Saba Valadkhan

2.6k total citations
39 papers, 1.8k citations indexed

About

Saba Valadkhan is a scholar working on Molecular Biology, Cancer Research and Immunology. According to data from OpenAlex, Saba Valadkhan has authored 39 papers receiving a total of 1.8k indexed citations (citations by other indexed papers that have themselves been cited), including 32 papers in Molecular Biology, 14 papers in Cancer Research and 9 papers in Immunology. Recurrent topics in Saba Valadkhan's work include RNA Research and Splicing (29 papers), RNA modifications and cancer (23 papers) and RNA and protein synthesis mechanisms (17 papers). Saba Valadkhan is often cited by papers focused on RNA Research and Splicing (29 papers), RNA modifications and cancer (23 papers) and RNA and protein synthesis mechanisms (17 papers). Saba Valadkhan collaborates with scholars based in United States, Spain and Switzerland. Saba Valadkhan's co-authors include James L. Manley, Farshad Niazi, Lalith Gunawardane, Bing Zhang, Jonathan Karn, Fereshteh Jahanbani, Curtis Dobrowolski, William P. Schiemann, Puri Fortes and Benjamin G. Luttge and has published in prestigious journals such as Nature, Science and Proceedings of the National Academy of Sciences.

In The Last Decade

Saba Valadkhan

38 papers receiving 1.8k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Saba Valadkhan United States 23 1.4k 703 291 204 134 39 1.8k
Kathleen Börner Germany 19 1.2k 0.9× 159 0.2× 250 0.9× 329 1.6× 273 2.0× 31 1.8k
Gabriele Fuchs United States 19 1.2k 0.9× 377 0.5× 174 0.6× 71 0.3× 173 1.3× 27 1.8k
Viktor Molnár Hungary 15 284 0.2× 242 0.3× 148 0.5× 58 0.3× 140 1.0× 47 815
Weihong Xie China 21 943 0.7× 335 0.5× 641 2.2× 38 0.2× 321 2.4× 37 1.7k
Junfeng Zheng China 14 562 0.4× 359 0.5× 214 0.7× 30 0.1× 147 1.1× 27 1.2k
Adri A.M. Thomas Netherlands 26 1.6k 1.2× 91 0.1× 215 0.7× 62 0.3× 133 1.0× 46 2.1k
Simon Yu United States 11 474 0.3× 93 0.1× 587 2.0× 71 0.3× 104 0.8× 12 1.1k
Jean-François Clément Canada 11 414 0.3× 154 0.2× 331 1.1× 137 0.7× 111 0.8× 14 742
Jaechul Lim South Korea 19 1.5k 1.1× 424 0.6× 253 0.9× 12 0.1× 61 0.5× 28 1.9k
Longwen Deng United States 20 758 0.5× 118 0.2× 505 1.7× 613 3.0× 292 2.2× 23 1.4k

Countries citing papers authored by Saba Valadkhan

Since Specialization
Citations

This map shows the geographic impact of Saba Valadkhan's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Saba Valadkhan with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Saba Valadkhan more than expected).

Fields of papers citing papers by Saba Valadkhan

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Saba Valadkhan. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Saba Valadkhan. The network helps show where Saba Valadkhan may publish in the future.

Co-authorship network of co-authors of Saba Valadkhan

This figure shows the co-authorship network connecting the top 25 collaborators of Saba Valadkhan. A scholar is included among the top collaborators of Saba Valadkhan based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Saba Valadkhan. Saba Valadkhan is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Gunawardane, Lalith, Farshad Niazi, Uri Mbonye, et al.. (2025). HIV infection reprogrammes CD4+ T cells for quiescence and entry into proviral latency. Nature Microbiology. 10(10). 2454–2471.
2.
Ye, Fengchun, David Alvarez-Carbonell, Kien Nguyen, et al.. (2022). Recruitment of the CoREST transcription repressor complexes by Nerve Growth factor IB-like receptor (Nurr1/NR4A2) mediates silencing of HIV in microglial cells. PLoS Pathogens. 18(7). e1010110–e1010110. 15 indexed citations
3.
Mbonye, Uri, Konstantin Leskov, Meenakshi Shukla, Saba Valadkhan, & Jonathan Karn. (2021). Biogenesis of P-TEFb in CD4+ T cells to reverse HIV latency is mediated by protein kinase C (PKC)-independent signaling pathways. PLoS Pathogens. 17(9). e1009581–e1009581. 15 indexed citations
4.
Parker, Kimberly A., et al.. (2019). The lncRNA BORG: a novel inducer of TNBC metastasis, chemoresistance, and disease recurrence. Journal of Cancer Metastasis and Treatment. 2019. 11 indexed citations
5.
Das, Biswajit, Curtis Dobrowolski, Benjamin G. Luttge, et al.. (2018). Estrogen receptor-1 is a key regulator of HIV-1 latency that imparts gender-specific restrictions on the latent reservoir. Proceedings of the National Academy of Sciences. 115(33). E7795–E7804. 115 indexed citations
6.
Zhang, Bing, et al.. (2018). The lncRNA BORG facilitates the survival and chemoresistance of triple-negative breast cancers. Oncogene. 38(12). 2020–2041. 75 indexed citations
7.
García‐Mesa, Yoelvis, Taylor R. Jay, Mary Ann Checkley, et al.. (2016). Immortalization of primary microglia: a new platform to study HIV regulation in the central nervous system. Journal of NeuroVirology. 23(1). 47–66. 133 indexed citations
8.
Zhang, Bing, Lalith Gunawardane, Farshad Niazi, et al.. (2014). A Novel RNA Motif Mediates the Strict Nuclear Localization of a Long Noncoding RNA. Molecular and Cellular Biology. 34(12). 2318–2329. 120 indexed citations
9.
Kambara, Hiroto, Farshad Niazi, Lenche Kostadinova, et al.. (2014). Negative regulation of the interferon response by an interferon-induced long non-coding RNA. Nucleic Acids Research. 42(16). 10668–10680. 179 indexed citations
10.
Valadkhan, Saba. (2013). The Role of snRNAs in Spliceosomal Catalysis. Progress in molecular biology and translational science. 120. 195–228. 4 indexed citations
11.
Niazi, Farshad & Saba Valadkhan. (2012). Computational analysis of functional long noncoding RNAs reveals lack of peptide-coding capacity and parallels with 3′ UTRs. RNA. 18(4). 825–843. 131 indexed citations
12.
Valadkhan, Saba. (2011). A snRNP's ordered path to maturity: Figure 1.. Genes & Development. 25(15). 1563–1567. 1 indexed citations
13.
Valadkhan, Saba. (2010). Role of the snRNAs in spliceosomal active site. RNA Biology. 7(3). 345–353. 36 indexed citations
14.
Valadkhan, Saba & Timothy W. Nilsen. (2010). Reprogramming of the non-coding transcriptome during brain development. Journal of Biology. 9(1). 5–5. 20 indexed citations
15.
Valadkhan, Saba, et al.. (2007). Protein-free spliceosomal snRNAs catalyze a reaction that resembles the first step of splicing. RNA. 13(12). 2300–2311. 33 indexed citations
16.
Valadkhan, Saba. (2007). The spliceosome: caught in a web of shifting interactions. Current Opinion in Structural Biology. 17(3). 310–315. 34 indexed citations
17.
Valadkhan, Saba. (2005). snRNAs as the catalysts of pre-mRNA splicing. Current Opinion in Chemical Biology. 9(6). 603–608. 101 indexed citations
18.
Valadkhan, Saba & James L. Manley. (2003). Characterization of the catalytic activity of U2 and U6 snRNAs. RNA. 9(7). 892–904. 59 indexed citations
19.
Valadkhan, Saba & James L. Manley. (2001). Splicing-related catalysis by protein-free snRNAs. Nature. 413(6857). 701–707. 159 indexed citations
20.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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