Scott Marshall

854 total citations
32 papers, 468 citations indexed

About

Scott Marshall is a scholar working on Genetics, Oncology and Immunology. According to data from OpenAlex, Scott Marshall has authored 32 papers receiving a total of 468 indexed citations (citations by other indexed papers that have themselves been cited), including 16 papers in Genetics, 12 papers in Oncology and 11 papers in Immunology. Recurrent topics in Scott Marshall's work include Chronic Lymphocytic Leukemia Research (15 papers), Cancer-related Molecular Pathways (8 papers) and Advanced Breast Cancer Therapies (6 papers). Scott Marshall is often cited by papers focused on Chronic Lymphocytic Leukemia Research (15 papers), Cancer-related Molecular Pathways (8 papers) and Advanced Breast Cancer Therapies (6 papers). Scott Marshall collaborates with scholars based in United Kingdom, Italy and United States. Scott Marshall's co-authors include Udo Holtick, Gösta Berlin, Eeva Juvonen, Julia Scarisbrick, Peter C. Taylor, Kenneth Douglas, Elaine Willmore, Jonathan P. Wallis, Helen Marr and Anne M. Dickinson and has published in prestigious journals such as SHILAP Revista de lepidopterología, Blood and International Journal of Molecular Sciences.

In The Last Decade

Scott Marshall

31 papers receiving 451 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Scott Marshall United Kingdom 11 161 141 135 84 78 32 468
Maciej Kaźmierczak Poland 13 63 0.4× 194 1.4× 90 0.7× 93 1.1× 18 0.2× 41 475
Christina Dickson United States 10 221 1.4× 276 2.0× 236 1.7× 177 2.1× 58 0.7× 13 931
Daniela Graham United States 9 442 2.7× 313 2.2× 128 0.9× 44 0.5× 95 1.2× 22 871
Sander Strengholt United States 8 101 0.6× 153 1.1× 103 0.8× 78 0.9× 31 0.4× 22 455
Liliana Morales Argentina 2 134 0.8× 236 1.7× 144 1.1× 141 1.7× 22 0.3× 2 625
Shigeyuki Toyoizumi Japan 9 138 0.9× 285 2.0× 116 0.9× 162 1.9× 37 0.5× 20 623
Joanne Ewing United Kingdom 8 91 0.6× 208 1.5× 55 0.4× 260 3.1× 14 0.2× 19 503
Pierre Brousset France 10 91 0.6× 130 0.9× 296 2.2× 85 1.0× 66 0.8× 23 582
Ilaria Nichele Italy 11 96 0.6× 142 1.0× 64 0.5× 209 2.5× 5 0.1× 23 413
Udomsak Bunworasate Thailand 12 132 0.8× 189 1.3× 208 1.5× 158 1.9× 63 0.8× 55 518

Countries citing papers authored by Scott Marshall

Since Specialization
Citations

This map shows the geographic impact of Scott Marshall's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Scott Marshall with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Scott Marshall more than expected).

Fields of papers citing papers by Scott Marshall

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Scott Marshall. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Scott Marshall. The network helps show where Scott Marshall may publish in the future.

Co-authorship network of co-authors of Scott Marshall

This figure shows the co-authorship network connecting the top 25 collaborators of Scott Marshall. A scholar is included among the top collaborators of Scott Marshall based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Scott Marshall. Scott Marshall is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Wallis, Jonathan P., Helen Marr, Scott Marshall, et al.. (2025). Targeting the MDM2-p53 Interaction with Siremadlin: A Promising Therapeutic Strategy for Treating TP53 Wild-Type Chronic Lymphocytic Leukemia. Cancers. 17(2). 274–274. 2 indexed citations
2.
Nakjang, Sirintra, Jonathan P. Wallis, Helen Marr, et al.. (2024). RNA Sequencing Reveals Candidate Genes and Pathways Associated with Resistance to MDM2 Antagonist Idasanutlin in TP53 Wild-Type Chronic Lymphocytic Leukemia. Biomedicines. 12(7). 1388–1388. 4 indexed citations
3.
Ciardullo, Carmela, Jonathan P. Wallis, Helen Marr, et al.. (2023). Splicing Modulation Results in Aberrant Isoforms and Protein Products of p53 Pathway Genes and the Sensitization of B Cells to Non-Genotoxic MDM2 Inhibition. International Journal of Molecular Sciences. 24(3). 2410–2410. 11 indexed citations
4.
Wallis, Jonathan P., Helen Marr, Scott Marshall, et al.. (2023). SF3B1 Mutations Are Associated with Resistance to Non-Genotoxic MDM2 Inhibition in Chronic Lymphocytic Leukemia. International Journal of Molecular Sciences. 24(14). 11335–11335. 7 indexed citations
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Barrow, Timothy M., Sirintra Nakjang, Susan J. Tudhope, et al.. (2020). Epigenome-wide analysis reveals functional modulators of drug sensitivity and post-treatment survival in chronic lymphocytic leukaemia. British Journal of Cancer. 124(2). 474–483. 15 indexed citations
8.
Szołtysek, Katarzyna, Carmela Ciardullo, Anna Walaszczyk, et al.. (2020). DAP Kinase-Related Apoptosis-Inducing Protein Kinase 2 (DRAK2) Is a Key Regulator and Molecular Marker in Chronic Lymphocytic Leukemia. International Journal of Molecular Sciences. 21(20). 7663–7663. 9 indexed citations
9.
Ciardullo, Carmela, Wei‐Yu Lin, Jonathan P. Wallis, et al.. (2019). Non-genotoxic MDM2 inhibition selectively induces a pro-apoptotic p53 gene signature in chronic lymphocytic leukemia cells. Haematologica. 104(12). 2429–2442. 23 indexed citations
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Howard, Dena, Talha Munir, Lucy McParland, et al.. (2017). Results of the randomized phase IIB ARCTIC trial of low-dose rituximab in previously untreated CLL. Leukemia. 31(11). 2416–2425. 21 indexed citations
13.
Herriott, Ashleigh, Susan J. Tudhope, Miranda Patterson, et al.. (2015). PARP1 expression, activity andex vivosensitivity to the PARP inhibitor, talazoparib (BMN 673), in chronic lymphocytic leukaemia. Oncotarget. 6(41). 43978–43991. 29 indexed citations
14.
Guennel, Tobias, et al.. (2014). Analytical strategies for discovery and replication of genetic effects in pharmacogenomic studies. Pharmacogenomics and Personalized Medicine. 7. 217–217. 1 indexed citations
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Alikhan, Raza, Rachel Rayment, David Keeling, et al.. (2013). The acute management of haemorrhage, surgery and overdose in patients receiving dabigatran. Emergency Medicine Journal. 31(2). 163–168. 62 indexed citations
17.
Marshall, Scott. (2009). Tigecycline-Induced Pancreatitis. Hospital Pharmacy. 44(3). 239–241. 13 indexed citations
18.
Scarisbrick, Julia, Peter C. Taylor, Udo Holtick, et al.. (2008). U.K. consensus statement on the use of extracorporeal photopheresis for treatment of cutaneous T-cell lymphoma and chronic graft-versus-host disease. British Journal of Dermatology. 158(4). 659–678. 105 indexed citations
19.
Marshall, Scott. (2006). Technology Insight: ECP for the treatment of GvHD—can we offer selective immune control without generalized immunosuppression?. Nature Clinical Practice Oncology. 3(6). 302–314. 81 indexed citations
20.
Wang, Xiaonong, Matthew Collin, Lisbet Sviland, et al.. (2006). Skin Explant Model of Human Graft-versus-Host Disease: Prediction of Clinical Outcome and Correlation with Biological Risk Factors. Biology of Blood and Marrow Transplantation. 12(2). 152–159. 14 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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