Ronald P. McCaffrey
- Molecular Biology top 10%
- Infectious Diseases top 5%
- Hematology top 2%
- Immunology top 5%
- Genetics top 2%
- Co-authors
- David BaltimoreRobertson ParkmanStanley M. MarksMichael E. OsbandDonna SmolerAlan M. SugarTimothy P. CooleyPatrick C. Kung
- Topics
- Acute Lymphoblastic Leukemia research (15 papers)Chronic Lymphocytic Leukemia Research (9 papers)HIV/AIDS drug development and treatment (7 papers)
- Cited by
- VirologyHematologyGenetics
- Partner nations
- United StatesUnited KingdomCanada
In The Last Decade
Ronald P. McCaffrey
58 papers receiving 2.3k citations
Hit Papers
Peers
Comparison fields: 5 of 116
- Molecular Biology 856
- Infectious Diseases 522
- Hematology 500
- Immunology 444
- Genetics 419
Countries citing papers authored by Ronald P. McCaffrey
This map shows the geographic impact of Ronald P. McCaffrey's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ronald P. McCaffrey with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ronald P. McCaffrey more than expected).
Fields of papers citing papers by Ronald P. McCaffrey
This network shows the impact of papers produced by Ronald P. McCaffrey. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ronald P. McCaffrey. The network helps show where Ronald P. McCaffrey may publish in the future.
Co-authorship network of co-authors of Ronald P. McCaffrey
This figure shows the co-authorship network connecting the top 25 collaborators of Ronald P. McCaffrey. A scholar is included among the top collaborators of Ronald P. McCaffrey based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ronald P. McCaffrey. Ronald P. McCaffrey is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 58 | |
| 2 | 102 | |
| 3 | 30 | |
| 4 | 95 | |
| 5 | 10 | |
| 6 | 43 | |
| 7 | 35 | |
| 8 | 6 | |
| 9 | 7 | |
| 10 | 15 | |
| 11 | 5 | |
| 12 | 180 | |
| 13 | 118 | |
| 14 | 0 | |
| 15 | 3 | |
| 16 | 111 | |
| 17 | 6 | |
| 18 | DNA polymerases in lymphoid cells. | 1 |
| 19 | 8 | |
| 20 | 25 |
About Ronald P. McCaffrey
Ronald P. McCaffrey is a scholar working on Genetics, Hematology and Virology, having authored 59 papers that have together received 2.6k indexed citations. Recurring topics across this work include Acute Lymphoblastic Leukemia research (15 papers), Chronic Lymphocytic Leukemia Research (9 papers) and HIV/AIDS drug development and treatment (7 papers). The work is most often cited by research in Virology (304 citations), Hematology (500 citations) and Genetics (419 citations). Ronald P. McCaffrey has collaborated with scholars based in United States, United Kingdom and Canada. Frequent co-authors include David Baltimore, Robertson Parkman, Stanley M. Marks, Michael E. Osband, Donna Smoler, Alan M. Sugar, Timothy P. Cooley, Patrick C. Kung, Philip T. Lavin and Howard A. Liebman. Their work appears in journals such as New England Journal of Medicine, Proceedings of the National Academy of Sciences and The Lancet.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.