Sara A. Mathie

1.8k total citations · 1 hit paper
13 papers, 1.4k citations indexed

About

Sara A. Mathie is a scholar working on Pulmonary and Respiratory Medicine, Physiology and Immunology. According to data from OpenAlex, Sara A. Mathie has authored 13 papers receiving a total of 1.4k indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Pulmonary and Respiratory Medicine, 8 papers in Physiology and 7 papers in Immunology. Recurrent topics in Sara A. Mathie's work include Asthma and respiratory diseases (8 papers), IL-33, ST2, and ILC Pathways (4 papers) and Neonatal Respiratory Health Research (4 papers). Sara A. Mathie is often cited by papers focused on Asthma and respiratory diseases (8 papers), IL-33, ST2, and ILC Pathways (4 papers) and Neonatal Respiratory Health Research (4 papers). Sara A. Mathie collaborates with scholars based in United Kingdom, Germany and Denmark. Sara A. Mathie's co-authors include Clare M. Lloyd, Lisa G. Gregory, Adam J. Byrne, Karen Buckland, Jennifer Kearley, Simone A. Walker, Andrew Bush, Sejal Saglani, Valerie B. O’Donnell and Carla P. Jones and has published in prestigious journals such as Journal of Biological Chemistry, Nature Immunology and American Journal of Respiratory and Critical Care Medicine.

In The Last Decade

Sara A. Mathie

13 papers receiving 1.4k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Pulmonary macrophages: key players in the innate defence of the airways

2015 365 citations Adam J. Byrne, Sara A. Mathie et al. Thorax profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Sara A. Mathie United Kingdom	13	779	567	445	284	233	13	1.4k
Mahesh Raundhal United States	12	545 0.7×	606 1.1×	262 0.6×	132 0.5×	206 0.9×	15	1.0k
Anupriya Khare United States	15	806 1.0×	550 1.0×	257 0.6×	143 0.5×	280 1.2×	18	1.4k
Yusuke Suzuki Japan	14	630 0.8×	582 1.0×	208 0.5×	309 1.1×	148 0.6×	39	1.1k
Susan Foley Canada	12	671 0.9×	903 1.6×	445 1.0×	347 1.2×	138 0.6×	18	1.4k
Taku Kodama United States	20	661 0.8×	634 1.1×	252 0.6×	133 0.5×	161 0.7×	28	1.2k
Fabrice Bureau Belgium	4	504 0.6×	346 0.6×	246 0.6×	166 0.6×	127 0.5×	5	868
Jyothi Nayar United States	14	609 0.8×	557 1.0×	285 0.6×	144 0.5×	451 1.9×	15	1.2k
Sally E. Wenzel United States	21	514 0.7×	1.1k 2.0×	846 1.9×	203 0.7×	296 1.3×	36	1.9k
Aaron M. Gibson United States	20	371 0.5×	441 0.8×	192 0.4×	148 0.5×	317 1.4×	32	1.3k
S. Létuvé France	16	439 0.6×	459 0.8×	342 0.8×	112 0.4×	386 1.7×	28	1.2k

Countries citing papers authored by Sara A. Mathie

Since Specialization

Citations

This map shows the geographic impact of Sara A. Mathie's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Sara A. Mathie with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Sara A. Mathie more than expected).

Fields of papers citing papers by Sara A. Mathie

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Sara A. Mathie. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Sara A. Mathie. The network helps show where Sara A. Mathie may publish in the future.

Co-authorship network of co-authors of Sara A. Mathie

This figure shows the co-authorship network connecting the top 25 collaborators of Sara A. Mathie. A scholar is included among the top collaborators of Sara A. Mathie based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Sara A. Mathie. Sara A. Mathie is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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13 of 13 papers shown

Khoyratty, Tariq, Zhichao Ai, Iván Ballesteros, et al.. (2021). Distinct transcription factor networks control neutrophil-driven inflammation. Nature Immunology. 22(9). 1093–1106. 107 indexed citations

Reynolds, Catherine J., Xiaoming Cheng, Fiyyaz Ahmed‐Jushuf, et al.. (2018). Lung Defense through IL-8 Carries a Cost of Chronic Lung Remodeling and Impaired Function. American Journal of Respiratory Cell and Molecular Biology. 59(5). 557–571. 45 indexed citations

Byrne, Adam J., Miriam Weiss, Sara A. Mathie, et al.. (2016). A critical role for IRF5 in regulating allergic airway inflammation. Mucosal Immunology. 10(3). 716–726. 31 indexed citations

Byrne, Adam J., Sara A. Mathie, Lisa G. Gregory, & Clare M. Lloyd. (2015). Pulmonary macrophages: key players in the innate defence of the airways. Thorax. 70(12). 1189–1196. 365 indexed citations breakdown →

Mathie, Sara A., Simone A. Walker, Victoria J. Tyrrell, et al.. (2014). Alveolar macrophages are sentinels of murine pulmonary homeostasis following inhaled antigen challenge. Allergy. 70(1). 80–89. 64 indexed citations

Saglani, Sejal, Nicola Ullmann, Gaynor A. Campbell, et al.. (2013). IL-33 promotes airway remodeling in pediatric patients with severe steroid-resistant asthma. Journal of Allergy and Clinical Immunology. 132(3). 676–685.e13. 198 indexed citations

Gregory, Lisa G., Carla P. Jones, Sara A. Mathie, Sophie Pégorier, & Clare M. Lloyd. (2013). Endothelin‐1 directs airway remodeling and hyper‐reactivity in a murine asthma model. Allergy. 68(12). 1579–1588. 25 indexed citations

Gregory, Lisa G., Sara A. Mathie, Simone A. Walker, et al.. (2010). Overexpression of Smad2 Drives House Dust Mite–mediated Airway Remodeling and Airway Hyperresponsiveness via Activin and IL-25. American Journal of Respiratory and Critical Care Medicine. 182(2). 143–154. 81 indexed citations

Kearley, Jennifer, Karen Buckland, Sara A. Mathie, & Clare M. Lloyd. (2009). Resolution of Allergic Inflammation and Airway Hyperreactivity Is Dependent upon Disruption of the T1/ST2–IL-33 Pathway. American Journal of Respiratory and Critical Care Medicine. 179(9). 772–781. 199 indexed citations

10.

Dioszeghy, Vincent, Benjamin H. Maskrey, Christopher P. Thomas, et al.. (2009). Phosphatidylethanolamine-esterified Eicosanoids in the Mouse. Journal of Biological Chemistry. 284(32). 21185–21191. 66 indexed citations

11.

Saglani, Sejal, Sara A. Mathie, Lisa G. Gregory, et al.. (2009). Pathophysiological Features of Asthma Develop in Parallel in House Dust Mite–Exposed Neonatal Mice. American Journal of Respiratory Cell and Molecular Biology. 41(3). 281–289. 76 indexed citations

12.

Gregory, Lisa G., Benjamin Causton, Jenna R. Murdoch, et al.. (2009). Inhaled house dust mite induces pulmonary T helper 2 cytokine production. Clinical & Experimental Allergy. 39(10). 1597–1610. 72 indexed citations

13.

Tourdot, Sophie, Sara A. Mathie, Tracy Hussell, et al.. (2008). Respiratory syncytial virus infection provokes airway remodelling in allergen‐exposed mice in absence of prior allergen sensitization. Clinical & Experimental Allergy. 38(6). 1016–1024. 41 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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