Roxane Lussier

705 total citations
15 papers, 571 citations indexed

About

Roxane Lussier is a scholar working on Surgery, Physiology and Molecular Biology. According to data from OpenAlex, Roxane Lussier has authored 15 papers receiving a total of 571 indexed citations (citations by other indexed papers that have themselves been cited), including 11 papers in Surgery, 9 papers in Physiology and 7 papers in Molecular Biology. Recurrent topics in Roxane Lussier's work include Pancreatic function and diabetes (10 papers), Adipose Tissue and Metabolism (9 papers) and Lipid metabolism and biosynthesis (5 papers). Roxane Lussier is often cited by papers focused on Pancreatic function and diabetes (10 papers), Adipose Tissue and Metabolism (9 papers) and Lipid metabolism and biosynthesis (5 papers). Roxane Lussier collaborates with scholars based in Canada, United States and Kuwait. Roxane Lussier's co-authors include Marie‐Line Peyot, Marc Prentki, Érik Joly, S.R. Murthy Madiraju, Marco Pineda, Julien Lamontagne, Martin G. Latour, Émilie Pépin, Shangang Zhao and Yves Mugabo and has published in prestigious journals such as Journal of Biological Chemistry, PLoS ONE and Cell Metabolism.

In The Last Decade

Roxane Lussier

14 papers receiving 568 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Roxane Lussier Canada	11	349	218	216	166	126	15	571
Clarissa Bartley Switzerland	8	353 1.0×	139 0.6×	268 1.2×	195 1.2×	36 0.3×	9	621
Anne Reifel Miller United States	8	265 0.8×	138 0.6×	307 1.4×	103 0.6×	73 0.6×	12	523
Katy J. Brocklehurst United Kingdom	10	385 1.1×	207 0.9×	546 2.5×	205 1.2×	59 0.5×	11	742
Rodolphe Dusaulcy Switzerland	12	164 0.5×	177 0.8×	384 1.8×	106 0.6×	71 0.6×	20	658
Brian A. Oldham United States	5	235 0.7×	131 0.6×	288 1.3×	103 0.6×	68 0.5×	6	479
Carol Lenaghan United Kingdom	7	162 0.5×	222 1.0×	249 1.2×	220 1.3×	27 0.2×	8	554
Adama Kamagaté United States	12	255 0.7×	156 0.7×	435 2.0×	191 1.2×	49 0.4×	19	748
Ingo Uphues Germany	10	206 0.6×	170 0.8×	404 1.9×	174 1.0×	34 0.3×	17	617
Habiba Najafi United States	14	566 1.6×	157 0.7×	476 2.2×	306 1.8×	53 0.4×	15	859
Tokuyuki Yamashita Japan	9	450 1.3×	128 0.6×	344 1.6×	410 2.5×	29 0.2×	14	735

Countries citing papers authored by Roxane Lussier

Since Specialization

Citations

This map shows the geographic impact of Roxane Lussier's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Roxane Lussier with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Roxane Lussier more than expected).

Fields of papers citing papers by Roxane Lussier

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Roxane Lussier. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Roxane Lussier. The network helps show where Roxane Lussier may publish in the future.

Co-authorship network of co-authors of Roxane Lussier

This figure shows the co-authorship network connecting the top 25 collaborators of Roxane Lussier. A scholar is included among the top collaborators of Roxane Lussier based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Roxane Lussier. Roxane Lussier is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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15 of 15 papers shown

Poursharifi, Pegah, Camille Attané, Isabelle Chénier, et al.. (2025). Suppression of adipocyte ABHD6 favors anti-inflammatory and adipogenic programs to preserve adipose tissue fitness in obesity. Molecular Metabolism. 101. 102241–102241.

Poursharifi, Pegah, C. Schmitt, Isabelle Chénier, et al.. (2023). ABHD6 suppression promotes anti-inflammatory polarization of adipose tissue macrophages via 2-monoacylglycerol/PPAR signaling in obese mice. Molecular Metabolism. 78. 101822–101822. 4 indexed citations

Al‐Mass, Anfal, Pegah Poursharifi, Marie‐Line Peyot, et al.. (2022). Hepatic glycerol shunt and glycerol-3-phosphate phosphatase control liver metabolism and glucodetoxification under hyperglycemia. Molecular Metabolism. 66. 101609–101609. 11 indexed citations

Al‐Mass, Anfal, Pegah Poursharifi, Marie‐Line Peyot, et al.. (2022). Glycerol-3-phosphate phosphatase operates a glycerol shunt in pancreatic β-cells that controls insulin secretion and metabolic stress. Molecular Metabolism. 60. 101471–101471. 10 indexed citations

Peyot, Marie‐Line, Anna Roubtsova, Roxane Lussier, et al.. (2021). Substantial PCSK9 inactivation in β-cells does not modify glucose homeostasis or insulin secretion in mice. Biochimica et Biophysica Acta (BBA) - Molecular and Cell Biology of Lipids. 1866(8). 158968–158968. 25 indexed citations

Poursharifi, Pegah, Camille Attané, Yves Mugabo, et al.. (2020). Adipose ABHD6 regulates tolerance to cold and thermogenic programs. JCI Insight. 5(24). 27 indexed citations

Attané, Camille, Marie‐Line Peyot, Roxane Lussier, et al.. (2016). A beta cell ATGL-lipolysis/adipose tissue axis controls energy homeostasis and body weight via insulin secretion in mice. Diabetologia. 59(12). 2654–2663. 43 indexed citations

Attané, Camille, Marie‐Line Peyot, Roxane Lussier, et al.. (2016). Differential Insulin Secretion of High-Fat Diet-Fed C57BL/6NN and C57BL/6NJ Mice: Implications of Mixed Genetic Background in Metabolic Studies. PLoS ONE. 11(7). e0159165–e0159165. 25 indexed citations

Pépin, Émilie, Anfal Al‐Mass, Camille Attané, et al.. (2016). Pancreatic β-Cell Dysfunction in Diet-Induced Obese Mice: Roles of AMP-Kinase, Protein Kinase Cε, Mitochondrial and Cholesterol Metabolism, and Alterations in Gene Expression. PLoS ONE. 11(4). e0153017–e0153017. 22 indexed citations

10.

Zhao, Shangang, Yves Mugabo, José Iglesias, et al.. (2014). α/β-Hydrolase Domain-6-Accessible Monoacylglycerol Controls Glucose-Stimulated Insulin Secretion. Cell Metabolism. 19(6). 993–1007. 127 indexed citations

11.

Attané, Camille, Shupei Wang, Grant A. Mitchell, et al.. (2013). Role of Adipose Triglyceride Lipase and Lipolysis in the Regulation of Insulin Secretion: Study in β-Cell-Specific ATGL-Deficient Mice. Canadian Journal of Diabetes. 37. S58–S58. 1 indexed citations

12.

Peyot, Marie‐Line, Émilie Pépin, Julien Lamontagne, et al.. (2010). β-Cell Failure in Diet-Induced Obese Mice Stratified According to Body Weight Gain: Secretory Dysfunction and Altered Islet Lipid Metabolism Without Steatosis or Reduced β-Cell Mass. Diabetes. 59(9). 2178–2187. 127 indexed citations

13.

Peyot, Marie‐Line, Claudiane Guay, Martin G. Latour, et al.. (2009). Adipose Triglyceride Lipase Is Implicated in Fuel- and Non-fuel-stimulated Insulin Secretion. Journal of Biological Chemistry. 284(25). 16848–16859. 71 indexed citations

14.

Würtele, Hugo, et al.. (2005). Characterization of in vivo recombination activities in the mouse embryo. Molecular Genetics and Genomics. 273(3). 252–263. 10 indexed citations

15.

Peyot, Marie‐Line, Christopher J. Nolan, Krishnakant G. Soni, et al.. (2004). Hormone-Sensitive Lipase Has a Role in Lipid Signaling for Insulin Secretion but Is Nonessential for the Incretin Action of Glucagon-Like Peptide 1. Diabetes. 53(7). 1733–1742. 68 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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