Samuel D. Reyes

444 total citations
9 papers, 291 citations indexed

About

Samuel D. Reyes is a scholar working on Developmental Neuroscience, Molecular Biology and Cellular and Molecular Neuroscience. According to data from OpenAlex, Samuel D. Reyes has authored 9 papers receiving a total of 291 indexed citations (citations by other indexed papers that have themselves been cited), including 5 papers in Developmental Neuroscience, 4 papers in Molecular Biology and 3 papers in Cellular and Molecular Neuroscience. Recurrent topics in Samuel D. Reyes's work include Neurogenesis and neuroplasticity mechanisms (5 papers), HIV Research and Treatment (2 papers) and RNA regulation and disease (2 papers). Samuel D. Reyes is often cited by papers focused on Neurogenesis and neuroplasticity mechanisms (5 papers), HIV Research and Treatment (2 papers) and RNA regulation and disease (2 papers). Samuel D. Reyes collaborates with scholars based in United States, Canada and Australia. Samuel D. Reyes's co-authors include Anthony T. Campagnoni, Robin S. Fisher, Erin C. Jacobs, Kathy Kampf, Celia W. Campagnoni, Vance Handley, Yuanyun Xie, Carol Readhead, William H. Yong and Russell E. Jacobs and has published in prestigious journals such as Journal of Neuroscience, Scientific Reports and Frontiers in Immunology.

In The Last Decade

Samuel D. Reyes

9 papers receiving 290 citations

Peers

Samuel D. Reyes

CMN

DN

MB

BE

RNMI

André W. Phillips United States

Lieven Huang United Kingdom

Kevin Eade United States

A. Luzhynskaya United Kingdom

Marie‐Pierre Morel France

Martin Belle France

Jin‐Yong Park South Korea

Yasufumi Hayano Japan

Claudia Karl Germany

Abderrahman El Maarouf United States

André W. Phillips United States

Samuel D. Reyes

88 ×0.7

CMN

81 ×0.7

DN

174 ×1.7

MB

47 ×0.9

BE

9 ×0.2

RNMI

Citations per year, relative to Samuel D. Reyes Samuel D. Reyes (= 1×) peers André W. Phillips

Countries citing papers authored by Samuel D. Reyes

Since Specialization

Citations

This map shows the geographic impact of Samuel D. Reyes's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Samuel D. Reyes with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Samuel D. Reyes more than expected).

Fields of papers citing papers by Samuel D. Reyes

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Samuel D. Reyes. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Samuel D. Reyes. The network helps show where Samuel D. Reyes may publish in the future.

Co-authorship network of co-authors of Samuel D. Reyes

This figure shows the co-authorship network connecting the top 25 collaborators of Samuel D. Reyes. A scholar is included among the top collaborators of Samuel D. Reyes based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Samuel D. Reyes. Samuel D. Reyes is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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9 of 9 papers shown

1.

Stern, John M., Norman M. Spivak, Sergio Becerra, et al.. (2021). Safety of focused ultrasound neuromodulation in humans with temporal lobe epilepsy. Brain stimulation. 14(4). 1022–1031. 63 indexed citations

2.

Chang, Julia W., Samuel D. Reyes, Sandi Lam, et al.. (2021). Clonally Focused Public and Private T Cells in Resected Brain Tissue From Surgeries to Treat Children With Intractable Seizures. Frontiers in Immunology. 12. 664344–664344. 10 indexed citations

3.

Fallah, Aria, H. Westley Phillips, Xavier Michalet, et al.. (2020). Novel tonometer device distinguishes brain stiffness in epilepsy surgery. Scientific Reports. 10(1). 20978–20978. 5 indexed citations

4.

Owens, Geoffrey C., Alejandro J. Garcia, Aaron Mochizuki, et al.. (2019). Evidence for Innate and Adaptive Immune Responses in a Cohort of Intractable Pediatric Epilepsy Surgery Patients. Frontiers in Immunology. 10. 121–121. 18 indexed citations

5.

Jacobs, Erin C., Samuel D. Reyes, Celia W. Campagnoni, et al.. (2009). Targeted Overexpression of a Golli-Myelin Basic Protein Isoform to Oligodendrocytes Results in Aberrant Oligodendrocyte Maturation and Myelination. ASN NEURO. 1(4). 15 indexed citations

6.

Jacobs, Erin C., Celia W. Campagnoni, Kathy Kampf, et al.. (2007). Visualization of corticofugal projections during early cortical development in a τ‐GFP‐transgenic mouse. European Journal of Neuroscience. 25(1). 17–30. 97 indexed citations

7.

Martin, Melanie, Samuel D. Reyes, Maria I. Givogri, et al.. (2006). T2-weighted μMRI and Evoked Potential of the Visual System Measurements During the Development of Hypomyelinated Transgenic Mice. Neurochemical Research. 32(2). 159–165. 10 indexed citations

8.

Jacobs, Erin C., T Pribýl, Kathy Kampf, et al.. (2005). Region-Specific Myelin Pathology in Mice Lacking the Golli Products of the Myelin Basic Protein Gene. Journal of Neuroscience. 25(30). 7004–7013. 42 indexed citations

9.

Reyes, Samuel D. & Anthony T. Campagnoni. (2002). Two separate domains in the golli myelin basic proteins are responsible for nuclear targeting and process extension in transfected cells. Journal of Neuroscience Research. 69(5). 587–596. 31 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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