Serdar Bedii Omay

752 citations
41 papers · 611 indexed · h-index 17
  • Hematology top 10%
    • Acute Myeloid Leukemia Research 3
    • Chronic Myeloid Leukemia Treatments 3
    • Cytokine Signaling Pathways and Interactions 3
    • Retinoids in leukemia and cellular processes 4
    • Ubiquitin and proteasome pathways 4
    • Protein Kinase Regulation and GTPase Signaling 3
    • Histone Deacetylase Inhibitors Research 3
    • Protein Tyrosine Phosphatases 3

Serdar Bedii Omay

39 papers receiving 594 citations

Peers

Serdar Bedii Omay
Comparison fields: 5 of 91
  • Hematology 149
  • Genetics 55
  • Oncology 138
  • Toxicology 17
  • Biochemistry 21
Replace Lisa M. Chelstrom with:
Lisa M. Chelstrom United States
Ying Di United Kingdom
Ariane de Agostini Switzerland
Yoshiko Hoshikawa Japan
Timo Siitonen Finland
J LEBLANC Canada
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H Brodthagen Denmark
Ina Nepstad Norway
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Citations per field
00.5×8.3×
Lisa M. Chelstrom · 1×
Citations per year

Countries citing papers authored by Serdar Bedii Omay

Since Specialization
Citations

This map shows the geographic impact of Serdar Bedii Omay's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Serdar Bedii Omay with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Serdar Bedii Omay more than expected).

Fields of papers citing papers by Serdar Bedii Omay

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Serdar Bedii Omay. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Serdar Bedii Omay. The network helps show where Serdar Bedii Omay may publish in the future.

Co-authorship network

The 25 scholars most cited alongside Serdar Bedii Omay, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Serdar Bedii Omay Line = papers co-authored together Serdar Bedii Omay links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown
#Work
1 20250
2 20250
3 20131
4 20117
5 200816
6 200866
7 200831
8 200717
9 200715
10 200535
11 200524
12 200424
13 200345
14 200221
15 20025
16 200247
17 20023
18 199511
19 19943
20 199322

About Serdar Bedii Omay

Serdar Bedii Omay is a scholar working on Hematology, Internal Medicine and Hepatology, having authored 41 papers that have together received 611 indexed citations. Recurring topics across this work include Retinoids in leukemia and cellular processes (4 papers), Ubiquitin and proteasome pathways (4 papers), Protein Kinase Regulation and GTPase Signaling (3 papers), Histone Deacetylase Inhibitors Research (3 papers), Acute Myeloid Leukemia Research (3 papers), Protein Tyrosine Phosphatases (3 papers), Chronic Myeloid Leukemia Treatments (3 papers) and Cytokine Signaling Pathways and Interactions (3 papers). The work is most often cited by research in Hematology (149 citations), Genetics (55 citations) and Oncology (138 citations). Serdar Bedii Omay has collaborated with scholars based in Türkiye, Japan and France. Frequent co-authors include Güray Saydam, Mehmet Sönmez, Ercüment Ovalı, Hikmet Hakan Aydın, Ender Terzıoğlu, Mustafa Yılmaz, Fahri Şahin, Elif Akdoğan, Rüçhan Uslu and Gülperi Öktem.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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