Marie Trudel

3.2k total citations
71 papers, 2.5k citations indexed

About

Marie Trudel is a scholar working on Molecular Biology, Genetics and Genetics. According to data from OpenAlex, Marie Trudel has authored 71 papers receiving a total of 2.5k indexed citations (citations by other indexed papers that have themselves been cited), including 45 papers in Molecular Biology, 37 papers in Genetics and 26 papers in Genetics. Recurrent topics in Marie Trudel's work include Genetic and Kidney Cyst Diseases (26 papers), Hemoglobinopathies and Related Disorders (26 papers) and Renal and related cancers (25 papers). Marie Trudel is often cited by papers focused on Genetic and Kidney Cyst Diseases (26 papers), Hemoglobinopathies and Related Disorders (26 papers) and Renal and related cancers (25 papers). Marie Trudel collaborates with scholars based in Canada, United States and France. Marie Trudel's co-authors include Vivette D. D’Agati, Frank Costantini, Almira Kurbegovic, Martin Couillard, Joël Lanoix, F Costantini, Richard Guillaume, M Meuth, Nacéra Saadane and M Szabolcs and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Nucleic Acids Research and Journal of Biological Chemistry.

In The Last Decade

Marie Trudel

70 papers receiving 2.4k citations

Peers

Marie Trudel
Thomas M. Ryan United States
G. Levan Sweden
Barbara K. Goodman United States
Quinten Waisfisz Netherlands
George F. Atweh United States
Christopher H. Lowrey United States
Nianxiang Zhang United States
R P Castleberry United States
Thomas M. Ryan United States
Marie Trudel
Citations per year, relative to Marie Trudel Marie Trudel (= 1×) peers Thomas M. Ryan

Countries citing papers authored by Marie Trudel

Since Specialization
Citations

This map shows the geographic impact of Marie Trudel's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Marie Trudel with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Marie Trudel more than expected).

Fields of papers citing papers by Marie Trudel

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Marie Trudel. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Marie Trudel. The network helps show where Marie Trudel may publish in the future.

Co-authorship network of co-authors of Marie Trudel

This figure shows the co-authorship network connecting the top 25 collaborators of Marie Trudel. A scholar is included among the top collaborators of Marie Trudel based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Marie Trudel. Marie Trudel is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Trudel, Marie & Vivette D. D’Agati. (2015). A Model of Polycystic Kidney Disease in SBM Transgenic Mice. Contributions to nephrology. 97. 47–59. 3 indexed citations
2.
Kurbegovic, Almira & Marie Trudel. (2013). Progressive development of polycystic kidney disease in the mouse model expressing Pkd1 extracellular domain. Human Molecular Genetics. 22(12). 2361–2375. 14 indexed citations
3.
Shmukler, Boris E., Ann Hsu, Marie Trudel, et al.. (2013). N-ethylmaleimide activates a Cl−-independent component of K+ flux in mouse erythrocytes. Blood Cells Molecules and Diseases. 51(1). 9–16. 5 indexed citations
4.
Alhashem, Yousef N., Catherine I. Dumur, Kellie J. Archer, et al.. (2012). Krüppel-Like Factor 1 (KLF1), KLF2, and Myc Control a Regulatory Network Essential for Embryonic Erythropoiesis. Molecular and Cellular Biology. 32(13). 2628–2644. 32 indexed citations
5.
Boosalis, Michael S., Serguei A. Castañeda, Marie Trudel, et al.. (2011). Novel therapeutic candidates, identified by molecular modeling, induce γ-globin gene expression in vivo. Blood Cells Molecules and Diseases. 47(2). 107–116. 15 indexed citations
6.
Vandorpe, David H., Chang Xu, Boris E. Shmukler, et al.. (2010). Hypoxia Activates a Ca2+-Permeable Cation Conductance Sensitive to Carbon Monoxide and to GsMTx-4 in Human and Mouse Sickle Erythrocytes. PLoS ONE. 5(1). e8732–e8732. 47 indexed citations
7.
Trudel, Marie, et al.. (2009). Successful correction of murine sickle cell disease with reduced stem cell requirements reinforced by fractionated marrow infusions. British Journal of Haematology. 148(4). 646–658. 4 indexed citations
8.
Couillard, Martin & Marie Trudel. (2009). C‐myc as a modulator of renal stem/progenitor cell population. Developmental Dynamics. 238(2). 405–414. 41 indexed citations
9.
Harada, Ryoko, Marilène Paquet, Olivier Côté, et al.. (2008). Polycystic Kidneys Caused by Sustained Expression of Cux1 Isoform p75. Journal of Biological Chemistry. 283(20). 13817–13824. 23 indexed citations
10.
Trudel, Marie, et al.. (2008). Evidence for a Bigenic Chromatin Subdomain in Regulation of the Fetal-to-Adult Hemoglobin Switch. Molecular and Cellular Biology. 29(6). 1635–1648. 6 indexed citations
11.
Bottardi, Stefania, Julie Ross, Vincent Bourgoin, et al.. (2008). Ikaros and GATA-1 Combinatorial Effect Is Required for Silencing of Human γ-Globin Genes. Molecular and Cellular Biology. 29(6). 1526–1537. 56 indexed citations
12.
Rust, Marco B., Seth L. Alper, York Rudhard, et al.. (2007). Disruption of erythroid K-Cl cotransporters alters erythrocyte volume and partially rescues erythrocyte dehydration in SAD mice. Journal of Clinical Investigation. 117(6). 1708–1717. 68 indexed citations
13.
Blouin, Marie‐José, Martin Sorette, Anne‐Marie Bleau, et al.. (2000). Genetic correction of sickle cell disease: Insights using transgenic mouse models. Nature Medicine. 6(2). 177–182. 43 indexed citations
14.
Guillaume, Richard & Marie Trudel. (2000). Distinct and common developmental expression patterns of the murine Pkd2 and Pkd1 genes. Mechanisms of Development. 93(1-2). 179–183. 36 indexed citations
15.
Trudel, Marie. (2000). Progress in molecular genetics of autosomal dominant polycystic kidney disease. Frontiers in bioscience. 5(1). d312–d312. 1 indexed citations
16.
Guillaume, Richard, et al.. (1999). MurinePkd1 is a developmentally regulated gene from morula to adulthood: Role in tissue condensation and patterning. Developmental Dynamics. 214(4). 337–348. 47 indexed citations
17.
Barisoni, Laura, et al.. (1995). Analysis of the role of membrane polarity in polycystic kidney disease of transgenic SBM mice.. PubMed. 147(6). 1728–35. 24 indexed citations
18.
Franceschi, Lucia De, Nacéra Saadane, Marie Trudel, et al.. (1994). Treatment with oral clotrimazole blocks Ca(2+)-activated K+ transport and reverses erythrocyte dehydration in transgenic SAD mice. A model for therapy of sickle cell disease.. Journal of Clinical Investigation. 93(4). 1670–1676. 120 indexed citations
19.
Trudel, Marie, Vivette D. D’Agati, & Frank Costantini. (1991). c-myc as an inducer of polycystic kidney disease in transgenic mice. Kidney International. 39(4). 665–671. 171 indexed citations
20.
Meuth, Mark, Marie Trudel, & Louis Siminovitch. (1979). Selection of Chinese hamster cells auxotrophic for thymidine by 1-#x03B2;-D-arabinofuranosyl cytosine. Somatic Cell and Molecular Genetics. 5(3). 303–318. 34 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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