Sari S. Hannila

664 total citations
18 papers, 489 citations indexed

About

Sari S. Hannila is a scholar working on Cellular and Molecular Neuroscience, Molecular Biology and Developmental Neuroscience. According to data from OpenAlex, Sari S. Hannila has authored 18 papers receiving a total of 489 indexed citations (citations by other indexed papers that have themselves been cited), including 16 papers in Cellular and Molecular Neuroscience, 12 papers in Molecular Biology and 6 papers in Developmental Neuroscience. Recurrent topics in Sari S. Hannila's work include Nerve injury and regeneration (16 papers), Signaling Pathways in Disease (9 papers) and Axon Guidance and Neuronal Signaling (6 papers). Sari S. Hannila is often cited by papers focused on Nerve injury and regeneration (16 papers), Signaling Pathways in Disease (9 papers) and Axon Guidance and Neuronal Signaling (6 papers). Sari S. Hannila collaborates with scholars based in Canada and United States. Sari S. Hannila's co-authors include Marie T. Filbin, Michael D. Kawaja, Mustafa M. Siddiq, Gregory M. Ross, Erica L. Richman, Javad Alizadeh, Saeid Ghavami, Jianwei Hou, Ronald P. Hart and Jason B. Carmel and has published in prestigious journals such as Journal of Biological Chemistry, Journal of Neuroscience and The Journal of Comparative Neurology.

In The Last Decade

Sari S. Hannila

18 papers receiving 485 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Sari S. Hannila Canada 12 305 197 168 112 47 18 489
Berit Powers United States 9 172 0.6× 350 1.8× 119 0.7× 106 0.9× 65 1.4× 14 636
Do-Hun Lee United States 8 256 0.8× 143 0.7× 159 0.9× 153 1.4× 88 1.9× 9 474
Bogdan Beirowski United Kingdom 6 317 1.0× 170 0.9× 142 0.8× 42 0.4× 79 1.7× 7 516
Masaya Nakamura Japan 10 193 0.6× 86 0.4× 120 0.7× 163 1.5× 48 1.0× 15 413
H. Hyatt Sachs United States 9 247 0.8× 191 1.0× 192 1.1× 51 0.5× 94 2.0× 10 467
Diana Kindler Switzerland 8 217 0.7× 98 0.5× 112 0.7× 137 1.2× 83 1.8× 12 449
Yang Qiu China 12 180 0.6× 155 0.8× 157 0.9× 71 0.6× 120 2.6× 27 541
Véronique Menet France 5 304 1.0× 187 0.9× 206 1.2× 122 1.1× 124 2.6× 5 517
Perrine Gaub Canada 10 362 1.2× 426 2.2× 228 1.4× 45 0.4× 39 0.8× 14 765

Countries citing papers authored by Sari S. Hannila

Since Specialization
Citations

This map shows the geographic impact of Sari S. Hannila's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Sari S. Hannila with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Sari S. Hannila more than expected).

Fields of papers citing papers by Sari S. Hannila

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Sari S. Hannila. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Sari S. Hannila. The network helps show where Sari S. Hannila may publish in the future.

Co-authorship network of co-authors of Sari S. Hannila

This figure shows the co-authorship network connecting the top 25 collaborators of Sari S. Hannila. A scholar is included among the top collaborators of Sari S. Hannila based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Sari S. Hannila. Sari S. Hannila is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

18 of 18 papers shown
1.
2.
Alizadeh, Javad, et al.. (2021). Inhibition of Autophagy Flux Promotes Secretion of Chondroitin Sulfate Proteoglycans in Primary Rat Astrocytes. Molecular Neurobiology. 58(12). 6077–6091. 28 indexed citations
3.
Siddiq, Mustafa M., Sari S. Hannila, Elena Nikulina, et al.. (2021). Extracellular histones, a new class of inhibitory molecules of CNS axonal regeneration. Brain Communications. 3(4). fcab271–fcab271. 5 indexed citations
4.
Hannila, Sari S., et al.. (2021). Working from within: how secretory leukocyte protease inhibitor regulates the expression of pro-inflammatory genes. Biochemistry and Cell Biology. 100(1). 1–8. 6 indexed citations
5.
Nikulina, Elena, et al.. (2020). Myelin‐associated glycoprotein inhibits neurite outgrowth through inactivation of the small GTPase Rap1. FEBS Letters. 594(9). 1389–1402. 2 indexed citations
6.
7.
Siddiq, Mustafa M., Sari S. Hannila, Jason B. Carmel, et al.. (2015). Metallothionein-I/II Promotes Axonal Regeneration in the Central Nervous System. Journal of Biological Chemistry. 290(26). 16343–16356. 17 indexed citations
8.
Siddiq, Mustafa M. & Sari S. Hannila. (2015). Looking downstream: the role of cyclic AMP-regulated genes in axonal regeneration. Frontiers in Molecular Neuroscience. 8. 26–26. 26 indexed citations
9.
Hannila, Sari S., et al.. (2014). Transforming growth factor β-induced expression of chondroitin sulfate proteoglycans is mediated through non-Smad signaling pathways. Experimental Neurology. 263. 372–384. 35 indexed citations
10.
Hannila, Sari S.. (2014). Secretory Leukocyte Protease Inhibitor (SLPI). The Neuroscientist. 21(6). 630–636. 11 indexed citations
11.
Hannila, Sari S., Mustafa M. Siddiq, Jason B. Carmel, et al.. (2013). Secretory Leukocyte Protease Inhibitor Reverses Inhibition by CNS Myelin, Promotes Regeneration in the Optic Nerve, and Suppresses Expression of the Transforming Growth Factor-β Signaling Protein Smad2. Journal of Neuroscience. 33(12). 5138–5151. 29 indexed citations
12.
Hannila, Sari S. & Marie T. Filbin. (2007). The role of cyclic AMP signaling in promoting axonal regeneration after spinal cord injury. Experimental Neurology. 209(2). 321–332. 217 indexed citations
13.
Hannila, Sari S., Mustafa M. Siddiq, & Marie T. Filbin. (2006). Therapeutic Approaches to Promoting Axonal Regeneration in the Adult Mammalian Spinal Cord. International review of neurobiology. 77. 57–105. 18 indexed citations
14.
Hannila, Sari S. & Michael D. Kawaja. (2005). Nerve growth factor‐mediated collateral sprouting of central sensory axons into deafferentated regions of the dorsal horn is enhanced in the absence of the p75 neurotrophin receptor. The Journal of Comparative Neurology. 486(4). 331–343. 23 indexed citations
15.
Hannila, Sari S., et al.. (2004). TrkA and mitogen‐activated protein kinase phosphorylation are enhanced in sympathetic neurons lacking functional p75 neurotrophin receptor expression. European Journal of Neuroscience. 19(10). 2903–2908. 21 indexed citations
16.
Hannila, Sari S. & Michael D. Kawaja. (2003). Distribution of central sensory axons in transgenic mice overexpressing nerve growth factor and lacking functional p75 neurotrophin receptor expression. European Journal of Neuroscience. 18(2). 312–322. 11 indexed citations
17.
Hannila, Sari S. & Michael D. Kawaja. (1999). Nerve growth factor-induced growth of sympathetic axons into the optic tract of mature mice is enhanced by an absence of p75NTR expression. Journal of Neurobiology. 39(1). 51–66. 23 indexed citations
18.
Hannila, Sari S. & Michael D. Kawaja. (1999). Nerve growth factor–induced growth of sympathetic axons into the optic tract of mature mice is enhanced by an absence of p75NTR expression. Journal of Neurobiology. 39(1). 51–66. 3 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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