S. Kammerer

523 total citations
12 papers, 390 citations indexed

About

S. Kammerer is a scholar working on Molecular Biology, Genetics and Hematology. According to data from OpenAlex, S. Kammerer has authored 12 papers receiving a total of 390 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Molecular Biology, 4 papers in Genetics and 4 papers in Hematology. Recurrent topics in S. Kammerer's work include Coagulation, Bradykinin, Polyphosphates, and Angioedema (4 papers), Blood Coagulation and Thrombosis Mechanisms (4 papers) and Peptidase Inhibition and Analysis (2 papers). S. Kammerer is often cited by papers focused on Coagulation, Bradykinin, Polyphosphates, and Angioedema (4 papers), Blood Coagulation and Thrombosis Mechanisms (4 papers) and Peptidase Inhibition and Analysis (2 papers). S. Kammerer collaborates with scholars based in Germany, Australia and Poland. S. Kammerer's co-authors include Andreas Braun, A. Roscher, Norbert Arnold, Hartwig Clevė, B. Müller, U. Löhrs, Ursula Kuhnle, Hans‐Peter Schwarz, Nathan Markward and Matthew R. Nelson and has published in prestigious journals such as Biochemical and Biophysical Research Communications, Human Molecular Genetics and Gene.

In The Last Decade

S. Kammerer

12 papers receiving 380 citations

Peers

Countries citing papers authored by S. Kammerer

Since Specialization

Citations

This map shows the geographic impact of S. Kammerer's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by S. Kammerer with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites S. Kammerer more than expected).

Fields of papers citing papers by S. Kammerer

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by S. Kammerer. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by S. Kammerer. The network helps show where S. Kammerer may publish in the future.

Co-authorship network of co-authors of S. Kammerer

This figure shows the co-authorship network connecting the top 25 collaborators of S. Kammerer. A scholar is included among the top collaborators of S. Kammerer based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with S. Kammerer. S. Kammerer is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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12 of 12 papers shown

#	Work	Indexed citations
1	Identification of the semaphorin receptor PLXNA2 as a candidate for susceptibility to schizophrenia 2006 ·Molecular Psychiatry ·Steven Mah, Matthew R. Nelson, Lynn E. DeLisi, (unknown), Nathan Markward, Michael R. James, Dale R. Nyholt, Nicholas K. Hayward, Herlina Y. Handoko, Bryan Mowry, S. Kammerer, Andreas Braun	107
2	Klinische und genetische Befunde unter besonderer Betrachtung kornealer Ver�nderungen bei Lowe-Syndrom 2004 ·Der Ophthalmologe ·Günther Rudolph, (unknown), (unknown), Christos Haritoglou, S. Kammerer, K.‐P. Boergen, Anselm Kampik	1
3	A novel sequence polymorphism in the promoter region of the human B2-bradykinin receptor gene 1996 ·Human Genetics ·Annika Braun, Esther M. Maier, S. Kammerer, (unknown), B. Müller	22
4	A novel sequence polymorphism in the promoter region of the human B2-bradykinin receptor gene 1996 ·Human Genetics ·Annika Braun, Esther M. Maier, S. Kammerer, B. Müller, (unknown)	6
5	Population study of a sequence polymorphism in intron 2 of the human ?-globin gene 1995 ·Human Genetics ·Andreas Braun, (unknown), (unknown), (unknown), S. Kammerer	6
6	The Human Bradykinin B2 Receptor Gene: Full-Length cDNA, Genomic Organization and Identification of the Regulatory Region 1995 ·Biochemical and Biophysical Research Communications ·S. Kammerer, Andreas Braun, Norbert Arnold, A. Roscher	64
7	Identification of Polymorphic Sites of the Human Bradykinin B2 Receptor Gene 1995 ·Biochemical and Biophysical Research Communications ·Andreas Braun, S. Kammerer, (unknown), B. Müller, A. Roscher	49
8	Characterization of mutants of the vitamin D-binding protein/group-specific component: molecular evolution of GC1A2 and GC1A3, common in some Asian populations 1995 ·Human Genetics ·Isao Yuasa, (unknown), Andreas Braun, K. Umetsu, (unknown), S. Kammerer, Hartwig Clevė	6
9	Mutations in the gene for X-linked adrenoleukodystrophy in patients with different clinical phenotypes. 1995 ·PubMed ·Andreas Braun, (unknown), S. Kammerer, Boris Rolinski, Sylvia Stöckler, Wolfgang Rabl, Jutta Gärtner, S. Zierz, A. Roscher	40
10	Sequence of a putative human housekeeping gene (HK33) localized on chromosome 1 1994 ·Gene ·Andreas Braun, S. Kammerer, Winfríed Weissenhorn, Elisabeth H. Weiss, Hartwig Clevė	22
11	True hermaphroditism in a 46,XY individual, caused by a postzygotic somatic point mutation in the male gonadal sex-determining locus (SRY): molecular genetics and histological findings in a sporadic case. 1993 ·PubMed ·Andreas Braun, S. Kammerer, Hartwig Clevė, U. Löhrs, Hans‐Peter Schwarz, Ursula Kuhnle	65
12	A novel sequence polymorphism in exon 1 of the human vitamin D-binding protein (GC) gene 1993 ·Human Molecular Genetics ·Andreas Braun, (unknown), (unknown), S. Kammerer, Hartwig Clevė	2

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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