Sanae Numata

810 total citations
21 papers, 495 citations indexed

About

Sanae Numata is a scholar working on Pathology and Forensic Medicine, Molecular Biology and Genetics. According to data from OpenAlex, Sanae Numata has authored 21 papers receiving a total of 495 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Pathology and Forensic Medicine, 6 papers in Molecular Biology and 6 papers in Genetics. Recurrent topics in Sanae Numata's work include Autoimmune Bullous Skin Diseases (7 papers), Platelet Disorders and Treatments (5 papers) and Coagulation, Bradykinin, Polyphosphates, and Angioedema (5 papers). Sanae Numata is often cited by papers focused on Autoimmune Bullous Skin Diseases (7 papers), Platelet Disorders and Treatments (5 papers) and Coagulation, Bradykinin, Polyphosphates, and Angioedema (5 papers). Sanae Numata collaborates with scholars based in Japan, Switzerland and France. Sanae Numata's co-authors include Kwesi Teye, Takashi Hashimoto, Norito Ishii, Daisuke Tsuruta, Tamihiro Kawakami, Hiroshi Koga, Atsunari Tsuchisaka, Ryosuke Sogame, Minao Furumura and T Hachiya and has published in prestigious journals such as PLoS ONE, Scientific Reports and Biochemical and Biophysical Research Communications.

In The Last Decade

Sanae Numata

20 papers receiving 489 citations

Peers

Sanae Numata
Yale Liu China
Bernd Lecher Germany
Linda Zhu United States
Vera Teixeira Portugal
Ashley M. Mahler United States
Susan C. Ridge United States
Sarah Swerdlow United States
Yale Liu China
Sanae Numata
Citations per year, relative to Sanae Numata Sanae Numata (= 1×) peers Yale Liu

Countries citing papers authored by Sanae Numata

Since Specialization
Citations

This map shows the geographic impact of Sanae Numata's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Sanae Numata with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Sanae Numata more than expected).

Fields of papers citing papers by Sanae Numata

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Sanae Numata. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Sanae Numata. The network helps show where Sanae Numata may publish in the future.

Co-authorship network of co-authors of Sanae Numata

This figure shows the co-authorship network connecting the top 25 collaborators of Sanae Numata. A scholar is included among the top collaborators of Sanae Numata based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Sanae Numata. Sanae Numata is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Teye, Kwesi, Hiroshi Koga, Takahiro Hamada, et al.. (2020). A Novel Microduplication Spanning Exons 8–16 of ATP2C1 That Was Undetectable by Standard Sanger Sequencing in a Japanese Patient With Hailey–Hailey Disease. Frontiers in Medicine. 7. 492–492. 1 indexed citations
2.
Teye, Kwesi, Koji Hashimoto, Sanae Numata, et al.. (2017). Multimerization is required for antigen binding activity of an engineered IgM/IgG chimeric antibody recognizing a skin-related antigen. Scientific Reports. 7(1). 8212–8212. 5 indexed citations
3.
Teye, Kwesi, Sanae Numata, Norito Ishii, et al.. (2016). Isolation of All CD44 Transcripts in Human Epidermis and Regulation of Their Expression by Various Agents. PLoS ONE. 11(8). e0160952–e0160952. 11 indexed citations
4.
Hashimoto, Takashi, Kwesi Teye, Sanae Numata, et al.. (2016). Detection of IgE autoantibodies to BP180 and BP230 and their relationship to clinical features in bullous pemphigoid. British Journal of Dermatology. 177(1). 141–151. 74 indexed citations
5.
Numata, Sanae, Takahiro Hamada, Shunpei Fukuda, et al.. (2015). Anti-laminin-γ1 Pemphigoid Developed in a Case of Autosomal Recessive Congenital Ichthyosis. Acta Dermato Venereologica. 95(1). 93–94. 4 indexed citations
6.
Yan, Yan, Minao Furumura, Kwesi Teye, et al.. (2015). Shikonin Promotes Skin Cell Proliferation and Inhibits Nuclear Factor-κB Translocation via Proteasome Inhibition In Vitro. Chinese Medical Journal. 128(16). 2228–2233. 31 indexed citations
7.
Tsuchisaka, Atsunari, Sanae Numata, Kwesi Teye, et al.. (2015). Epiplakin Is a Paraneoplastic Pemphigus Autoantigen and Related to Bronchiolitis Obliterans in Japanese Patients. Journal of Investigative Dermatology. 136(2). 399–408. 46 indexed citations
8.
Ishii, Norito, Kwesi Teye, Rie Uehara, et al.. (2015). Anti-desmocollin autoantibodies in nonclassical pemphigus. British Journal of Dermatology. 173(1). 59–68. 64 indexed citations
9.
Sogame, Ryosuke, Kwesi Teye, Atsunari Tsuchisaka, et al.. (2015). Clinical and immunological findings in 104 cases of paraneoplastic pemphigus. British Journal of Dermatology. 173(6). 1447–1452. 107 indexed citations
10.
Teye, Kwesi, Takahiro Hamada, Sanae Numata, et al.. (2014). Homozygous deletion of six genes including corneodesmosin on chromosome 6p21.3 is associated with generalized peeling skin disease. Journal of Dermatological Science. 75(1). 36–42. 12 indexed citations
11.
Numata, Sanae, Kwesi Teye, Daisuke Tsuruta, et al.. (2013). Anti-Alpha-2-Macroglobulin-Like-1 Autoantibodies Are Detected Frequently and May Be Pathogenic in Paraneoplastic Pemphigus. Journal of Investigative Dermatology. 133(7). 1785–1793. 38 indexed citations
12.
Teye, Kwesi, Norito Ishii, Hiroshi Koga, et al.. (2012). Newly developed IgA ELISAs of eukaryotic recombinant proteins of desmocollins 1-3 are more sensitive and specific than baculoprotein ELISAs. Journal of Dermatological Science. 69(2). e8–e9. 1 indexed citations
13.
Hamada, Takahiro, Daisuke Tsuruta, Shunpei Fukuda, et al.. (2012). How do keratinizing disorders and blistering disorders overlap?. Experimental Dermatology. 22(2). 83–87. 25 indexed citations
14.
Numata, Sanae, Yoshiro Koda, Kenji Ihara, et al.. (2009). Mutant alleles associated with late-onset ornithine transcarbamylase deficiency in male patients have recurrently arisen and have been retained in some populations. Journal of Human Genetics. 55(1). 18–22. 5 indexed citations
15.
Yoshino, Makoto, Yoriko Watanabe, Yasuyuki Tokunaga, et al.. (2007). Roles of specific cytokines in bone remodeling and hematopoiesis in Gaucher disease. Pediatrics International. 49(6). 959–965. 28 indexed citations
16.
Numata, Sanae, Yasuki Maeno, Yoriko Watanabe, et al.. (2007). Paternal transmission and slow elimination of mutant alleles associated with late-onset ornithine transcarbamylase deficiency in male patients. Journal of Human Genetics. 53(1). 10–17. 8 indexed citations
18.
Nakano, Yuko, et al.. (2004). Adenovirus type 11 infected cells of two patients of ATL (Adult T cell leukemia) treated by stem cell transplant. The Journal of the Japanese Society of Clinical Cytology. 43(3). 178–184.
19.
Numata, Sanae, et al.. (1994). Phosphorylation of Rabphilin-3A, a Putative Target Protein for Rab3A, by Cyclic AMP-Dependent Protein Kinase. Biochemical and Biophysical Research Communications. 203(3). 1927–1934. 23 indexed citations
20.
Miyoshi, Keiko, et al.. (1981). [B-lymphocyte leukemia. B-lymphocytosis and immunoglobulins].. PubMed. 39(8). 2806–14. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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