Saghir Akhtar

6.8k total citations
137 papers, 5.5k citations indexed

About

Saghir Akhtar is a scholar working on Molecular Biology, Cardiology and Cardiovascular Medicine and Endocrinology, Diabetes and Metabolism. According to data from OpenAlex, Saghir Akhtar has authored 137 papers receiving a total of 5.5k indexed citations (citations by other indexed papers that have themselves been cited), including 87 papers in Molecular Biology, 24 papers in Cardiology and Cardiovascular Medicine and 17 papers in Endocrinology, Diabetes and Metabolism. Recurrent topics in Saghir Akhtar's work include RNA Interference and Gene Delivery (53 papers), Advanced biosensing and bioanalysis techniques (38 papers) and DNA and Nucleic Acid Chemistry (24 papers). Saghir Akhtar is often cited by papers focused on RNA Interference and Gene Delivery (53 papers), Advanced biosensing and bioanalysis techniques (38 papers) and DNA and Nucleic Acid Chemistry (24 papers). Saghir Akhtar collaborates with scholars based in United Kingdom, Kuwait and Qatar. Saghir Akhtar's co-authors include Ibrahim F. Benter, R. L. Juliano, Colin W. Pouton, Mariam H.M. Yousif, Andrew J. Hollins, Yadollah Omidi, Sudhir Agrawal, Mustapha Benboubetra, Ryszard Kole and W.J. Irwin and has published in prestigious journals such as Nucleic Acids Research, Journal of Clinical Investigation and Nature Medicine.

In The Last Decade

Saghir Akhtar

136 papers receiving 5.3k citations

Peers

Saghir Akhtar
Peter M. Kang United States
Jun Wu China
Ming Zhao China
Lei Cai China
Pankaj Gupta United States
Sun Hwa Kim South Korea
Peter M. Kang United States
Saghir Akhtar
Citations per year, relative to Saghir Akhtar Saghir Akhtar (= 1×) peers Peter M. Kang

Countries citing papers authored by Saghir Akhtar

Since Specialization
Citations

This map shows the geographic impact of Saghir Akhtar's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Saghir Akhtar with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Saghir Akhtar more than expected).

Fields of papers citing papers by Saghir Akhtar

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Saghir Akhtar. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Saghir Akhtar. The network helps show where Saghir Akhtar may publish in the future.

Co-authorship network of co-authors of Saghir Akhtar

This figure shows the co-authorship network connecting the top 25 collaborators of Saghir Akhtar. A scholar is included among the top collaborators of Saghir Akhtar based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Saghir Akhtar. Saghir Akhtar is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Akhtar, Saghir, et al.. (2025). Recent advances in stem cell-based therapies for type 1 diabetes: A glimpse into the future. Biomolecules and Biomedicine. 26(1). 5–23. 2 indexed citations
2.
Akhtar, Saghir, et al.. (2024). The potential role of cyclosporine A in cancer treatment: a comprehensive literature review. Współczesna Onkologia. 28(4). 271–282. 1 indexed citations
3.
Rachid, Ousama, et al.. (2021). Emerging innate biological properties of nano-drug delivery systems: A focus on PAMAM dendrimers and their clinical potential. Advanced Drug Delivery Reviews. 178. 113908–113908. 105 indexed citations
4.
Benter, Ibrahim F., et al.. (2021). The Role of Epidermal Growth Factor Receptor Family of Receptor Tyrosine Kinases in Mediating Diabetes-Induced Cardiovascular Complications. Frontiers in Pharmacology. 12. 701390–701390. 26 indexed citations
5.
Babiker, Fawzi, Ibrahim F. Benter, & Saghir Akhtar. (2020). <p>Nanotoxicology of Dendrimers in the Mammalian Heart: ex vivo and in vivo Administration of G6 PAMAM Nanoparticles Impairs Recovery of Cardiac Function Following Ischemia-Reperfusion Injury</p>. International Journal of Nanomedicine. Volume 15. 4393–4405. 10 indexed citations
6.
Akhtar, Saghir, Ibrahim F. Benter, Mohammed Danjuma, et al.. (2020). Pharmacotherapy in COVID-19 patients: a review of ACE2-raising drugs and their clinical safety. Journal of drug targeting. 28(7-8). 683–699. 36 indexed citations
7.
Akhtar, Saghir, Semir Vranić, Farhan Cyprian, & Ala‐Eddin Al Moustafa. (2018). Epstein–Barr Virus in Gliomas: Cause, Association, or Artifact?. Frontiers in Oncology. 8. 123–123. 36 indexed citations
8.
Dhaunsi, Gursev S., et al.. (2017). Angiotensin-(1-7) Downregulates Diabetes-Induced cGMP Phosphodiesterase Activation in Rat Corpus Cavernosum. BioMed Research International. 2017. 1–7. 7 indexed citations
11.
El‐Hashim, Ahmed Z., et al.. (2012). Angiotensin‐(1–7) inhibits allergic inflammation, via the MAS1 receptor, through suppression of ERK1/2‐ and NF‐κB‐dependent pathways. British Journal of Pharmacology. 166(6). 1964–1976. 129 indexed citations
12.
Akhtar, Saeed, Turki Almubrad, Anthony J. Bron, et al.. (2009). Role of epidermal growth factor receptor (EGFR) in corneal remodelling in diabetes. Acta Ophthalmologica. 87(8). 881–889. 17 indexed citations
13.
Yousif, Mariam H.M., Ibrahim F. Benter, & Saghir Akhtar. (2005). The role of tyrosine kinase‐mediated pathways in diabetes‐induced alterations in responsiveness of rat carotid artery. Autonomic and Autacoid Pharmacology. 25(2). 69–78. 22 indexed citations
14.
Benter, Ibrahim F., Ijaz A. Khan, C. Cojocel, et al.. (2005). Signal transduction involving Ras-GTPase contributes to development of hypertension and end-organ damage in spontaneously hypertensive rats-treated with L-NAME. Pharmacological Research. 52(5). 401–412. 11 indexed citations
15.
Benboubetra, Mustapha, Pakeeza Sayyed, Keng Wooi Ng, et al.. (2004). Sustained Polymeric Delivery of Gene Silencing Antisense ODNs, siRNA, DNAzymes and Ribozymes:In VitroandIn VivoStudies. Journal of drug targeting. 12(6). 393–404. 107 indexed citations
16.
Sohail, Muhammad, Marcus D. Hughes, Ibrahim F. Benter, et al.. (2003). Messenger RNA expression profiling of genes involved in epidermal growth factor receptor signalling in human cancer cells treated with scanning array-designed antisense oligonucleotides. Biochemical Pharmacology. 66(5). 819–830. 26 indexed citations
17.
Coulson, Judy M. & Saghir Akhtar. (1997). A c‐myc Phosphorothioate Oligonucleotide Protects U87‐MG Glioblastoma Cells from Toxicity at High Concentrations of Lipofectin and Streptolysin O. Pharmacy and Pharmacology Communications. 3(4). 197–201. 3 indexed citations
18.
Hudson, Andrew J., et al.. (1997). Cellular Uptake Properties of a 2‵-Amino/2‵-O-Methyl-Modified Chimeric Hammerhead Ribozyme Targeted to the Epidermal Growth Factor Receptor mRNA. Antisense and Nucleic Acid Drug Development. 7(4). 319–326. 23 indexed citations
19.
Moore, Vanessa, et al.. (1997). Interaction of oligonucleotide-conjugates with the dipeptide transporter system in CACO-2 cells. Biochemical Pharmacology. 53(9). 1223–1228. 13 indexed citations
20.
Akhtar, Saghir & John J. Rossi. (1996). Anti-HIV therapy with antisense oligonucleotides and ribozymes: realistic approaches or expensive myths?. Journal of Antimicrobial Chemotherapy. 38(2). 159–165. 17 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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