Sendoa Tajada

1.1k total citations · 1 hit paper
15 papers, 845 citations indexed

About

Sendoa Tajada is a scholar working on Molecular Biology, Sensory Systems and Cardiology and Cardiovascular Medicine. According to data from OpenAlex, Sendoa Tajada has authored 15 papers receiving a total of 845 indexed citations (citations by other indexed papers that have themselves been cited), including 13 papers in Molecular Biology, 8 papers in Sensory Systems and 7 papers in Cardiology and Cardiovascular Medicine. Recurrent topics in Sendoa Tajada's work include Ion channel regulation and function (11 papers), Ion Channels and Receptors (8 papers) and Cardiac electrophysiology and arrhythmias (6 papers). Sendoa Tajada is often cited by papers focused on Ion channel regulation and function (11 papers), Ion Channels and Receptors (8 papers) and Cardiac electrophysiology and arrhythmias (6 papers). Sendoa Tajada collaborates with scholars based in Spain, United States and Cuba. Sendoa Tajada's co-authors include José R. López‐López, M. Teresa Pérez‐García, Carlos Villalobos, Luis F. Santana, Claudia M. Moreno, Thomas Voets, Pilar Cidad, Karel Talavera, Yeranddy A. Alpízar and Rose E. Dixon and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Nature Communications and The Journal of Physiology.

In The Last Decade

Sendoa Tajada

15 papers receiving 840 citations

Hit Papers

TRPA1 channels mediate acute neurogenic inflammation and ... 2014 2026 2018 2022 2014 100 200 300

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Sendoa Tajada Spain 12 446 258 218 189 170 15 845
Ken‐ichi Otsuguro Japan 18 328 0.7× 322 1.2× 277 1.3× 235 1.2× 141 0.8× 60 1.1k
Hayato Matsuyama Japan 18 367 0.8× 194 0.8× 216 1.0× 149 0.8× 63 0.4× 49 1.1k
Gábor Pozsgai Hungary 20 233 0.5× 476 1.8× 407 1.9× 377 2.0× 52 0.3× 37 1.2k
Florentina Pluteanu Germany 17 367 0.8× 164 0.6× 179 0.8× 162 0.9× 241 1.4× 27 775
Carmen A. Ufret-Vincenty United States 14 554 1.2× 542 2.1× 336 1.5× 252 1.3× 127 0.7× 16 1.1k
Diego Varela Chile 21 825 1.8× 326 1.3× 385 1.8× 213 1.1× 196 1.2× 47 1.5k
Bruce Wyse Australia 20 592 1.3× 106 0.4× 358 1.6× 428 2.3× 142 0.8× 34 1.1k
Jose F. Perez‐Zoghbi United States 20 481 1.1× 210 0.8× 174 0.8× 349 1.8× 111 0.7× 43 1.2k
G. Droogmans Belgium 12 435 1.0× 510 2.0× 198 0.9× 199 1.1× 63 0.4× 16 886
Mario Barbieri Italy 17 579 1.3× 293 1.1× 508 2.3× 272 1.4× 416 2.4× 47 1.4k

Countries citing papers authored by Sendoa Tajada

Since Specialization
Citations

This map shows the geographic impact of Sendoa Tajada's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Sendoa Tajada with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Sendoa Tajada more than expected).

Fields of papers citing papers by Sendoa Tajada

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Sendoa Tajada. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Sendoa Tajada. The network helps show where Sendoa Tajada may publish in the future.

Co-authorship network of co-authors of Sendoa Tajada

This figure shows the co-authorship network connecting the top 25 collaborators of Sendoa Tajada. A scholar is included among the top collaborators of Sendoa Tajada based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Sendoa Tajada. Sendoa Tajada is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

15 of 15 papers shown
1.
Tajada, Sendoa, et al.. (2024). Mitoception, or transfer of normal cell mitochondria to cancer cells, reverses remodeling of store-operated Ca2+ entry in tumor cells. Biochimica et Biophysica Acta (BBA) - Molecular Cell Research. 1872(1). 119862–119862. 1 indexed citations
2.
Tajada, Sendoa, et al.. (2023). Transcriptional Basis of Ca2+ Remodeling Reversal Induced by Polyamine Synthesis Inhibition in Colorectal Cancer Cells. Cancers. 15(5). 1600–1600. 10 indexed citations
3.
Lee, Frank K., Jane C. Lee, Bo Shui, et al.. (2021). Genetically engineered mice for combinatorial cardiovascular optobiology. eLife. 10. 9 indexed citations
4.
Tajada, Sendoa & Carlos Villalobos. (2020). Calcium Permeable Channels in Cancer Hallmarks. Frontiers in Pharmacology. 11. 968–968. 69 indexed citations
5.
Palacio, Stephanie, Nicholas R. Klug, Sendoa Tajada, et al.. (2020). Kv2.1 channels play opposing roles in regulating membrane potential, Ca 2+ channel function, and myogenic tone in arterial smooth muscle. Proceedings of the National Academy of Sciences. 117(7). 3858–3866. 31 indexed citations
6.
Sato, Daisuke, Sendoa Tajada, Claudia M. Moreno, et al.. (2019). A stochastic model of ion channel cluster formation in the plasma membrane. The Journal of General Physiology. 151(9). 1116–1134. 28 indexed citations
7.
Ghosh, Debapriya, Madeline Nieves‐Cintrón, Sendoa Tajada, et al.. (2018). Dynamic L-type CaV1.2 channel trafficking facilitates CaV1.2 clustering and cooperative gating. Biochimica et Biophysica Acta (BBA) - Molecular Cell Research. 1865(9). 1341–1355. 27 indexed citations
8.
Mata, Ana de la, Sendoa Tajada, Rose E. Dixon, et al.. (2018). BIN1 Induces the Formation of T-Tubules and Adult-Like Ca2+ Release Units in Developing Cardiomyocytes. Stem Cells. 37(1). 54–64. 41 indexed citations
9.
Tajada, Sendoa, et al.. (2017). Distance constraints on activation of TRPV4 channels by AKAP150-bound PKCα in arterial myocytes. The Journal of General Physiology. 149(6). 639–659. 42 indexed citations
10.
Moreno, Claudia M., Rose E. Dixon, Sendoa Tajada, et al.. (2016). Ca2+ entry into neurons is facilitated by cooperative gating of clustered CaV1.3 channels. eLife. 5. 62 indexed citations
11.
Meseguer, Víctor, Yeranddy A. Alpízar, Enoch Luis, et al.. (2014). TRPA1 channels mediate acute neurogenic inflammation and pain produced by bacterial endotoxins. Nature Communications. 5(1). 3125–3125. 356 indexed citations breakdown →
12.
Alpízar, Yeranddy A., Sendoa Tajada, Bernd Nilius, et al.. (2014). Cinnamaldehyde inhibits L-type calcium channels in mouse ventricular cardiomyocytes and vascular smooth muscle cells. Pflügers Archiv - European Journal of Physiology. 466(11). 2089–2099. 32 indexed citations
13.
Tajada, Sendoa, Pilar Cidad, Olaia Colinas, et al.. (2013). Down‐regulation of CaV1.2 channels during hypertension: how fewer CaV1.2 channels allow more Ca2+ into hypertensive arterial smooth muscle. The Journal of Physiology. 591(24). 6175–6191. 30 indexed citations
14.
Tajada, Sendoa, Pilar Cidad, Alejandro Moreno‐Domínguez, M. Teresa Pérez‐García, & José R. López‐López. (2012). High blood pressure associates with the remodelling of inward rectifier K+ channels in mice mesenteric vascular smooth muscle cells. The Journal of Physiology. 590(23). 6075–6091. 37 indexed citations
15.
Cidad, Pilar, Daniel García‐Arribas, Eduardo Miguel‐Velado, et al.. (2012). Kv1.3 Channels Can Modulate Cell Proliferation During Phenotypic Switch by an Ion-Flux Independent Mechanism. Arteriosclerosis Thrombosis and Vascular Biology. 32(5). 1299–1307. 70 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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