Scott M. Ocheltree

559 citations
12 papers · 454 indexed · h-index 11
Topics
Drug Transport and Resistance Mechanisms (6 papers)Amino Acid Enzymes and Metabolism (4 papers)Barrier Structure and Function Studies (3 papers)

In The Last Decade

Scott M. Ocheltree

12 papers receiving 447 citations

Peers

Scott M. Ocheltree
Comparison fields: 5 of 69
  • Oncology 220
  • Pediatrics, Perinatology and Child Health 96
  • Molecular Biology 93
  • Neurology 87
  • Biochemistry 72
Replace Salah Yousif with:
Salah Yousif France
Hitoshi Maegawa Japan
T. Kitazawa Japan
Takeshi Suzuki Japan
Destiny B. Sykes United States
Lucy Sanchez‐Covarrubias United States
Kouichi Kawazu Japan
Toshiki Kurosawa Japan
Shizuo Yamada Japan
Dušan Mitrović Serbia
Scott M. Ocheltree relative to Salah Yousif France Salah Yousif's profile →
Citations per field
00.5×1.5×
Salah Yousif · 1×
Citations per year

Countries citing papers authored by Scott M. Ocheltree

Since Specialization
Citations

This map shows the geographic impact of Scott M. Ocheltree's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Scott M. Ocheltree with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Scott M. Ocheltree more than expected).

Fields of papers citing papers by Scott M. Ocheltree

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Scott M. Ocheltree. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Scott M. Ocheltree. The network helps show where Scott M. Ocheltree may publish in the future.

Co-authorship network of co-authors of Scott M. Ocheltree

This figure shows the co-authorship network connecting the top 25 collaborators of Scott M. Ocheltree. A scholar is included among the top collaborators of Scott M. Ocheltree based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Scott M. Ocheltree. Scott M. Ocheltree is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

12 of 12 papers shown
#WorkIndexed citations
1 20
2 31
3 2
4 32
5 66
6 46
7 67
8 17
9 70
10 40
11 32
12 31

About Scott M. Ocheltree

Scott M. Ocheltree is a scholar working on Biochemistry, Neurology and Physiology, having authored 12 papers that have together received 454 indexed citations. Recurring topics across this work include Drug Transport and Resistance Mechanisms (6 papers), Amino Acid Enzymes and Metabolism (4 papers) and Barrier Structure and Function Studies (3 papers). The work is most often cited by research in Neurology (87 citations), Biochemistry (72 citations) and Oncology (220 citations). Scott M. Ocheltree has collaborated with scholars based in United States, France and Singapore. Frequent co-authors include David E. Smith, Richard F. Keep, Yongjun Hu, Hong Shen, Richard D. Egleton, Brian T. Hawkins, Jianming Xiang, Thomas P. Davis, Marcus Hompesch and Scott J. Jacober. Their work appears in journals such as Brain Research, Journal of Pharmacology and Experimental Therapeutics and Pharmaceutical Research.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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