R.S. Cornelis
Impact in
-
- Genetic factors in colorectal cancer
- Cancer Research top 10%
- Cancer Genomics and Diagnostics
Papers in
-
- Cancer Genomics and Diagnostics 4
- Cancer, Lipids, and Metabolism 1
-
- Hedgehog Signaling Pathway Studies 2
- DNA Repair Mechanisms 1
- Co-authors
- Peter Devilee (6 shared papers)A.M. Cleton-Jansen (3 shared papers)Cees J. Cornelisse (3 shared papers)Richard Wooster (1 shared paper)Christopher S. Cooper (1 shared paper)B. A. J. Ponder (1 shared paper)Andreas von Deimling (1 shared paper)B A Gusterson (1 shared paper)
- Journals
- Human Mutation (2 papers)Nature Genetics (1 paper)Cancer Genetics and Cytogenetics (1 paper)Pathology - Research and Practice (1 paper)PubMed (1 paper)
- Partner nations
- NetherlandsUnited KingdomGermany
In The Last Decade
R.S. Cornelis
6 papers receiving 476 citations
Peers
Comparison fields: 5 of 47
- Pathology and Forensic Medicine 276
- Cancer Research 212
- Oncology 189
- Molecular Biology 258
- Genetics 101
Countries citing papers authored by R.S. Cornelis
This map shows the geographic impact of R.S. Cornelis's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by R.S. Cornelis with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites R.S. Cornelis more than expected).
Fields of papers citing papers by R.S. Cornelis
This network shows the impact of papers produced by R.S. Cornelis. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by R.S. Cornelis. The network helps show where R.S. Cornelis may publish in the future.
Co-authors
The 25 scholars most cited alongside R.S. Cornelis, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 1994 | 340 | |
| 2 | Evidence for a gene on 17p13.3, distal to TP53, as a target for allele loss in breast tumors without p53 mutations. | 1994 | 81 |
| 3 | 1994 | 28 | |
| 4 | 1997 | 18 | |
| 5 | 1996 | 17 | |
| 6 | 1997 | 5 |
About R.S. Cornelis
R.S. Cornelis is a scholar working on Cancer Research, Molecular Biology, Oncology, Pathology and Forensic Medicine and Cellular and Molecular Neuroscience, having authored 6 papers that have together received 489 indexed citations. Recurring topics across this work include Cancer Genomics and Diagnostics (4 papers), Cancer-related Molecular Pathways (4 papers), Hedgehog Signaling Pathway Studies (2 papers), BRCA gene mutations in cancer (1 paper), Genetic factors in colorectal cancer (1 paper), Cancer, Lipids, and Metabolism (1 paper), Genetic Neurodegenerative Diseases (1 paper) and DNA Repair Mechanisms (1 paper). The work is most often cited by research in Pathology and Forensic Medicine (276 citations), Cancer Research (212 citations), Oncology (189 citations), Molecular Biology (258 citations) and Genetics (101 citations). R.S. Cornelis has collaborated with scholars based in Netherlands, United Kingdom and Germany. Frequent co-authors include Peter Devilee, A.M. Cleton-Jansen, Cees J. Cornelisse, Richard Wooster, Christopher S. Cooper, B. A. J. Ponder, Andreas von Deimling, B A Gusterson, Nadine Collins and O. D. Wiestler. Their work appears in journals such as Human Mutation, Nature Genetics, Cancer Genetics and Cytogenetics, Pathology - Research and Practice and PubMed.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.