempen et al.	26
12	Slow‐pressor angiotensin II hypertension and concomitant dendritic NMDA receptor trafficking in estrogen receptor β–containing neurons of the mouse hypothalamic paraventricular nucleus are sex and age dependent	The Journal of Comparative Neurology	José Marques-Lopes, Elizabeth M. Waters et al.	33
13	Membrane Trafficking of NADPH Oxidase p47^phoxin Paraventricular Hypothalamic Neurons Parallels Local Free Radical Production in Angiotensin II Slow-Pressor Hypertension	Journal of Neuroscience	Christal G. Coleman, Gang Wang et al.	35
14	Angiotensin II slow-pressor hypertension enhances NMDA currents and NOX2-dependent superoxide production in hypothalamic paraventricular neurons	American Journal of Physiology-Regulatory, Integrative and Comparative Physiology	Gang Wang, Christal G. Coleman et al.	50
15	Distribution of angiotensin type 1a receptor-containing cells in the brains of bacterial artificial chromosome transgenic mice	Neuroscience	Elizabeth M. Waters, Robert C. Speth et al.	54
16	Decrease in the expression of N‐methyl‐D‐aspartate receptors in the nucleus tractus solitarii induces antinociception and increases blood pressure	Journal of Neuroscience Research	José Marques-Lopes, Isabel Martins et al.	4
17	Does Chronic Pain Alter the Normal Interaction Between Cardiovascular and Pain Regulatory Systems? Pain Modulation in the Hypertensive-Monoarthritic Rat	Journal of Pain	Dora Pinho, Manuela Morato et al.	11
18	The hyperalgesic effects induced by the injection of angiotensin II into the caudal ventrolateral medulla are mediated by the pontine A5 noradrenergic cell group	Brain Research	José Marques-Lopes, Dora Pinho et al.	18
19	Role of superoxide and hydrogen peroxide in hypertension induced by an antagonist of adenosine receptors	European Journal of Pharmacology	Teresa Sousa, Dora Pinho et al.	39
20	Microinjection of angiotensin II in the caudal ventrolateral medulla induces hyperalgesia	Neuroscience	José Marques-Lopes, Marta Pinto et al.	30

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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