Sarah E. Heuer

536 total citations
21 papers, 295 citations indexed

About

Sarah E. Heuer is a scholar working on Neurology, Physiology and Rheumatology. According to data from OpenAlex, Sarah E. Heuer has authored 21 papers receiving a total of 295 indexed citations (citations by other indexed papers that have themselves been cited), including 11 papers in Neurology, 7 papers in Physiology and 6 papers in Rheumatology. Recurrent topics in Sarah E. Heuer's work include Neuroinflammation and Neurodegeneration Mechanisms (11 papers), Alzheimer's disease research and treatments (7 papers) and Systemic Lupus Erythematosus Research (6 papers). Sarah E. Heuer is often cited by papers focused on Neuroinflammation and Neurodegeneration Mechanisms (11 papers), Alzheimer's disease research and treatments (7 papers) and Systemic Lupus Erythematosus Research (6 papers). Sarah E. Heuer collaborates with scholars based in United States and Belgium. Sarah E. Heuer's co-authors include Catherine C. Kaczorowski, Sarah M. Neuner, Kristen M.S. O’Connell, Matthew J. Huentelman, Amrie C. Grammer, Michelle D. Catalina, Robert Robl, Peter E. Lipsky, Gareth R. Howell and Vivek M. Philip and has published in prestigious journals such as Neuron, Blood and The Journal of Immunology.

In The Last Decade

Sarah E. Heuer

20 papers receiving 282 citations

Peers

Sarah E. Heuer
Roger Willian de Lábio Brazil
Panagiota Davaki Greece
Ditte Olsen Denmark
Norikazu Hara Japan
Mibo Tang China
Lilja Jansson Finland
Galina Marsh United States
D. Lefranc France
Yajuan Wu China
Sofia Hassiotis Australia
Roger Willian de Lábio Brazil
Sarah E. Heuer
Citations per year, relative to Sarah E. Heuer Sarah E. Heuer (= 1×) peers Roger Willian de Lábio

Countries citing papers authored by Sarah E. Heuer

Since Specialization
Citations

This map shows the geographic impact of Sarah E. Heuer's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Sarah E. Heuer with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Sarah E. Heuer more than expected).

Fields of papers citing papers by Sarah E. Heuer

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Sarah E. Heuer. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Sarah E. Heuer. The network helps show where Sarah E. Heuer may publish in the future.

Co-authorship network of co-authors of Sarah E. Heuer

This figure shows the co-authorship network connecting the top 25 collaborators of Sarah E. Heuer. A scholar is included among the top collaborators of Sarah E. Heuer based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Sarah E. Heuer. Sarah E. Heuer is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Pearse, Richard V., Sarah E. Heuer, Peter R. Galle, et al.. (2025). CLU alleviates Alzheimer’s disease-relevant processes by modulating astrocyte reactivity and microglia-dependent synaptic density. Neuron. 113(12). 1925–1946.e11. 8 indexed citations
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Pearse, Richard V., Peter R. Galle, Sarah E. Heuer, et al.. (2025). Astrocyte induction of disease-associated microglia is suppressed by acute exposure to fAD neurons in human iPSC triple cultures. Cell Reports. 44(6). 115777–115777. 2 indexed citations
4.
Heuer, Sarah E., Erik B. Bloss, & Gareth R. Howell. (2024). Strategies to dissect microglia-synaptic interactions during aging and in Alzheimer's disease. Neuropharmacology. 254. 109987–109987. 6 indexed citations
5.
MacLean, Michael, et al.. (2024). Microglia depletion leads to increased susceptibility to ocular hypertension-dependent glaucoma. Frontiers in Aging Neuroscience. 16. 1396443–1396443. 8 indexed citations
6.
Heuer, Sarah E., et al.. (2023). Control of hippocampal synaptic plasticity by microglia–dendrite interactions depends on genetic context in mouse models of Alzheimer's disease. Alzheimer s & Dementia. 20(1). 601–614. 7 indexed citations
7.
Heuer, Sarah E., et al.. (2023). Genetic context drives age‐related disparities in synaptic maintenance and structure across cortical and hippocampal neuronal circuits. Aging Cell. 23(2). e14033–e14033. 5 indexed citations
8.
Reagan, Alaina M., Kristen D. Onos, Sarah E. Heuer, Michael Sasner, & Gareth R. Howell. (2022). Improving mouse models for the study of Alzheimer's disease. Current topics in developmental biology. 148. 79–113. 2 indexed citations
9.
Kingsmore, Kathryn M., Prathyusha Bachali, Michelle D. Catalina, et al.. (2021). Altered expression of genes controlling metabolism characterizes the tissue response to immune injury in lupus. Scientific Reports. 11(1). 14789–14789. 23 indexed citations
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Heuer, Sarah E., Sarah M. Neuner, Niran Hadad, et al.. (2020). Identifying the molecular systems that influence cognitive resilience to Alzheimer's disease in genetically diverse mice. Learning & Memory. 27(9). 355–371. 13 indexed citations
12.
Catalina, Michelle D., Sarah E. Heuer, Prathyusha Bachali, et al.. (2020). Analysis of gene expression from systemic lupus erythematosus synovium reveals myeloid cell-driven pathogenesis of lupus arthritis. Scientific Reports. 10(1). 17361–17361. 15 indexed citations
13.
Neuner, Sarah M., Sarah E. Heuer, Jigang Zhang, Vivek M. Philip, & Catherine C. Kaczorowski. (2019). Identification of Pre-symptomatic Gene Signatures That Predict Resilience to Cognitive Decline in the Genetically Diverse AD-BXD Model. Frontiers in Genetics. 10. 35–35. 20 indexed citations
14.
Kaczorowski, Catherine C., Sarah E. Heuer, & Chris Gaiteri. (2019). MULTI-OMICS ANALYSIS IDENTIFIES GENE NETWORKS ASSOCIATED WITH COGNITIVE AGING AND ALZHEIMER’S DISEASE. Innovation in Aging. 3(Supplement_1). S587–S588. 1 indexed citations
15.
Loberg, Matthew A., et al.. (2019). Cell-Extrinsic Stressors from the Aging Bone Marrow (BM) Microenvironment Promote Dnmt3a-Mutant Clonal Hematopoiesis. Blood. 134(Supplement_1). 5–5. 10 indexed citations
16.
Neuner, Sarah M., Sarah E. Heuer, Matthew J. Huentelman, Kristen M.S. O’Connell, & Catherine C. Kaczorowski. (2018). Harnessing Genetic Complexity to Enhance Translatability of Alzheimer’s Disease Mouse Models: A Path toward Precision Medicine. Neuron. 101(3). 399–411.e5. 131 indexed citations
17.
Heuer, Sarah E., et al.. (2017). Tissue infiltration of plasma cells/plasmablasts in patients with active Systemic Lupus Erthematosus (SLE). The Journal of Immunology. 198(Supplement_1). 55.8–55.8. 1 indexed citations
18.
Heuer, Sarah E., Michelle D. Catalina, Robert Robl, et al.. (2017). Common Patterns of Gene Expression in Tissues of Patients with Systemic Lupus Erythematosus Imply Similar Pathways of Molecular Pathogenesis. The Journal of Immunology. 198(Supplement_1). 224.17–224.17. 1 indexed citations
19.
Grammer, Amrie C., Sarah E. Heuer, Robert Robl, et al.. (2016). Drug repositioning in SLE: crowd-sourcing, literature-mining and Big Data analysis. Lupus. 25(10). 1150–1170. 35 indexed citations
20.
Heuer, Sarah E., et al.. (2001). Peripheral blood leukocyte response and macrophage function during Eimeria adenoeides infection in turkey poults. Journal of the Arkansas Academy of Science. 2(1). 13–20. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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