Sam N. Pennington

1.5k total citations
87 papers, 1.3k citations indexed

About

Sam N. Pennington is a scholar working on Molecular Biology, Pediatrics, Perinatology and Child Health and Biochemistry. According to data from OpenAlex, Sam N. Pennington has authored 87 papers receiving a total of 1.3k indexed citations (citations by other indexed papers that have themselves been cited), including 26 papers in Molecular Biology, 25 papers in Pediatrics, Perinatology and Child Health and 18 papers in Biochemistry. Recurrent topics in Sam N. Pennington's work include Birth, Development, and Health (21 papers), Prenatal Substance Exposure Effects (19 papers) and Peroxisome Proliferator-Activated Receptors (12 papers). Sam N. Pennington is often cited by papers focused on Birth, Development, and Health (21 papers), Prenatal Substance Exposure Effects (19 papers) and Peroxisome Proliferator-Activated Receptors (12 papers). Sam N. Pennington collaborates with scholars based in United States and Canada. Sam N. Pennington's co-authors include Ivan A. Shibley, Carlton Smith, Gerhard W. Kalmus, J. S. Pennington, R.D. Myers, Amy K. Roscoe, M.F. Lankford, F. Melinda Carver, Harry D. Brown and Larry W. Means and has published in prestigious journals such as Analytical Chemistry, Analytical Biochemistry and Biochemical and Biophysical Research Communications.

In The Last Decade

Sam N. Pennington

85 papers receiving 1.2k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Sam N. Pennington United States 21 405 402 169 156 139 87 1.3k
H.‐Ch. Curtius Switzerland 28 227 0.6× 849 2.1× 110 0.7× 268 1.7× 289 2.1× 84 1.8k
Frank L. Siegel United States 25 154 0.4× 1.0k 2.6× 218 1.3× 116 0.7× 188 1.4× 73 1.7k
P.-M. Sinet France 18 136 0.3× 532 1.3× 173 1.0× 127 0.8× 249 1.8× 27 1.4k
Ephraim Y. Levin United States 17 387 1.0× 675 1.7× 99 0.6× 69 0.4× 132 0.9× 25 1.3k
Dennis M. Delfert United States 8 97 0.2× 418 1.0× 137 0.8× 61 0.4× 128 0.9× 10 1.1k
Zbigniew Binienda United States 26 194 0.5× 777 1.9× 492 2.9× 62 0.4× 305 2.2× 72 1.9k
Frank Welsch United States 22 282 0.7× 406 1.0× 129 0.8× 29 0.2× 68 0.5× 88 1.9k
J.B. Jepson United Kingdom 13 60 0.1× 518 1.3× 145 0.9× 190 1.2× 148 1.1× 30 1.4k
Jadwiga Bryła Poland 19 84 0.2× 697 1.7× 93 0.6× 269 1.7× 423 3.0× 66 1.5k
H. Herken Germany 20 80 0.2× 521 1.3× 227 1.3× 242 1.6× 199 1.4× 134 1.3k

Countries citing papers authored by Sam N. Pennington

Since Specialization
Citations

This map shows the geographic impact of Sam N. Pennington's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Sam N. Pennington with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Sam N. Pennington more than expected).

Fields of papers citing papers by Sam N. Pennington

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Sam N. Pennington. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Sam N. Pennington. The network helps show where Sam N. Pennington may publish in the future.

Co-authorship network of co-authors of Sam N. Pennington

This figure shows the co-authorship network connecting the top 25 collaborators of Sam N. Pennington. A scholar is included among the top collaborators of Sam N. Pennington based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Sam N. Pennington. Sam N. Pennington is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Shibley, Ivan A., et al.. (1999). Experimental models used to measure direct and indirect ethanol teratogenicity.. Alcohol and Alcoholism. 34(2). 125–140. 12 indexed citations
2.
Shibley, Ivan A. & Sam N. Pennington. (1997). METABOLIC AND MITOTIC CHANGES ASSOCIATED WITH THE FETAL ALCOHOL SYNDROME. Alcohol and Alcoholism. 32(4). 423–434. 30 indexed citations
3.
Shibley, Ivan A., et al.. (1997). Changes in brain glucose levels and glucose transporter protein isoforms in alcohol- or nicotine-treated chick embryos. Developmental Brain Research. 103(1). 59–65. 20 indexed citations
4.
Nagy, Laura E. & Sam N. Pennington. (1996). Alcohol and Tyrosine Kinase/Phosphatase Mediated Processes. Alcoholism Clinical and Experimental Research. 20(s8). 106A–108A. 10 indexed citations
5.
Shibley, Ivan A. & Sam N. Pennington. (1995). Signaling Pathways Regulating Ornithine Decarboxylase Activity in the Embryonic Chicken. Neonatology. 67(6). 441–449. 7 indexed citations
6.
Pennington, Sam N., et al.. (1995). Insulin Signaling in Chick Embryos Exposed to Alcohol. Alcoholism Clinical and Experimental Research. 19(3). 701–707. 8 indexed citations
7.
Shibley, Ivan A., et al.. (1995). Ethanol's Effect on Tissue Polyamines and Ornithine Decarboxylase Activity: A Concise Review. Alcoholism Clinical and Experimental Research. 19(1). 209–215. 24 indexed citations
8.
Smith, Lucille L., et al.. (1993). Increases in Plasma Prostaglandin E2After Eccentric Exercise. Hormone and Metabolic Research. 25(8). 451–452. 16 indexed citations
9.
Sandstrom, Paul, et al.. (1993). Ethanol-induced insulin resistance suppresses the expression of embryonic ornithine decarboxylase activity. Alcohol. 10(4). 303–310. 12 indexed citations
10.
Pennington, Sam N., et al.. (1993). Embryonic Growth Inhibition Induced by Cocaine Is Associated with the Suppression of Ornithine Decarboxylase Activity. Experimental Biology and Medicine. 202(4). 491–498. 6 indexed citations
11.
Smith, Carlton, et al.. (1992). Effect of cocaine, ethanol or nicotine on ornithine decarboxylase activity in early chick embryo brain. Developmental Brain Research. 69(1). 51–57. 17 indexed citations
12.
Lankford, M.F., Amy K. Roscoe, Sam N. Pennington, & R.D. Myers. (1991). Drinking of high concentrations of ethanol versus palatable fluids in alcohol-preferring (P) rats: valid animal model of alcoholism. Alcohol. 8(4). 293–299. 112 indexed citations
13.
Smith, Carlton, Benjamin R. King, & Sam N. Pennington. (1991). Cyclic AMP-dependent protein kinase activity in the brains of alcohol-preferring (P) and nonpreferring (NP) rats. Alcohol. 8(5). 329–332. 4 indexed citations
14.
Smith, Carlton, et al.. (1990). Genetically Determined Alcohol Preference and Cyclic AMP Binding Proteins in Mouse Brain. Alcoholism Clinical and Experimental Research. 14(2). 158–164. 3 indexed citations
15.
Pennington, Sam N.. (1990). Molecular Changes Associated with Ethanol‐Induced Growth Suppression in the Chick Embryo. Alcoholism Clinical and Experimental Research. 14(6). 832–837. 39 indexed citations
16.
Means, Larry W., et al.. (1989). Embryonic ethanol exposure impairs detour learning in chicks. Alcohol. 6(4). 327–330. 23 indexed citations
17.
Pennington, Sam N.. (1987). Brain growth during ethanol-induced hypoplasia. Drug and Alcohol Dependence. 20(3). 279–286. 29 indexed citations
18.
Pennington, Sam N., et al.. (1984). Brain prostaglandins and free choice ethanol consumption. Alcohol. 1(2). 170–170. 3 indexed citations
19.
Pennington, Sam N., et al.. (1982). Ethanol-induced changes in the oxidative metabolism of arachidonic acid. Prostaglandins Leukotrienes and Medicine. 9(2). 151–157. 16 indexed citations
20.
Pennington, Sam N.. (1968). Determination of oxidizing agents by gas chromatography-iodometry. Journal of Chromatography A. 36(3). 400–401. 2 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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