Sara E. Beese‐Sims

435 total citations
8 papers, 324 citations indexed

About

Sara E. Beese‐Sims is a scholar working on Molecular Biology, Aging and Cell Biology. According to data from OpenAlex, Sara E. Beese‐Sims has authored 8 papers receiving a total of 324 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Molecular Biology, 4 papers in Aging and 2 papers in Cell Biology. Recurrent topics in Sara E. Beese‐Sims's work include Genetics, Aging, and Longevity in Model Organisms (4 papers), Epigenetics and DNA Methylation (4 papers) and DNA Repair Mechanisms (3 papers). Sara E. Beese‐Sims is often cited by papers focused on Genetics, Aging, and Longevity in Model Organisms (4 papers), Epigenetics and DNA Methylation (4 papers) and DNA Repair Mechanisms (3 papers). Sara E. Beese‐Sims collaborates with scholars based in United States, Austria and United Kingdom. Sara E. Beese‐Sims's co-authors include Mónica P. Colaiácovo, Yang Shi, David E. Levin, Wolfgang Reiter, Karl Kuchler, Amanda C. Nottke, V Reinke, Gustav Ammerer, Ilse Dohnal and Christa Gregori and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Genes & Development and Genetics.

In The Last Decade

Sara E. Beese‐Sims

7 papers receiving 323 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Sara E. Beese‐Sims United States 6 273 116 45 37 31 8 324
Yaniv Harari Israel 10 200 0.7× 86 0.7× 48 1.1× 23 0.6× 33 1.1× 13 287
James Matthew Ragle United States 10 189 0.7× 182 1.6× 34 0.8× 16 0.4× 39 1.3× 19 310
Isaiah A.A. Neve United States 3 219 0.8× 125 1.1× 18 0.4× 7 0.2× 23 0.7× 3 336
Hirofumi Furuhashi Japan 10 257 0.9× 65 0.6× 104 2.3× 12 0.3× 42 1.4× 14 312
Inish O’Doherty United States 10 135 0.5× 68 0.6× 244 5.4× 13 0.4× 29 0.9× 16 510
Reza K. Oqani South Korea 12 224 0.8× 29 0.3× 29 0.6× 77 2.1× 45 1.5× 23 392
Natalibeth Barrera Uruguay 11 263 1.0× 24 0.2× 39 0.9× 19 0.5× 198 6.4× 14 528
Jean-Pierre J.-P. Ozil France 3 124 0.5× 17 0.1× 27 0.6× 27 0.7× 50 1.6× 3 397
Giorgia Pirino United States 5 164 0.6× 7 0.1× 62 1.4× 42 1.1× 11 0.4× 6 243
K. Papis Poland 10 151 0.6× 15 0.1× 46 1.0× 6 0.2× 47 1.5× 23 488

Countries citing papers authored by Sara E. Beese‐Sims

Since Specialization
Citations

This map shows the geographic impact of Sara E. Beese‐Sims's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Sara E. Beese‐Sims with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Sara E. Beese‐Sims more than expected).

Fields of papers citing papers by Sara E. Beese‐Sims

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Sara E. Beese‐Sims. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Sara E. Beese‐Sims. The network helps show where Sara E. Beese‐Sims may publish in the future.

Co-authorship network of co-authors of Sara E. Beese‐Sims

This figure shows the co-authorship network connecting the top 25 collaborators of Sara E. Beese‐Sims. A scholar is included among the top collaborators of Sara E. Beese‐Sims based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Sara E. Beese‐Sims. Sara E. Beese‐Sims is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

8 of 8 papers shown
1.
Martínez‐García, Marina, Saravanapriah Nadarajan, Nara Shin, et al.. (2023). GRAS-1 is a novel regulator of early meiotic chromosome dynamics in C. elegans. PLoS Genetics. 19(2). e1010666–e1010666.
2.
Zhang, Xiaojuan, et al.. (2021). Histone demethylase AMX-1 is necessary for proper sensitivity to interstrand crosslink DNA damage. PLoS Genetics. 17(7). e1009715–e1009715. 5 indexed citations
3.
Kim, Hyun‐Min, Sara E. Beese‐Sims, & Mónica P. Colaiácovo. (2018). Fanconi Anemia FANCM/FNCM-1 and FANCD2/FCD-2 Are Required for Maintaining Histone Methylation Levels and Interact with the Histone Demethylase LSD1/SPR-5 in Caenorhabditis elegans. Genetics. 209(2). 409–423. 12 indexed citations
4.
Greer, Eric Lieberman, Sara E. Beese‐Sims, Emily Brookes, et al.. (2014). A Histone Methylation Network Regulates Transgenerational Epigenetic Memory in C. elegans. Cell Reports. 7(1). 113–126. 129 indexed citations
5.
Reiter, Wolfgang, Ilse Dohnal, Christa Gregori, et al.. (2013). MAPK Hog1 closes the S. cerevisiae glycerol channel Fps1 by phosphorylating and displacing its positive regulators. Genes & Development. 27(23). 2590–2601. 85 indexed citations
6.
Beese‐Sims, Sara E., et al.. (2012). Mutants in the Candida glabrata Glycerol Channels Are Sensitized to Cell Wall Stress. Eukaryotic Cell. 11(12). 1512–1519. 13 indexed citations
7.
Nottke, Amanda C., et al.. (2011). SPR-5 is a histone H3K4 demethylase with a role in meiotic double-strand break repair. Proceedings of the National Academy of Sciences. 108(31). 12805–12810. 63 indexed citations
8.
Beese‐Sims, Sara E., et al.. (2011). Yeast Fps1 glycerol facilitator functions as a homotetramer. Yeast. 28(12). 815–819. 17 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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