San‐e Ishikawa

4.7k total citations
177 papers, 3.3k citations indexed

About

San‐e Ishikawa is a scholar working on Pulmonary and Respiratory Medicine, Molecular Biology and Endocrinology, Diabetes and Metabolism. According to data from OpenAlex, San‐e Ishikawa has authored 177 papers receiving a total of 3.3k indexed citations (citations by other indexed papers that have themselves been cited), including 72 papers in Pulmonary and Respiratory Medicine, 65 papers in Molecular Biology and 47 papers in Endocrinology, Diabetes and Metabolism. Recurrent topics in San‐e Ishikawa's work include Electrolyte and hormonal disorders (63 papers), Ion Transport and Channel Regulation (44 papers) and Neuroendocrine regulation and behavior (44 papers). San‐e Ishikawa is often cited by papers focused on Electrolyte and hormonal disorders (63 papers), Ion Transport and Channel Regulation (44 papers) and Neuroendocrine regulation and behavior (44 papers). San‐e Ishikawa collaborates with scholars based in Japan, United States and Italy. San‐e Ishikawa's co-authors include Toshikazu Saito, Masanobu Kawakami, Robert W. Schrier, Koji Okada, Takako Saitô, Shoichiro Nagasaka, Hiroyuki Tamemoto, Ikuyo Kusaka, Tomoatsu Nakamura and Genro Fujisawa and has published in prestigious journals such as Journal of Clinical Investigation, Annals of Internal Medicine and The Journal of Clinical Endocrinology & Metabolism.

In The Last Decade

San‐e Ishikawa

171 papers receiving 3.1k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
San‐e Ishikawa Japan 32 1.3k 1.2k 731 613 558 177 3.3k
Hiroshi Nonoguchi Japan 42 2.5k 1.9× 1.4k 1.1× 551 0.8× 1.0k 1.7× 396 0.7× 144 5.2k
Rainer Rettig Germany 35 1.1k 0.8× 338 0.3× 1.4k 1.9× 1.5k 2.5× 487 0.9× 161 4.1k
Ole Skøtt Denmark 36 1.8k 1.3× 577 0.5× 976 1.3× 1.4k 2.3× 372 0.7× 118 3.9k
Fumiki Yoshihara Japan 35 1.2k 0.9× 664 0.5× 589 0.8× 1.9k 3.1× 578 1.0× 132 4.2k
Robert W. Schrier United States 24 650 0.5× 810 0.7× 174 0.2× 375 0.6× 468 0.8× 41 2.5k
Kazuo Kanno Japan 25 1.0k 0.8× 520 0.4× 428 0.6× 979 1.6× 294 0.5× 40 3.0k
Regien G. Schoemaker Netherlands 35 1.0k 0.8× 464 0.4× 286 0.4× 1.5k 2.5× 485 0.9× 108 4.8k
Allen W. Cowley United States 26 669 0.5× 378 0.3× 399 0.5× 690 1.1× 161 0.3× 56 2.2k
Fumio Niimura Japan 25 1.5k 1.1× 310 0.3× 1.2k 1.6× 1.5k 2.5× 347 0.6× 57 3.7k
H. Sonnenberg Canada 26 1.7k 1.3× 1.1k 0.9× 453 0.6× 2.7k 4.4× 326 0.6× 87 4.7k

Countries citing papers authored by San‐e Ishikawa

Since Specialization
Citations

This map shows the geographic impact of San‐e Ishikawa's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by San‐e Ishikawa with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites San‐e Ishikawa more than expected).

Fields of papers citing papers by San‐e Ishikawa

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by San‐e Ishikawa. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by San‐e Ishikawa. The network helps show where San‐e Ishikawa may publish in the future.

Co-authorship network of co-authors of San‐e Ishikawa

This figure shows the co-authorship network connecting the top 25 collaborators of San‐e Ishikawa. A scholar is included among the top collaborators of San‐e Ishikawa based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with San‐e Ishikawa. San‐e Ishikawa is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Ishikawa, San‐e & Hiroshi Funayama. (2023). Hyponatremia Associated with Congestive Heart Failure: Involvement of Vasopressin and Efficacy of Vasopressin Receptor Antagonists. Journal of Clinical Medicine. 12(4). 1482–1482. 5 indexed citations
2.
Yamada, Hodaka, Tomio Umemoto, Mikihiko Kawano, et al.. (2017). High-density lipoprotein and apolipoprotein A-I inhibit palmitate-induced translocation of toll-like receptor 4 into lipid rafts and inflammatory cytokines in 3T3-L1 adipocytes. Biochemical and Biophysical Research Communications. 484(2). 403–408. 12 indexed citations
3.
Murata, Miho, et al.. (2012). Endothelial impairment and bone marrow‐derived CD34+/133+ cells in diabetic patients with erectile dysfunction. Journal of Diabetes Investigation. 3(6). 526–533. 11 indexed citations
4.
Yoshioka, Toru, Hiroshi Funayama, Tomohiro Nakamura, et al.. (2010). Association of CD40 ligand levels in the culprit coronary arteries with subsequent prognosis of acute myocardial infarction. Atherosclerosis. 213(1). 268–272. 5 indexed citations
5.
Yoshida, Masashi, Masanori Nakata, Katsuya Dezaki, et al.. (2010). Voltage-dependent metabolic regulation of Kv2.1 channels in pancreatic β-cells. Biochemical and Biophysical Research Communications. 396(2). 304–309. 11 indexed citations
6.
Kakehashi, Akihiro, Yuka Saito, Hiroko Yamagami, et al.. (2006). Characteristics of diabetic retinopathy in SDT rats. Diabetes/Metabolism Research and Reviews. 22(6). 455–461. 44 indexed citations
7.
Ueba, Hiroto, Masatoshi Kuroki, Shigemasa Hashimoto, et al.. (2005). Glimepiride induces nitric oxide production in human coronary artery endothelial cells via a PI3-kinase-Akt dependent pathway. Atherosclerosis. 183(1). 35–39. 27 indexed citations
8.
9.
Saitô, Takako, Takahisa Kawano, Tomoyuki Saito, et al.. (2005). Elevation of serum adiponectin levels in Basedow disease. Metabolism. 54(11). 1461–1466. 33 indexed citations
10.
Kusaka, Ikuyo, Takako Saitô, Tomoatsu Nakamura, et al.. (2002). Urinary Excretion of Aquaporin-2 Water Channel in Diabetic Ketoacidosis. ˜The œNephron journals/Nephron journals. 91(1). 167–169. 3 indexed citations
11.
Saitô, Takako, et al.. (2001). Role of aquaporin-2 gene expression in hyponatremic rats with chronic vasopressin-induced antidiuresis. Kidney International. 60(4). 1266–1276. 31 indexed citations
12.
Saitô, Takako, et al.. (2001). Urinary excretion of the aquaporin‐2 water channel exaggerated in pathological states of impaired water excretion. Clinical Endocrinology. 55(2). 217–221. 6 indexed citations
13.
Radetti, Giorgio, et al.. (2001). Urinary aquaporin-2 excretion in nocturnal enuresis. European Journal of Endocrinology. 145(4). 435–438. 14 indexed citations
14.
Kinjo, Takao, et al.. (2000). Rhabdoid tumour of the lung is a dedifferentiated phenotype of pulmonary adenocarcinoma. Histopathology. 37(1). 37–44. 30 indexed citations
15.
Nagasaka, Shoichiro, San‐e Ishikawa, Tomoatsu Nakamura, et al.. (1998). Association of endogenous insulin secretion and mode of therapy with body fat and serum leptin levels in diabetic subjects. Metabolism. 47(11). 1391–1396. 19 indexed citations
16.
Tsuboi, Yasushi, et al.. (1994). In vivo diuretic effect of a new non-peptide arginine vasopressin antagonist, OPC-31260, in conscious rats. Journal of Endocrinology. 143(2). 227–234. 18 indexed citations
17.
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19.
Ishikawa, San‐e, et al.. (1984). Interrelationship between subpressor effects of vasopressin and other vasoactive hormones in the rat.. PubMed. 10(3). 184–9. 40 indexed citations
20.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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