Samuel Flashner
Impact in
-
- Cancer Cells and Metastasis
Papers in
- Oncology 10
- Cancer Cells and Metastasis 7
- Pancreatic and Hepatic Oncology Research 3
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- DNA Repair Mechanisms 2
- Epigenetics and DNA Methylation 2
- Co-authors
- Hiroshi Nakagawa (10 shared papers)Kate Beishline (2 shared papers)Jane Azizkhan‐Clifford (3 shared papers)Masataka Shimonosono (4 shared papers)Joel Gabre (5 shared papers)Alfred D. Doyle (1 shared paper)Matthew A. Rank (1 shared paper)Hirohito Kita (1 shared paper)
- Journals
- Biomolecules (2 papers)Carcinogenesis (1 paper)The American Journal of Gastroenterology (1 paper)Cancer Letters (1 paper)Cell Reports (1 paper)
- Partner nations
- United StatesJapanCanada
In The Last Decade
Samuel Flashner
14 papers receiving 204 citations
Peers
Comparison fields: 5 of 57
- Oncology 61
- Cancer Research 28
- Dermatology 14
- Immunology and Allergy 8
- Surgery 52
Countries citing papers authored by Samuel Flashner
This map shows the geographic impact of Samuel Flashner's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Samuel Flashner with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Samuel Flashner more than expected).
Fields of papers citing papers by Samuel Flashner
This network shows the impact of papers produced by Samuel Flashner. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Samuel Flashner. The network helps show where Samuel Flashner may publish in the future.
Co-authors
The 25 scholars most cited alongside Samuel Flashner, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2022 | 64 | |
| 2 | 2021 | 34 | |
| 3 | 2021 | 33 | |
| 4 | 2017 | 22 | |
| 5 | 2021 | 10 | |
| 6 | 2021 | 10 | |
| 7 | 2023 | 9 | |
| 8 | 2023 | 8 | |
| 9 | 2025 | 4 | |
| 10 | 2022 | 4 | |
| 11 | 2024 | 2 | |
| 12 | 2022 | 2 | |
| 13 | 2023 | 2 | |
| 14 | 2024 | 1 | |
| 15 | 2025 | 0 |
About Samuel Flashner
Samuel Flashner is a scholar working on Oncology, Molecular Biology, Surgery, Cancer Research and Pathology and Forensic Medicine, having authored 15 papers that have together received 205 indexed citations. Recurring topics across this work include Cancer Cells and Metastasis (7 papers), Pancreatic and Hepatic Oncology Research (3 papers), Cancer Genomics and Diagnostics (3 papers), Esophageal Cancer Research and Treatment (3 papers), DNA Repair Mechanisms (2 papers), Epigenetics and DNA Methylation (2 papers), Tumors and Oncological Cases (2 papers) and Neonatal Respiratory Health Research (2 papers). The work is most often cited by research in Oncology (61 citations), Cancer Research (28 citations), Dermatology (14 citations), Immunology and Allergy (8 citations) and Surgery (52 citations). Samuel Flashner has collaborated with scholars based in United States, Japan and Canada. Frequent co-authors include Hiroshi Nakagawa, Kate Beishline, Jane Azizkhan‐Clifford, Masataka Shimonosono, Joel Gabre, Alfred D. Doyle, Matthew A. Rank, Hirohito Kita, Ricardo Cruz‐Acuña and Benjamin L. Wright. Their work appears in journals such as Biomolecules, Carcinogenesis, The American Journal of Gastroenterology, Cancer Letters and Cell Reports.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.