Sauli Haataja
- Molecular Biology
- Public Health, Environmental and Occupational Health top 5%
- Epidemiology top 10%
- Infectious Diseases top 5%
- Microbiology top 2%
- Co-authors
- Jukka FinneJukka HytönenArto T. PulliainenAnastassios C. PapageorgiouGöran MagnussonDamien McDevittGee W. LauAlexander P. Bryant
- Topics
- Streptococcal Infections and Treatments (20 papers)Antimicrobial Resistance in Staphylococcus (12 papers)Toxin Mechanisms and Immunotoxins (9 papers)
- Partner nations
- FinlandSwedenNetherlands
In The Last Decade
Sauli Haataja
36 papers receiving 1.3k citations
Peers
Comparison fields: 5 of 84
- Molecular Biology 566
- Public Health, Environmental and Occupational Health 484
- Epidemiology 357
- Infectious Diseases 279
- Microbiology 217
Countries citing papers authored by Sauli Haataja
This map shows the geographic impact of Sauli Haataja's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Sauli Haataja with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Sauli Haataja more than expected).
Fields of papers citing papers by Sauli Haataja
This network shows the impact of papers produced by Sauli Haataja. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Sauli Haataja. The network helps show where Sauli Haataja may publish in the future.
Co-authorship network of co-authors of Sauli Haataja
This figure shows the co-authorship network connecting the top 25 collaborators of Sauli Haataja. A scholar is included among the top collaborators of Sauli Haataja based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Sauli Haataja. Sauli Haataja is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 11 | |
| 2 | 10 | |
| 3 | 33 | |
| 4 | 28 | |
| 5 | 36 | |
| 6 | 14 | |
| 7 | 45 | |
| 8 | 25 | |
| 9 | 26 | |
| 10 | 36 | |
| 11 | 38 | |
| 12 | 20 | |
| 13 | 44 | |
| 14 | 31 | |
| 15 | 55 | |
| 16 | 10 | |
| 17 | 75 | |
| 18 | 234 | |
| 19 | 17 | |
| 20 | 26 |
About Sauli Haataja
Sauli Haataja is a scholar working on Structural Biology, Public Health, Environmental and Occupational Health and Infectious Diseases, having authored 36 papers that have together received 1.3k indexed citations. Recurring topics across this work include Streptococcal Infections and Treatments (20 papers), Antimicrobial Resistance in Staphylococcus (12 papers) and Toxin Mechanisms and Immunotoxins (9 papers). The work is most often cited by research in Microbiology (217 citations), Endocrinology (104 citations) and Public Health, Environmental and Occupational Health (484 citations). Sauli Haataja has collaborated with scholars based in Finland, Sweden and Netherlands. Frequent co-authors include Jukka Finne, Jukka Hytönen, Arto T. Pulliainen, Anastassios C. Papageorgiou, Göran Magnusson, Damien McDevitt, Gee W. Lau, Alexander P. Bryant, Andrea Marra and Michael A. Lonetto. Their work appears in journals such as Journal of the American Chemical Society, Journal of Biological Chemistry and PLoS ONE.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.