Senping Cheng

943 total citations
16 papers, 768 citations indexed

About

Senping Cheng is a scholar working on Molecular Biology, Health, Toxicology and Mutagenesis and Pharmacology. According to data from OpenAlex, Senping Cheng has authored 16 papers receiving a total of 768 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Molecular Biology, 5 papers in Health, Toxicology and Mutagenesis and 3 papers in Pharmacology. Recurrent topics in Senping Cheng's work include Cell death mechanisms and regulation (3 papers), Pesticide Exposure and Toxicity (3 papers) and Effects and risks of endocrine disrupting chemicals (2 papers). Senping Cheng is often cited by papers focused on Cell death mechanisms and regulation (3 papers), Pesticide Exposure and Toxicity (3 papers) and Effects and risks of endocrine disrupting chemicals (2 papers). Senping Cheng collaborates with scholars based in China, United States and South Korea. Senping Cheng's co-authors include Amit Budhraja, Xianglin Shi, Zhuo Zhang, Young‐Ok Son, Ning Gao, Songze Ding, Jiayin Liu, Yankai Xia, Jia Luo and Shoulin Wang and has published in prestigious journals such as Clinical Cancer Research, British Journal of Pharmacology and Annals of the Rheumatic Diseases.

In The Last Decade

Senping Cheng

15 papers receiving 752 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Senping Cheng China	12	365	168	127	104	100	16	768
Alaa E. El‐Sisi Egypt	17	312 0.9×	100 0.6×	56 0.4×	107 1.0×	132 1.3×	58	958
Shikha Sharma India	17	236 0.6×	138 0.8×	104 0.8×	77 0.7×	32 0.3×	44	837
Ghanshyam Upadhyay United States	16	395 1.1×	77 0.5×	233 1.8×	135 1.3×	85 0.8×	23	1.1k
Kai-Lee Wang Taiwan	21	453 1.2×	66 0.4×	90 0.7×	97 0.9×	47 0.5×	49	1.1k
Dominique Ziech United States	8	438 1.2×	94 0.6×	66 0.5×	125 1.2×	46 0.5×	8	838
Seon Yeong Ji South Korea	18	392 1.1×	60 0.4×	67 0.5×	81 0.8×	82 0.8×	46	783
Paul Hanlon United States	17	405 1.1×	95 0.6×	127 1.0×	72 0.7×	64 0.6×	30	949
Keita Kanki Japan	19	535 1.5×	97 0.6×	86 0.7×	255 2.5×	101 1.0×	50	994
Sheau‐Fung Thai United States	20	389 1.1×	170 1.0×	103 0.8×	197 1.9×	39 0.4×	30	996
Brooke G. Pantazides United States	13	204 0.6×	127 0.8×	191 1.5×	66 0.6×	51 0.5×	20	523

Countries citing papers authored by Senping Cheng

Since Specialization

Citations

This map shows the geographic impact of Senping Cheng's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Senping Cheng with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Senping Cheng more than expected).

Fields of papers citing papers by Senping Cheng

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Senping Cheng. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Senping Cheng. The network helps show where Senping Cheng may publish in the future.

Co-authorship network of co-authors of Senping Cheng

This figure shows the co-authorship network connecting the top 25 collaborators of Senping Cheng. A scholar is included among the top collaborators of Senping Cheng based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Senping Cheng. Senping Cheng is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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16 of 16 papers shown

Zhou, Ying, Meiju Ji, Libo Wang, et al.. (2024). POS1361 A NOVEL ORAL 3D-PRINTED DELAYED- AND EXTENDED-RELEASE TOFACITINIB (T19) FOR THE TREATMENT OF RHEUMATOID ARTHRITIS AND RELATED INFLAMMATORY DISEASES. Annals of the Rheumatic Diseases. 83. 592–592.

Budhraja, Amit, Ning Gao, Zhuo Zhang, et al.. (2011). Apigenin Induces Apoptosis in Human Leukemia Cells and Exhibits Anti-Leukemic Activity In Vivo. Molecular Cancer Therapeutics. 11(1). 132–142. 82 indexed citations

Gao, Ning, et al.. (2011). Phenethyl isothiocyanate exhibits antileukemic activity in vitro and in vivo by inactivation of Akt and activation of JNK pathways. Cell Death and Disease. 2(4). e140–e140. 86 indexed citations

Gao, Ning, Senping Cheng, Amit Budhraja, et al.. (2011). Ursolic acid induces apoptosis in human leukaemia cells and exhibits anti‐leukaemic activity in nude mice through the PKB pathway. British Journal of Pharmacology. 165(6). 1813–1826. 51 indexed citations

Son, Young‐Ok, Xianwei Wang, John Andrew Hitron, et al.. (2011). Cadmium induces autophagy through ROS-dependent activation of the LKB1–AMPK signaling in skin epidermal cells. Toxicology and Applied Pharmacology. 255(3). 287–296. 116 indexed citations

Zhang, Zhuo, Wenqi Li, Senping Cheng, et al.. (2011). Nickel-induced down-regulation of ΔNp63 and its role in the proliferation of keratinocytes. Toxicology and Applied Pharmacology. 253(3). 235–243. 5 indexed citations

Gao, Ning, Amit Budhraja, Senping Cheng, et al.. (2011). Interruption of the MEK/ERK signaling cascade promotes dihydroartemisinin-induced apoptosis in vitro and in vivo. APOPTOSIS. 16(5). 511–523. 56 indexed citations

Zhang, Zhuo, Xin Wang, Senping Cheng, et al.. (2011). Reactive oxygen species mediate arsenic induced cell transformation and tumorigenesis through Wnt/β-catenin pathway in human colorectal adenocarcinoma DLD1 cells. Toxicology and Applied Pharmacology. 256(2). 114–121. 53 indexed citations

Cheng, Senping, Ning Gao, Zhuo Zhang, et al.. (2010). Quercetin Induces Tumor-Selective Apoptosis through Downregulation of Mcl-1 and Activation of Bax. Clinical Cancer Research. 16(23). 5679–5691. 78 indexed citations

10.

Son, Young‐Ok, John Andrew Hitron, Senping Cheng, et al.. (2010). The Dual Roles of c-Jun NH2-Terminal Kinase Signaling in Cr(VI)-Induced Apoptosis in JB6 Cells. Toxicological Sciences. 119(2). 335–345. 11 indexed citations

11.

Gao, Ning, Amit Budhraja, Senping Cheng, et al.. (2008). Induction of Apoptosis in Human Leukemia Cells by Grape Seed Extract Occurs via Activation of c-Jun NH2-Terminal Kinase. Clinical Cancer Research. 15(1). 140–149. 47 indexed citations

12.

Xia, Hong, Jianhua Qu, Jianfeng Chen, et al.. (2007). Effects of trichlorfon on progesterone production in cultured human granulosa–lutein cells. Toxicology in Vitro. 21(5). 912–918. 7 indexed citations

13.

Cheng, Senping, Jianfeng Chen, Yang Qiu, et al.. (2006). Carbaryl inhibits basal and FSH-induced progesterone biosynthesis of primary human granulosa-lutein cells. Toxicology. 220(1). 37–45. 16 indexed citations

14.

Cui, Lunbiao, Guidong Dai, Jianfeng Chen, et al.. (2006). Elevated prostaglandin E2 level via cPLA2–COX-2–mPGES-1 pathway involved in bladder carcinogenesis induced by terephthalic acid-calculi in Wistar rats. Prostaglandins Leukotrienes and Essential Fatty Acids. 74(5). 309–315. 19 indexed citations

15.

Xia, Yankai, Senping Cheng, Qian Bian, et al.. (2004). Genotoxic Effects on Spermatozoa of Carbaryl-Exposed Workers. Toxicological Sciences. 85(1). 615–623. 52 indexed citations

16.

Xia, Yankai, Qian Bian, Lichun Xu, et al.. (2004). Genotoxic effects on human spermatozoa among pesticide factory workers exposed to fenvalerate. Toxicology. 203(1-3). 49–60. 89 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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