RoseAnn Berlin

917 total citations
16 papers, 704 citations indexed

About

RoseAnn Berlin is a scholar working on Cellular and Molecular Neuroscience, Sensory Systems and Immunology. According to data from OpenAlex, RoseAnn Berlin has authored 16 papers receiving a total of 704 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Cellular and Molecular Neuroscience, 4 papers in Sensory Systems and 4 papers in Immunology. Recurrent topics in RoseAnn Berlin's work include Nuclear Receptors and Signaling (4 papers), Olfactory and Sensory Function Studies (4 papers) and Systemic Lupus Erythematosus Research (3 papers). RoseAnn Berlin is often cited by papers focused on Nuclear Receptors and Signaling (4 papers), Olfactory and Sensory Function Studies (4 papers) and Systemic Lupus Erythematosus Research (3 papers). RoseAnn Berlin collaborates with scholars based in United States, Japan and Serbia. RoseAnn Berlin's co-authors include Bruce T. Volpe, Harriet Baker, Betty Diamond, Patricio T. Huerta, Thomas A. Houpt, Jie Zhang, Sachiko Saino‐Saito, Czeslawa Kowal, Eric H. Chang and Kazuto Kobayashi and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Neuroscience and The Journal of Comparative Neurology.

In The Last Decade

RoseAnn Berlin

16 papers receiving 697 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
RoseAnn Berlin United States 13 227 182 157 156 155 16 704
Alexandria Hughes United States 8 142 0.6× 30 0.2× 169 1.1× 98 0.6× 214 1.4× 11 595
Rory Spence United States 11 109 0.5× 34 0.2× 142 0.9× 206 1.3× 206 1.3× 13 784
Takashi Kodama Japan 16 136 0.6× 45 0.2× 184 1.2× 14 0.1× 183 1.2× 29 731
M Carreras Italy 12 186 0.8× 81 0.4× 94 0.6× 34 0.2× 98 0.6× 34 776
Vanessa Brochard France 9 467 2.1× 40 0.2× 258 1.6× 142 0.9× 734 4.7× 11 1.3k
Sonia Cabrera Spain 8 203 0.9× 57 0.3× 522 3.3× 41 0.3× 266 1.7× 19 995
Iska Moxon‐Emre Canada 12 75 0.3× 15 0.1× 186 1.2× 78 0.5× 217 1.4× 30 792
Milena Laure‐Kamionowska Poland 13 129 0.6× 12 0.1× 342 2.2× 103 0.7× 99 0.6× 46 782
M. Tohyama Japan 9 153 0.7× 29 0.2× 173 1.1× 87 0.6× 36 0.2× 10 464
Noëlle Hanoteau France 6 168 0.7× 12 0.1× 229 1.5× 118 0.8× 177 1.1× 7 602

Countries citing papers authored by RoseAnn Berlin

Since Specialization
Citations

This map shows the geographic impact of RoseAnn Berlin's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by RoseAnn Berlin with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites RoseAnn Berlin more than expected).

Fields of papers citing papers by RoseAnn Berlin

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by RoseAnn Berlin. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by RoseAnn Berlin. The network helps show where RoseAnn Berlin may publish in the future.

Co-authorship network of co-authors of RoseAnn Berlin

This figure shows the co-authorship network connecting the top 25 collaborators of RoseAnn Berlin. A scholar is included among the top collaborators of RoseAnn Berlin based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with RoseAnn Berlin. RoseAnn Berlin is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

16 of 16 papers shown
1.
Mader, Simone, Lior Brimberg, An Vo, et al.. (2022). In utero exposure to maternal anti–aquaporin-4 antibodies alters brain vasculature and neural dynamics in male mouse offspring. Science Translational Medicine. 14(641). eabe9726–eabe9726. 10 indexed citations
2.
Huerta, Patricio T., Davide Comoletti, RoseAnn Berlin, et al.. (2020). In utero exposure to endogenous maternal polyclonal anti-Caspr2 antibody leads to behavioral abnormalities resembling autism spectrum disorder in male mice. Scientific Reports. 10(1). 14446–14446. 14 indexed citations
3.
Huerta, Patricio T., Sergio Robbiati, Tomás Huerta, et al.. (2016). Preclinical Models of Overwhelming Sepsis Implicate the Neural System that Encodes Contextual Fear Memory. Molecular Medicine. 22(1). 789–799. 22 indexed citations
4.
Brimberg, Lior, Simone Mader, Venkatesh Jeganathan, et al.. (2016). Caspr2-reactive antibody cloned from a mother of an ASD child mediates an ASD-like phenotype in mice. Molecular Psychiatry. 21(12). 1663–1671. 70 indexed citations
5.
Chang, Eric H., Bruce T. Volpe, Meggan Mackay, et al.. (2015). Selective Impairment of Spatial Cognition Caused by Autoantibodies to the N-Methyl-d-Aspartate Receptor. EBioMedicine. 2(7). 755–764. 63 indexed citations
6.
Faust, Thomas W., Eric H. Chang, Czeslawa Kowal, et al.. (2010). Neurotoxic lupus autoantibodies alter brain function through two distinct mechanisms. Proceedings of the National Academy of Sciences. 107(43). 18569–18574. 161 indexed citations
7.
Cave, John W., et al.. (2010). Differential Regulation of Dopaminergic Gene Expression byEr81. Journal of Neuroscience. 30(13). 4717–4724. 37 indexed citations
8.
Zhang, Jie, Annett M. Jacobi, Tao Wang, et al.. (2009). Polyreactive autoantibodies in systemic lupus erythematosus have pathogenic potential. Journal of Autoimmunity. 33(3-4). 270–274. 62 indexed citations
9.
Saino‐Saito, Sachiko, John W. Cave, Yosuke Akiba, et al.. (2007). ER81 and CaMKIV identify anatomically and phenotypically defined subsets of mouse olfactory bulb interneurons. The Journal of Comparative Neurology. 502(4). 485–496. 33 indexed citations
10.
Sasaki, Hayato, RoseAnn Berlin, & Harriet Baker. (2004). Transient expression of tyrosine hydroxylase promoter/reporter gene constructs in the olfactory epithelium of transgenic mice. Journal of Neurocytology. 33(6). 681–692. 2 indexed citations
11.
Saino‐Saito, Sachiko, Hayato Sasaki, Bruce T. Volpe, et al.. (2004). Differentiation of the dopaminergic phenotype in the olfactory system of neonatal and adult mice. The Journal of Comparative Neurology. 479(4). 389–398. 64 indexed citations
12.
Saino‐Saito, Sachiko, RoseAnn Berlin, & Harriet Baker. (2003). Dlx‐1 and Dlx‐2 expression in the adult mouse brain: Relationship to dopaminergic phenotypic regulation. The Journal of Comparative Neurology. 461(1). 18–30. 26 indexed citations
13.
Baker, Harriet, Nian Liu, Hong Sung Chun, et al.. (2001). Phenotypic Differentiation during Migration of Dopaminergic Progenitor Cells to the Olfactory Bulb. Journal of Neuroscience. 21(21). 8505–8513. 79 indexed citations
14.
Houpt, Thomas A. & RoseAnn Berlin. (1999). Rapid, Labile, and Protein Synthesis– Independent Short-Term Memory in Conditioned Taste Aversion. Learning & Memory. 6(1). 37–46. 44 indexed citations
15.
Houpt, Thomas A. & RoseAnn Berlin. (1998). Intracerebroventricular Angiotensin II Increases Intraoral Intake of Water in Rats. Peptides. 19(1). 171–173. 5 indexed citations
16.
Houpt, Thomas A., RoseAnn Berlin, & Gerard P. Smith. (1997). Subdiaphragmatic vagotomy does not attenuate c-Fos induction in the nucleus of the solitary tract after conditioned taste aversion expression. Brain Research. 747(1). 85–91. 12 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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