S. Ipsen

419 citations
8 papers · 338 · h-index 6

Impact in

  • Physiology top 10%
    • Alzheimer's disease research and treatments
  • Neurology top 10%
    • Parkinson's Disease Mechanisms and Treatments
    • Neurological diseases and metabolism

Papers in

S. Ipsen

8 papers receiving 333 citations

Peers

S. Ipsen
Comparison fields: 5 of 45
  • Physiology 255
  • Neurology 72
  • Neurology 80
  • Pharmacology 91
  • Aging 8
Replace Rejith Dayanandan with:
Rejith Dayanandan United Kingdom
Norimichi Nakamura Japan
Sarah Scott United States
Alice Bittar United States
Diana Thyssen United States
Eleni Gkanatsiou Sweden
Ľubica Fialová Slovakia
Bonnie Cheung United States
Yuan-Wen Ge United States
Bianca T. Corjuc United States
S. Ipsen relative to Rejith Dayanandan United Kingdom Rejith Dayanandan's profile →
Citations per field
00.5×1.5×
Rejith Dayanandan · 1×
Citations per year

Countries citing papers authored by S. Ipsen

Since Specialization
Citations

This map shows the geographic impact of S. Ipsen's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by S. Ipsen with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites S. Ipsen more than expected).

Fields of papers citing papers by S. Ipsen

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by S. Ipsen. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by S. Ipsen. The network helps show where S. Ipsen may publish in the future.

Co-authors

The 19 scholars most cited alongside S. Ipsen, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with S. Ipsen Line = papers co-authored together S. Ipsen links everyone, so they are left out of the graph.

All Works

8 of 8 papers shown
#Work
1 199097
2 199189
3 199973
4 199838
5 200124
6 201114
7 20152
8 20011

About S. Ipsen

S. Ipsen is a scholar working on Physiology, Molecular Biology, Cellular and Molecular Neuroscience, Neurology and Biochemistry, having authored 8 papers that have together received 338 indexed citations. Recurring topics across this work include Alzheimer's disease research and treatments (6 papers), Neuroscience and Neuropharmacology Research (3 papers), Prion Diseases and Protein Misfolding (2 papers), Neuroinflammation and Neurodegeneration Mechanisms (2 papers), Amino Acid Enzymes and Metabolism (1 paper), Cholinesterase and Neurodegenerative Diseases (1 paper), Biochemical Acid Research Studies (1 paper) and Genetics, Aging, and Longevity in Model Organisms (1 paper). The work is most often cited by research in Physiology (255 citations), Neurology (72 citations), Neurology (80 citations), Pharmacology (91 citations) and Aging (8 citations). S. Ipsen has collaborated with scholars based in Switzerland, Germany and United States. Frequent co-authors include A. Probst, J. Ulrich, Markus Tolnay, Eddi Meier, W. Meier‐Ruge, Nikolaos K. Robakis, Giovannina Botez, Dominique Langui, Claudia Mistl and Martin C. Herzig. Their work appears in journals such as Acta Neuropathologica, Neuropathology and Applied Neurobiology, Advances in experimental medicine and biology, Clinical Neuropathology and PLoS ONE.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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