Seham Skah

640 total citations
11 papers, 498 citations indexed

About

Seham Skah is a scholar working on Molecular Biology, Endocrinology, Diabetes and Metabolism and Genetics. According to data from OpenAlex, Seham Skah has authored 11 papers receiving a total of 498 indexed citations (citations by other indexed papers that have themselves been cited), including 5 papers in Molecular Biology, 4 papers in Endocrinology, Diabetes and Metabolism and 4 papers in Genetics. Recurrent topics in Seham Skah's work include Wnt/β-catenin signaling in development and cancer (3 papers), Acute Lymphoblastic Leukemia research (2 papers) and Genetics and Neurodevelopmental Disorders (2 papers). Seham Skah is often cited by papers focused on Wnt/β-catenin signaling in development and cancer (3 papers), Acute Lymphoblastic Leukemia research (2 papers) and Genetics and Neurodevelopmental Disorders (2 papers). Seham Skah collaborates with scholars based in France, Norway and Belgium. Seham Skah's co-authors include Michelina Plateroti, Julien Nadjar, M Sirakov, Jacques Samarut, Amélie Rezza, Colette Roche, Heidi Kiil Blomhoff, Ellen Ruud, Sheerazed Boulkroun and Annabel Berthon and has published in prestigious journals such as Gastroenterology, Journal of Cell Science and Human Molecular Genetics.

In The Last Decade

Seham Skah

10 papers receiving 495 citations

Peers

Seham Skah
Sushela Chaidarun United States
Ferdous M. Barlaskar United States
María Roqué Argentina
Catherine McCaig United Kingdom
Carey Welsh United States
Xinbo Liao United States
Stacey E. Wirt United States
Yael Bar Israel
Sushela Chaidarun United States
Seham Skah
Citations per year, relative to Seham Skah Seham Skah (= 1×) peers Sushela Chaidarun

Countries citing papers authored by Seham Skah

Since Specialization
Citations

This map shows the geographic impact of Seham Skah's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Seham Skah with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Seham Skah more than expected).

Fields of papers citing papers by Seham Skah

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Seham Skah. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Seham Skah. The network helps show where Seham Skah may publish in the future.

Co-authorship network of co-authors of Seham Skah

This figure shows the co-authorship network connecting the top 25 collaborators of Seham Skah. A scholar is included among the top collaborators of Seham Skah based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Seham Skah. Seham Skah is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

11 of 11 papers shown
1.
Skah, Seham, et al.. (2025). Development of a Novel Bifunctional Anti-CD47 Fusion Protein with Improved Efficacy and a Favorable Safety Profile. Molecular Cancer Therapeutics. 24(6). 816–827.
2.
Ford, Anthony M., et al.. (2019). Targeting cyclooxygenase by indomethacin decelerates progression of acute lymphoblastic leukemia in a xenograft model. Blood Advances. 3(21). 3181–3190. 7 indexed citations
3.
Skah, Seham, Karin M. Gilljam, Christian Bindesbøll, et al.. (2018). cAMP-mediated autophagy inhibits DNA damage-induced death of leukemia cells independent of p53. Oncotarget. 9(54). 30434–30449. 19 indexed citations
4.
Skah, Seham, et al.. (2017). The thyroid hormone nuclear receptors and the Wnt/β-catenin pathway: An intriguing liaison. Developmental Biology. 422(2). 71–82. 32 indexed citations
5.
Skah, Seham, Julien Nadjar, M Sirakov, & Michelina Plateroti. (2015). 795 The Secreted Frizzled-Related Protein 2 Modulates Cell Fate and the WNT Pathway in the Murine Intestinal Epithelium. Gastroenterology. 148(4). S–156. 1 indexed citations
6.
Skah, Seham, Hege Ugland, Ola Myklebost, et al.. (2015). Bone marrow stroma-derived PGE2 protects BCP-ALL cells from DNA damage-induced p53 accumulation and cell death. Molecular Cancer. 14(1). 14–14. 73 indexed citations
7.
Skah, Seham, Julien Nadjar, M Sirakov, & Michelina Plateroti. (2014). The secreted Frizzled-Related Protein 2 modulates cell fate and the Wnt pathway in the murine intestinal epithelium. Experimental Cell Research. 330(1). 56–65. 21 indexed citations
8.
Berthon, Annabel, Coralie Drelon, Bruno Ragazzon, et al.. (2013). WNT/β-catenin signalling is activated in aldosterone-producing adenomas and controls aldosterone production. Human Molecular Genetics. 23(4). 889–905. 148 indexed citations
9.
Sirakov, M, Seham Skah, Julien Nadjar, & Michelina Plateroti. (2012). Thyroid hormone's action on progenitor/stem cell biology: New challenge for a classic hormone?. Biochimica et Biophysica Acta (BBA) - General Subjects. 1830(7). 3917–3927. 39 indexed citations
10.
Rezza, Amélie, Seham Skah, Colette Roche, et al.. (2010). The overexpression of the putative gut stem cell marker Musashi-1 induces tumorigenesis through Wnt and Notch activation. Journal of Cell Science. 123(19). 3256–3265. 94 indexed citations
11.
Kress, Elsa, Seham Skah, M Sirakov, et al.. (2010). Cooperation Between the Thyroid Hormone Receptor TRα1 and the WNT Pathway in the Induction of Intestinal Tumorigenesis. Gastroenterology. 138(5). 1863–1874.e1. 64 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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