S. Streit
Impact in
- Oncology top 10%
- HER2/EGFR in Cancer Research
Papers in ⓘ
-
- Fibroblast Growth Factor Research 3
- Epigenetics and DNA Methylation 2
- Kruppel-like factors research 1
- Oncology 5
- HER2/EGFR in Cancer Research 2
- Co-authors
- A. Ullrich (4 shared papers)Stefan Hart (2 shared papers)Oliver M. Fischer (1 shared paper)Norbert Prenzel (1 shared paper)Carola Berking (1 shared paper)M. Schmidt (1 shared paper)Jens E. Ruhe (2 shared papers)Dominik Mestel (1 shared paper)
- Journals
- Oncogene (2 papers)International Journal of Molecular Sciences (1 paper)British Journal of Cancer (1 paper)Cellular Signalling (1 paper)Endocrine Related Cancer (1 paper)
- Partner nations
- GermanySingaporeUnited States
In The Last Decade
S. Streit
8 papers receiving 807 citations
Hit Papers
Peers
Comparison fields: 5 of 88
- Oncology 364
- Cancer Research 130
- Immunology and Allergy 50
- Molecular Biology 481
- Drug Discovery 1
Countries citing papers authored by S. Streit
This map shows the geographic impact of S. Streit's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by S. Streit with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites S. Streit more than expected).
Fields of papers citing papers by S. Streit
This network shows the impact of papers produced by S. Streit. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by S. Streit. The network helps show where S. Streit may publish in the future.
Co-authors
The 25 scholars most cited alongside S. Streit, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | The epidermal growth factor receptor family as a central element for cellular signal transduction and diversification. Hit paper breakdown → | 2001 | 553 |
| 2 | 2010 | 80 | |
| 3 | 2006 | 69 | |
| 4 | 2009 | 46 | |
| 5 | 2006 | 40 | |
| 6 | 2024 | 27 | |
| 7 | 2006 | 6 | |
| 8 | 2008 | 2 |
About S. Streit
S. Streit is a scholar working on Molecular Biology, Oncology, Radiology, Nuclear Medicine and Imaging, Cancer Research and Pathology and Forensic Medicine, having authored 8 papers that have together received 823 indexed citations. Recurring topics across this work include Fibroblast Growth Factor Research (3 papers), Cancer, Hypoxia, and Metabolism (2 papers), HER2/EGFR in Cancer Research (2 papers), Epigenetics and DNA Methylation (2 papers), Monoclonal and Polyclonal Antibodies Research (2 papers), Kruppel-like factors research (1 paper), Mast cells and histamine (1 paper) and Hippo pathway signaling and YAP/TAZ (1 paper). The work is most often cited by research in Oncology (364 citations), Cancer Research (130 citations), Immunology and Allergy (50 citations), Molecular Biology (481 citations) and Drug Discovery (1 citation). S. Streit has collaborated with scholars based in Germany, Singapore and United States. Frequent co-authors include A. Ullrich, Stefan Hart, Oliver M. Fischer, Norbert Prenzel, Carola Berking, M. Schmidt, Jens E. Ruhe, Dominik Mestel, Sarah Hart and Christoph Michalski. Their work appears in journals such as Oncogene, International Journal of Molecular Sciences, British Journal of Cancer, Cellular Signalling and Endocrine Related Cancer.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.