Scott Zeitlin

8.2k total citations · 1 hit paper
47 papers, 5.5k citations indexed

About

Scott Zeitlin is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Neurology. According to data from OpenAlex, Scott Zeitlin has authored 47 papers receiving a total of 5.5k indexed citations (citations by other indexed papers that have themselves been cited), including 39 papers in Molecular Biology, 39 papers in Cellular and Molecular Neuroscience and 11 papers in Neurology. Recurrent topics in Scott Zeitlin's work include Genetic Neurodegenerative Diseases (36 papers), Mitochondrial Function and Pathology (30 papers) and Muscle Physiology and Disorders (13 papers). Scott Zeitlin is often cited by papers focused on Genetic Neurodegenerative Diseases (36 papers), Mitochondrial Function and Pathology (30 papers) and Muscle Physiology and Disorders (13 papers). Scott Zeitlin collaborates with scholars based in United States, United Kingdom and Italy. Scott Zeitlin's co-authors include Ioannis Dragatsis, Argiris Efstratiadis, Michael S. Levine, Jeh-Ping Liu, Marie‐Françoise Chesselet, Virginia E. Papaioannou, Deborah L. Chapman, Liliana Menalled, Marcelo B. Soares and Paula Dietrich and has published in prestigious journals such as Science, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.

In The Last Decade

Scott Zeitlin

47 papers receiving 5.5k citations

Hit Papers

Increased apoptosis and early embryonic lethality in mice... 1995 2026 2005 2015 1995 100 200 300 400 500

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Scott Zeitlin United States 34 4.0k 3.6k 1.2k 507 436 47 5.5k
Ioannis Dragatsis United States 38 3.0k 0.8× 2.6k 0.7× 711 0.6× 513 1.0× 482 1.1× 57 5.0k
Chiara Zuccato Italy 32 5.0k 1.3× 5.1k 1.4× 1.6k 1.4× 481 0.9× 389 0.9× 60 7.2k
Shihua Li China 38 4.8k 1.2× 3.7k 1.0× 1.3k 1.1× 528 1.0× 614 1.4× 114 6.3k
Stefan M. Pulst United States 44 4.1k 1.0× 3.3k 0.9× 2.3k 2.0× 571 1.1× 445 1.0× 157 6.2k
Caterina Mariotti Italy 45 4.4k 1.1× 3.7k 1.0× 1.8k 1.6× 393 0.8× 375 0.9× 158 6.3k
Toshiyuki Araki Japan 33 2.7k 0.7× 2.2k 0.6× 529 0.5× 320 0.6× 437 1.0× 94 5.6k
Riccardo Brambilla Italy 37 2.8k 0.7× 2.5k 0.7× 397 0.3× 579 1.1× 649 1.5× 75 5.0k
Sang H. Lee United States 33 2.6k 0.6× 2.2k 0.6× 471 0.4× 270 0.5× 667 1.5× 62 4.7k
Willeke van Roon‐Mom Netherlands 39 2.8k 0.7× 2.1k 0.6× 778 0.7× 359 0.7× 170 0.4× 109 4.6k
Patrice D. Smith Canada 20 1.8k 0.5× 2.1k 0.6× 960 0.8× 200 0.4× 477 1.1× 33 3.9k

Countries citing papers authored by Scott Zeitlin

Since Specialization
Citations

This map shows the geographic impact of Scott Zeitlin's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Scott Zeitlin with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Scott Zeitlin more than expected).

Fields of papers citing papers by Scott Zeitlin

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Scott Zeitlin. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Scott Zeitlin. The network helps show where Scott Zeitlin may publish in the future.

Co-authorship network of co-authors of Scott Zeitlin

This figure shows the co-authorship network connecting the top 25 collaborators of Scott Zeitlin. A scholar is included among the top collaborators of Scott Zeitlin based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Scott Zeitlin. Scott Zeitlin is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Barnat, Monia, Mariacristina Capizzi, Radhia Kacher, et al.. (2020). Huntington’s disease alters human neurodevelopment. Science. 369(6505). 787–793. 188 indexed citations
2.
McKinstry, Spencer U., Namsoo Kim, M. Ilcim Ozlu, et al.. (2020). Striatal Projection Neurons Require Huntingtin for Synaptic Connectivity and Survival. Cell Reports. 30(3). 642–657.e6. 38 indexed citations
3.
Franich, Nicholas R., Manuela Basso, Joseph Ochaba, et al.. (2018). Striatal Mutant Huntingtin Protein Levels Decline with Age in Homozygous Huntington’s Disease Knock-In Mouse Models. Journal of Huntington s Disease. 7(2). 137–150. 13 indexed citations
4.
Liu, Jeh-Ping & Scott Zeitlin. (2017). Is Huntingtin Dispensable in the Adult Brain?. Journal of Huntington s Disease. 6(1). 1–17. 36 indexed citations
5.
Fang, Cheng, et al.. (2012). Neurobehavioral abnormalities in a brain-specific NADPH-cytochrome P450 reductase knockout mouse model. Neuroscience. 218. 170–180. 4 indexed citations
7.
Conforti, Paola, Stefano Camnasio, Marta Valenza, et al.. (2012). Lack of huntingtin promotes neural stem cells differentiation into glial cells while neurons expressing huntingtin with expanded polyglutamine tracts undergo cell death. Neurobiology of Disease. 50. 160–170. 30 indexed citations
8.
Sardo, Valentina Lo, Chiara Zuccato, Germano Gaudenzi, et al.. (2012). An evolutionary recent neuroepithelial cell adhesion function of huntingtin implicates ADAM10-Ncadherin. Nature Neuroscience. 15(5). 713–721. 87 indexed citations
9.
Zheng, Shuqiu, Erin B. D. Clabough, Sovan Sarkar, et al.. (2010). Deletion of the Huntingtin Polyglutamine Stretch Enhances Neuronal Autophagy and Longevity in Mice. PLoS Genetics. 6(2). e1000838–e1000838. 119 indexed citations
10.
Zhang, Hua, Sudipto Das, Quan‐Zhen Li, et al.. (2008). Elucidating a normal function of huntingtin by functional and microarray analysis of huntingtin-null mouse embryonic fibroblasts. BMC Neuroscience. 9(1). 38–38. 22 indexed citations
11.
Li, Xueyi, Ellen Sapp, Antonio Valencia, et al.. (2008). A function of huntingtin in guanine nucleotide exchange on Rab11. Neuroreport. 19(16). 1643–1647. 51 indexed citations
12.
Zuccato, Chiara, Nikolai N. Belyaev, Paola Conforti, et al.. (2007). Widespread Disruption of Repressor Element-1 Silencing Transcription Factor/Neuron-Restrictive Silencer Factor Occupancy at Its Target Genes in Huntington's Disease. Journal of Neuroscience. 27(26). 6972–6983. 224 indexed citations
14.
Menalled, Liliana, et al.. (2003). Time course of early motor and neuropathological anomalies in a knock‐in mouse model of Huntington's disease with 140 CAG repeats. The Journal of Comparative Neurology. 465(1). 11–26. 382 indexed citations
15.
Zakharenko, Stanislav S., Susan L. Patterson, Ioannis Dragatsis, et al.. (2003). Presynaptic BDNF Required for a Presynaptic but Not Postsynaptic Component of LTP at Hippocampal CA1-CA3 Synapses. Neuron. 39(6). 975–990. 259 indexed citations
16.
Dragatsis, Ioannis, Paula Dietrich, & Scott Zeitlin. (2000). Expression of the Huntingtin-associated protein 1 gene in the developing and adult mouse. Neuroscience Letters. 282(1-2). 37–40. 45 indexed citations
17.
Dragatsis, Ioannis, Michael S. Levine, & Scott Zeitlin. (2000). Inactivation of Hdh in the brain and testis results in progressive neurodegeneration and sterility in mice. Nature Genetics. 26(3). 300–306. 430 indexed citations
18.
Dragatsis, Ioannis & Scott Zeitlin. (2000). CaMKII?-cre transgene expression and recombination patterns in the mouse brain. genesis. 26(2). 133–135. 144 indexed citations
19.
Zeitlin, Scott, Jeh-Ping Liu, Deborah L. Chapman, Virginia E. Papaioannou, & Argiris Efstratiadis. (1995). Increased apoptosis and early embryonic lethality in mice nullizygous for the Huntington's disease gene homologue. Nature Genetics. 11(2). 155–163. 597 indexed citations breakdown →
20.
Chan, Shu Jin, Vasso Episkopou, Scott Zeitlin, et al.. (1984). Guinea pig preproinsulin gene: an evolutionary compromise?. Proceedings of the National Academy of Sciences. 81(16). 5046–5050. 62 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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