S. Ipsen

417 total citations
8 papers, 337 citations indexed

About

S. Ipsen is a scholar working on Physiology, Molecular Biology and Cellular and Molecular Neuroscience. According to data from OpenAlex, S. Ipsen has authored 8 papers receiving a total of 337 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Physiology, 3 papers in Molecular Biology and 3 papers in Cellular and Molecular Neuroscience. Recurrent topics in S. Ipsen's work include Alzheimer's disease research and treatments (6 papers), Neuroscience and Neuropharmacology Research (3 papers) and Prion Diseases and Protein Misfolding (2 papers). S. Ipsen is often cited by papers focused on Alzheimer's disease research and treatments (6 papers), Neuroscience and Neuropharmacology Research (3 papers) and Prion Diseases and Protein Misfolding (2 papers). S. Ipsen collaborates with scholars based in Switzerland, Sweden and Germany. S. Ipsen's co-authors include A. Probst, J. Ulrich, Markus Tolnay, Eddi Meier, W. Meier‐Ruge, Giovannina Botez, Dominique Langui, Nikolaos K. Robakis, Claudia Mistl and Martin C. Herzig and has published in prestigious journals such as PLoS ONE, Acta Neuropathologica and Advances in experimental medicine and biology.

In The Last Decade

S. Ipsen

8 papers receiving 332 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
S. Ipsen Switzerland	6	262	124	94	81	77	8	337
Q.-X. Li Australia	7	206 0.8×	138 1.1×	108 1.1×	102 1.3×	110 1.4×	10	386
Hedvig Welander Sweden	10	352 1.3×	191 1.5×	81 0.9×	95 1.2×	94 1.2×	13	465
Ola Philipson Sweden	9	294 1.1×	183 1.5×	50 0.5×	55 0.7×	91 1.2×	9	417
Sarah Scott United States	8	190 0.7×	96 0.8×	41 0.4×	109 1.3×	70 0.9×	10	321
Astrid Gumucio Sweden	8	240 0.9×	139 1.1×	48 0.5×	82 1.0×	100 1.3×	9	402
Norimichi Nakamura Japan	8	192 0.7×	116 0.9×	69 0.7×	57 0.7×	72 0.9×	13	365
Steve Kucera United States	3	270 1.0×	150 1.2×	53 0.6×	91 1.1×	71 0.9×	6	337
Tetsuo Fukushima Japan	6	273 1.0×	128 1.0×	70 0.7×	29 0.4×	45 0.6×	8	357
Krishna Bharani United States	6	200 0.8×	111 0.9×	68 0.7×	34 0.4×	73 0.9×	13	398
Yasuko Komatsuzaki Japan	8	243 0.9×	167 1.3×	34 0.4×	123 1.5×	92 1.2×	12	428

Countries citing papers authored by S. Ipsen

Since Specialization

Citations

This map shows the geographic impact of S. Ipsen's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by S. Ipsen with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites S. Ipsen more than expected).

Fields of papers citing papers by S. Ipsen

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by S. Ipsen. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by S. Ipsen. The network helps show where S. Ipsen may publish in the future.

Co-authorship network of co-authors of S. Ipsen

This figure shows the co-authorship network connecting the top 25 collaborators of S. Ipsen. A scholar is included among the top collaborators of S. Ipsen based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with S. Ipsen. S. Ipsen is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

8 of 8 papers shown

1.

Stalder, Anna K., et al.. (2015). mTOR is expressed in polymyositis but not in sporadic inclusion body myositis. Clinical Neuropathology. 34(11). 371–373. 2 indexed citations

2.

Hench, Jürgen, Claire Pujol, S. Ipsen, et al.. (2011). A Tissue-Specific Approach to the Analysis of Metabolic Changes in Caenorhabditis elegans. PLoS ONE. 6(12). e28417–e28417. 13 indexed citations

3.

Probst, A., Claudia Mistl, S. Ipsen, & Markus Tolnay. (2001). Perisomatic Granules of Hippocampal CA1 Neurons in Alzheimer’s Disease, Pre-Alzheimer Stage and Pick’s Disease: An Overlooked Pathological Entity. Advances in experimental medicine and biology. 487. 187–198. 1 indexed citations

4.

Probst, A., Martin C. Herzig, Claudia Mistl, S. Ipsen, & Markus Tolnay. (2001). Perisomatic granules (non-plaque dystrophic dendrites) of hippocampal CA1 neurons in Alzheimer's disease and Pick's disease: a lesion distinct from granulovacuolar degeneration. Acta Neuropathologica. 102(6). 636–644. 24 indexed citations

5.

Botez, Giovannina, A. Probst, S. Ipsen, & Markus Tolnay. (1999). Astrocytes expressing hyperphosphorylated tau protein without glial fibrillary tangles in argyrophilic grain disease. Acta Neuropathologica. 98(3). 251–256. 73 indexed citations

6.

Tolnay, Markus, Claudia Mistl, S. Ipsen, & A. Probst. (1998). Argyrophilic grains of Braak: occurrence in dendrites of neurons containing hyperphosphorylated tau protein. Neuropathology and Applied Neurobiology. 24(1). 53–59. 38 indexed citations

7.

Probst, A., Dominique Langui, S. Ipsen, Nikolaos K. Robakis, & J. Ulrich. (1991). Deposition of ?/A4 protein along neuronal plasma membranes in diffuse senile plaques. Acta Neuropathologica. 83(1). 21–29. 89 indexed citations

8.

Ulrich, J., W. Meier‐Ruge, A. Probst, Eddi Meier, & S. Ipsen. (1990). Senile plaques: staining for acetylcholinesterase and A4 protein: a comparative study in the hippocampus and entorhinal cortex. Acta Neuropathologica. 80(6). 624–628. 97 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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