Sara D’Andrea

1.2k total citations
23 papers, 807 citations indexed

About

Sara D’Andrea is a scholar working on Oncology, Molecular Biology and Cancer Research. According to data from OpenAlex, Sara D’Andrea has authored 23 papers receiving a total of 807 indexed citations (citations by other indexed papers that have themselves been cited), including 15 papers in Oncology, 14 papers in Molecular Biology and 8 papers in Cancer Research. Recurrent topics in Sara D’Andrea's work include Cancer Cells and Metastasis (8 papers), Microtubule and mitosis dynamics (7 papers) and Cancer-related Molecular Pathways (7 papers). Sara D’Andrea is often cited by papers focused on Cancer Cells and Metastasis (8 papers), Microtubule and mitosis dynamics (7 papers) and Cancer-related Molecular Pathways (7 papers). Sara D’Andrea collaborates with scholars based in Italy, Ukraine and United States. Sara D’Andrea's co-authors include Gustavo Baldassarre, Barbara Belletti, Mónica Schiappacassi, Stefania Berton, Alfonso Colombatti, Vincenzo Canzonieri, Francesca Lovat, Andrea Vecchione, Tiziana Perin and Maura Sonego and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Clinical Oncology and PLoS ONE.

In The Last Decade

Sara D’Andrea

23 papers receiving 799 citations

Peers

Sara D’Andrea

ONCOL

PRM

Stefania Berton Italy

Aashish Soni Germany

Morihiro Higo Japan

Takaaki Yasuhara Japan

Yukinao Kouzu Japan

Céline Clémenson France

Leila R. Martins Portugal

Natalia Bogdanova Germany

Jones T. Nauseef United States

James R. Whittle Australia

Stefania Berton Italy

Sara D’Andrea

400 ×0.9

405 ×1.2

ONCOL

265 ×0.8

193 ×1.3

66 ×0.7

PRM

Citations per year, relative to Sara D’Andrea Sara D’Andrea (= 1×) peers Stefania Berton

Countries citing papers authored by Sara D’Andrea

Since Specialization

Citations

This map shows the geographic impact of Sara D’Andrea's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Sara D’Andrea with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Sara D’Andrea more than expected).

Fields of papers citing papers by Sara D’Andrea

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Sara D’Andrea. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Sara D’Andrea. The network helps show where Sara D’Andrea may publish in the future.

Co-authorship network of co-authors of Sara D’Andrea

This figure shows the co-authorship network connecting the top 25 collaborators of Sara D’Andrea. A scholar is included among the top collaborators of Sara D’Andrea based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Sara D’Andrea. Sara D’Andrea is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Favero, Andrea, Ilenia Segatto, Alessandra Capuano, et al.. (2024). Loss of the extracellular matrix glycoprotein EMILIN1 accelerates Δ16HER2-driven breast cancer initiation in mice. npj Breast Cancer. 10(1). 5–5. 6 indexed citations

Pellarin, Ilenia, Maura Sonego, Alessandra Dall’Acqua, et al.. (2020). Serum- and glucocorticoid- inducible kinase 2, SGK2, is a novel autophagy regulator and modulates platinum drugs response in cancer cells. Oncogene. 39(40). 6370–6386. 11 indexed citations

Pellarin, Ilenia, Alessandra Dall’Acqua, Sara D’Andrea, et al.. (2020). Splicing factor proline- and glutamine-rich (SFPQ) protein regulates platinum response in ovarian cancer-modulating SRSF2 activity. Oncogene. 39(22). 4390–4403. 47 indexed citations

Citron, Francesca, Ilenia Segatto, Gian Luca Rampioni Vinciguerra, et al.. (2019). Downregulation of miR-223 Expression Is an Early Event during Mammary Transformation and Confers Resistance to CDK4/6 Inhibitors in Luminal Breast Cancer. Cancer Research. 80(5). 1064–1077. 54 indexed citations

Marco, Carmela De, Floriana Forzati, Valentina Iovane, et al.. (2018). The T197A Knock-in Model of Cdkn1b Gene to Study the Effects of p27 Restoration In Vivo. Molecular Cancer Therapeutics. 18(2). 482–493. 5 indexed citations

Segatto, Ilenia, Mara De Marco Zompit, Francesca Citron, et al.. (2018). Stathmin Is Required for Normal Mouse Mammary Gland Development and Δ16HER2-Driven Tumorigenesis. Cancer Research. 79(2). 397–409. 15 indexed citations

Dall’Acqua, Alessandra, Maura Sonego, Ilenia Pellarin, et al.. (2017). CDK6 protects epithelial ovarian cancer from platinum‐induced death via FOXO3 regulation. EMBO Molecular Medicine. 9(10). 1415–1433. 67 indexed citations

Berton, Stefania, Ilenia Segatto, Francesca Citron, et al.. (2017). Loss of p27kip1 increases genomic instability and induces radio-resistance in luminal breast cancer cells. Scientific Reports. 7(1). 595–595. 21 indexed citations

Fabris, Linda, Stefania Berton, Francesca Citron, et al.. (2016). Radiotherapy-induced miR-223 prevents relapse of breast cancer by targeting the EGF pathway. Oncogene. 35(37). 4914–4926. 48 indexed citations

10.

Fabris, Linda, Stefania Berton, Ilenia Segatto, et al.. (2016). p27kip1 expression limits H-Ras-driven transformation and tumorigenesis by both canonical and non-canonical mechanisms. Oncotarget. 7(40). 64560–64574. 5 indexed citations

11.

Fabris, Linda, Stefania Berton, Ilenia Segatto, et al.. (2015). p27 ^kip1 controls H-Ras/MAPK activation and cell cycle entry via modulation of MT stability. Proceedings of the National Academy of Sciences. 112(45). 13916–13921. 39 indexed citations

12.

Berton, Stefania, Linda Fabris, Sara D’Andrea, et al.. (2014). Genetic characterization of p27^kip1and stathmin in controlling cell proliferation in vivo. Cell Cycle. 13(19). 3100–3111. 25 indexed citations

13.

Armenia, Joshua, Linda Fabris, Francesca Lovat, et al.. (2014). Contact inhibition modulates intracellular levels of miR-223 in a p27kip1-dependent manner. Oncotarget. 5(5). 1185–1197. 14 indexed citations

14.

D’Andrea, Sara, Stefania Berton, Ilenia Segatto, et al.. (2012). Stathmin Is Dispensable for Tumor Onset in Mice. PLoS ONE. 7(9). e45561–e45561. 9 indexed citations

15.

Belletti, Barbara, Stefania Berton, Linda Fabris, et al.. (2010). p27^kip1 Controls Cell Morphology and Motility by Regulating Microtubule-Dependent Lipid Raft Recycling. Molecular and Cellular Biology. 30(9). 2229–2240. 57 indexed citations

16.

Belletti, Barbara, Jayant S. Vaidya, Sara D’Andrea, et al.. (2008). Targeted Intraoperative Radiotherapy Impairs the Stimulation of Breast Cancer Cell Proliferation and Invasion Caused by Surgical Wounding. Clinical Cancer Research. 14(5). 1325–1332. 156 indexed citations

17.

Belletti, Barbara, Milena S. Nicoloso, Mónica Schiappacassi, et al.. (2008). Stathmin Activity Influences Sarcoma Cell Shape, Motility, and Metastatic Potential. Molecular Biology of the Cell. 19(5). 2003–2013. 115 indexed citations

18.

Biondi, Carla, Maria Enrica Ferretti, Laura Lunghi, et al.. (2008). Somatostatin as a Regulator of First-Trimester Human Trophoblast Functions. Placenta. 29(8). 660–670. 4 indexed citations

19.

Baldassarre, Gustavo, Barbara Belletti, Jayant S. Vaidya, et al.. (2007). Intraoperative radiotherapy (IORT) impairs surgical wound-stimulated breast cancer cell invasion. Journal of Clinical Oncology. 25(18_suppl). 21139–21139. 3 indexed citations

20.

Massarut, Samuele, Gustavo Baldassarre, Barbara Belletti, et al.. (2006). Intraoperative radiotherapy impairs breast cancer cell motility induced by surgical wound fluid. Journal of Clinical Oncology. 24(18_suppl). 10611–10611. 6 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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