Samuel I. Bloom

1.0k total citations · 1 hit paper
25 papers, 713 citations indexed

About

Samuel I. Bloom is a scholar working on Physiology, Immunology and Molecular Biology. According to data from OpenAlex, Samuel I. Bloom has authored 25 papers receiving a total of 713 indexed citations (citations by other indexed papers that have themselves been cited), including 15 papers in Physiology, 9 papers in Immunology and 6 papers in Molecular Biology. Recurrent topics in Samuel I. Bloom's work include Telomeres, Telomerase, and Senescence (13 papers), Neutrophil, Myeloperoxidase and Oxidative Mechanisms (8 papers) and Circadian rhythm and melatonin (4 papers). Samuel I. Bloom is often cited by papers focused on Telomeres, Telomerase, and Senescence (13 papers), Neutrophil, Myeloperoxidase and Oxidative Mechanisms (8 papers) and Circadian rhythm and melatonin (4 papers). Samuel I. Bloom collaborates with scholars based in United States, China and Netherlands. Samuel I. Bloom's co-authors include Anthony J. Donato, Lisa A. Lesniewski, Md Torikul Islam, Yu Liu, Daniel R. Machin, Tam T. T. Phuong, Phillip E. Gates, Matthew T. Rondina, Robert A. Campbell and Raphael M. Franzini and has published in prestigious journals such as Journal of the American Chemical Society, The Journal of Physiology and The FASEB Journal.

In The Last Decade

Samuel I. Bloom

23 papers receiving 704 citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Mechanisms and consequences of endothelial cell senescence

2022 267 citations Samuel I. Bloom, Md Torikul Islam et al. Nature Reviews Cardiology profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Samuel I. Bloom United States	12	263	225	137	81	71	25	713
Panagiotis Vasileiou Greece	15	265 1.0×	213 0.9×	97 0.7×	243 3.0×	133 1.9×	31	902
Damian Klóska Poland	15	430 1.6×	62 0.3×	80 0.6×	36 0.4×	54 0.8×	28	697
Yinchuan Xu China	20	477 1.8×	115 0.5×	91 0.7×	188 2.3×	188 2.6×	32	1.0k
Andrey Lozhkin United States	13	229 0.9×	132 0.6×	149 1.1×	138 1.7×	100 1.4×	27	665
Filip Sedlić United States	17	499 1.9×	107 0.5×	35 0.3×	96 1.2×	63 0.9×	30	922
Xiangquan Kong China	15	179 0.7×	69 0.3×	92 0.7×	119 1.5×	56 0.8×	36	546
Laura Braud France	12	278 1.1×	133 0.6×	124 0.9×	49 0.6×	130 1.8×	18	696
Calum Forteath United Kingdom	5	331 1.3×	121 0.5×	71 0.5×	58 0.7×	108 1.5×	7	657
Lefeng Wang China	16	208 0.8×	50 0.2×	93 0.7×	187 2.3×	96 1.4×	52	746
Aleksandra Kopacz Poland	13	348 1.3×	51 0.2×	64 0.5×	27 0.3×	41 0.6×	22	543

Countries citing papers authored by Samuel I. Bloom

Since Specialization

Citations

This map shows the geographic impact of Samuel I. Bloom's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Samuel I. Bloom with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Samuel I. Bloom more than expected).

Fields of papers citing papers by Samuel I. Bloom

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Samuel I. Bloom. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Samuel I. Bloom. The network helps show where Samuel I. Bloom may publish in the future.

Co-authorship network of co-authors of Samuel I. Bloom

This figure shows the co-authorship network connecting the top 25 collaborators of Samuel I. Bloom. A scholar is included among the top collaborators of Samuel I. Bloom based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Samuel I. Bloom. Samuel I. Bloom is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Bloom, Samuel I., et al.. (2025). Senolytic therapy increases replicative capacity by eliminating senescent endothelial cells. Experimental Gerontology. 211. 112885–112885.

Mahoney, Sophia, Samuel I. Bloom, Douglas R. Seals, et al.. (2025). Mechanisms of cellular senescence-induced vascular aging: evidence of senotherapeutic strategies. PubMed. 5(1). 1 indexed citations

Bloom, Samuel I., et al.. (2024). MitoQ Treatment Mitigates Senescence Burden in DOXO-Induced Senescent Endothelial Cells Through Reductions in mtROS and DNA Damage. Physiology. 39(S1). 1 indexed citations

Islam, Md Torikul, Jinjin Cai, Samuel I. Bloom, et al.. (2024). Endothelial-Specific Reduction in Arf6 Impairs Insulin-Stimulated Vasodilation and Skeletal Muscle Blood Flow Resulting in Systemic Insulin Resistance in Mice. Arteriosclerosis Thrombosis and Vascular Biology. 44(5). 1101–1113. 4 indexed citations

Bloom, Samuel I., Md Torikul Islam, Daniel R. Machin, et al.. (2023). Endothelial cell telomere dysfunction induces senescence and results in vascular and metabolic impairments. Aging Cell. 22(8). e13875–e13875. 37 indexed citations

Bloom, Samuel I., et al.. (2023). Senolytics reduce endothelial cell DNA damage and telomere dysfunction in old age. Physiology. 38(S1). 1 indexed citations

Donato, Anthony J., et al.. (2023). Analysis of Somatic Mutations in Senescent Cells Using Single-Cell Whole-Genome Sequencing. PubMed. 1(1). 20230007–20230007.

Machin, Daniel R., Daniel W. Trott, Md Torikul Islam, et al.. (2023). Glycocalyx-targeted therapy ameliorates age-related arterial dysfunction. GeroScience. 45(4). 2351–2365. 17 indexed citations

Bloom, Samuel I., et al.. (2023). Reduction of double-strand DNA break repair exacerbates vascular aging. Aging. 15(19). 9913–9947. 3 indexed citations

10.

Bloom, Samuel I., et al.. (2022). Aging results in DNA damage and telomere dysfunction that is greater in endothelial versus vascular smooth muscle cells and is exacerbated in atheroprone regions. GeroScience. 44(6). 2741–2755. 23 indexed citations

11.

Bloom, Samuel I., Yu Liu, Daniel R. Machin, et al.. (2022). Aging results in endothelial cell telomere uncapping that induces senescence and physiological dysfunction. The FASEB Journal. 36(S1). 1 indexed citations

12.

Bloom, Samuel I., Md Torikul Islam, Lisa A. Lesniewski, & Anthony J. Donato. (2022). Mechanisms and consequences of endothelial cell senescence. Nature Reviews Cardiology. 20(1). 38–51. 267 indexed citations breakdown →

13.

Yuen, Lik Hang, Yu Liu, Samuel I. Bloom, et al.. (2021). Correction to “A Focused DNA-Encoded Chemical Library for the Discovery of Inhibitors of NAD⁺-Dependent Enzymes”. Journal of the American Chemical Society. 143(29). 11272–11273. 2 indexed citations

14.

Bloom, Samuel I., et al.. (2020). High‐fat diet induced obesity and age influence the telomere shelterin complex and telomerase gene expression in mouse adipose tissue. Physiological Reports. 8(11). e14461–e14461. 14 indexed citations

15.

Smith, Denise L., Samuel I. Bloom, Brandt D. Pence, et al.. (2019). Firefighting Induces Acute Inflammatory Responses that are not Relieved by Aspirin in Older Firefighters. Journal of Occupational and Environmental Medicine. 61(7). 617–622. 10 indexed citations

16.

Yuen, Lik Hang, Yu Liu, Samuel I. Bloom, et al.. (2019). A Focused DNA-Encoded Chemical Library for the Discovery of Inhibitors of NAD⁺-Dependent Enzymes. Journal of the American Chemical Society. 141(13). 5169–5181. 92 indexed citations

17.

Liu, Yu, Samuel I. Bloom, & Anthony J. Donato. (2018). The role of senescence, telomere dysfunction and shelterin in vascular aging. Microcirculation. 26(2). e12487–e12487. 54 indexed citations

18.

Morgan, R. Garrett, Ashley E. Walker, Daniel W. Trott, et al.. (2018). Induced Trf2 deletion leads to aging vascular phenotype in mice associated with arterial telomere uncapping, senescence signaling, and oxidative stress. Journal of Molecular and Cellular Cardiology. 127. 74–82. 25 indexed citations

19.

Frech, Tracy, Daniel R. Machin, Maureen A. Murtaugh, et al.. (2018). Implications of endothelial shear stress on systemic sclerosis vasculopathy and treatment. Clinical and Experimental Rheumatology. 36 Suppl 113(4). 175–182. 5 indexed citations

20.

Ives, Stephen J., et al.. (2017). Effects of a combined protein and antioxidant supplement on recovery of muscle function and soreness following eccentric exercise. Journal of the International Society of Sports Nutrition. 14(1). 21–21. 24 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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