Scott Lowe
Impact in
- Cancer Research top 0.02%
- MicroRNA in disease regulation
- Oncology top 0.02%
- Cancer-related Molecular Pathways
Papers in
- Oncology 144
- Cancer-related Molecular Pathways 91
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- RNA modifications and cancer 37
- Epigenetics and DNA Methylation 36
- CRISPR and Genetic Engineering 31
- Cell death mechanisms and regulation 29
- Ubiquitin and proteasome pathways 26
- Co-authors
- Tyler Jacks (9 shared papers)Athena W. Lin (6 shared papers)David E. Housman (4 shared papers)Gregory James Hannon (21 shared papers)Carlos Cordon‐Cardo (16 shared papers)Jordan S. Fridman (7 shared papers)H. Earl Ruley (3 shared papers)Edward R. Kastenhuber (12 shared papers)
- Journals
- Nature (38 papers)Cell (32 papers)Proceedings of the National Academy of Sciences (30 papers)Blood (22 papers)Cancer Discovery (19 papers)
- Partner nations
- United StatesGermanyCanada
In The Last Decade
Scott Lowe
358 papers receiving 79.7k citations
Scott Lowe's Hit Papers
Peers
Comparison fields: 5 of 198
- Cancer Research 17.6k
- Oncology 23.7k
- Molecular Biology 52.0k
- Aging 1.2k
- Immunology 9.6k
Countries citing papers authored by Scott Lowe
This map shows the geographic impact of Scott Lowe's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Scott Lowe with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Scott Lowe more than expected).
Fields of papers citing papers by Scott Lowe
This network shows the impact of papers produced by Scott Lowe. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Scott Lowe. The network helps show where Scott Lowe may publish in the future.
Co-authors
The 25 scholars most cited alongside Scott Lowe, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
Showing the 20 most-cited of 360 papers — load more, or switch the sort, to bring in the rest.
| # | Work | ||
|---|---|---|---|
| 1 | Molecular mechanisms of cell death: recommendations of the Nomenclature Committee on Cell Death 2018 Hit paper breakdown → | 2018 | 5296 |
| 2 | A microRNA polycistron as a potential human oncogene Hit paper breakdown → | 2005 | 3033 |
| 3 | p53-dependent apoptosis modulates the cytotoxicity of anticancer agents Hit paper breakdown → | 1993 | 2632 |
| 4 | p53 is required for radiation-induced apoptosis in mouse thymocytes Hit paper breakdown → | 1993 | 2567 |
| 5 | A microRNA component of the p53 tumour suppressor network Hit paper breakdown → | 2007 | 2304 |
| 6 | Senescence and tumour clearance is triggered by p53 restoration in murine liver carcinomas Hit paper breakdown → | 2007 | 2061 |
| 7 | Hypoxia-mediated selection of cells with diminished apoptotic potential in solid tumours Hit paper breakdown → | 1996 | 2039 |
| 8 | Apoptosis Hit paper breakdown → | 2002 | 1981 |
| 9 | Rb-Mediated Heterochromatin Formation and Silencing of E2F Target Genes during Cellular Senescence Hit paper breakdown → | 2003 | 1848 |
| 10 | Apoptosis in cancer Hit paper breakdown → | 2000 | 1630 |
| 11 | RNAi screen identifies Brd4 as a therapeutic target in acute myeloid leukaemia Hit paper breakdown → | 2011 | 1581 |
| 12 | Putting p53 in Context Hit paper breakdown → | 2017 | 1518 |
| 13 | p53 Status and the Efficacy of Cancer Therapy in Vivo Hit paper breakdown → | 1994 | 1313 |
| 14 | Control of apoptosis by p53 Hit paper breakdown → | 2003 | 1209 |
| 15 | Reversal of human cellular senescence: roles of the p53 and p16 pathways Hit paper breakdown → | 2003 | 1163 |
| 16 | Differential Requirement for Caspase 9 in Apoptotic Pathways In Vivo Hit paper breakdown → | 1998 | 1149 |
| 17 | Intrinsic tumour suppression Hit paper breakdown → | 2004 | 1063 |
| 18 | The microcosmos of cancer Hit paper breakdown → | 2012 | 965 |
| 19 | Identification and Validation of Oncogenes in Liver Cancer Using an Integrative Oncogenomic Approach Hit paper breakdown → | 2006 | 916 |
| 20 | A Senescence Program Controlled by p53 and p16INK4a Contributes to the Outcome of Cancer Therapy Hit paper breakdown → | 2002 | 895 |
About Scott Lowe
Scott Lowe is a scholar working on Oncology, Molecular Biology, Cancer Research, Hematology and Biotechnology, having authored 360 papers that have together received 81.5k indexed citations. Recurring topics across this work include Cancer-related Molecular Pathways (91 papers), RNA modifications and cancer (37 papers), Epigenetics and DNA Methylation (36 papers), CRISPR and Genetic Engineering (31 papers), Acute Myeloid Leukemia Research (31 papers), Cell death mechanisms and regulation (29 papers), Cancer Research and Treatments (28 papers) and Ubiquitin and proteasome pathways (26 papers). The work is most often cited by research in Cancer Research (17.6k citations), Oncology (23.7k citations), Molecular Biology (52.0k citations), Aging (1.2k citations) and Immunology (9.6k citations). Scott Lowe has collaborated with scholars based in United States, Germany and Canada. Frequent co-authors include Tyler Jacks, Athena W. Lin, David E. Housman, Gregory James Hannon, Carlos Cordon‐Cardo, Jordan S. Fridman, H. Earl Ruley, Edward R. Kastenhuber, Eva Hernando and Michael T. Hemann. Their work appears in journals such as Nature, Cell, Proceedings of the National Academy of Sciences, Blood and Cancer Discovery.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.