Ronald G. Gill

5.2k citations
121 papers · 3.9k indexed · h-index 33
Topics
Pancreatic function and diabetes (61 papers)Diabetes and associated disorders (48 papers)Immune Cell Function and Interaction (39 papers)

In The Last Decade

Ronald G. Gill

121 papers receiving 3.8k citations

Peers

Ronald G. Gill
Comparison fields: 5 of 114
  • Surgery 1.9k
  • Genetics 1.6k
  • Immunology 1.5k
  • Endocrinology, Diabetes and Metabolism 726
  • Molecular Biology 666
Replace Paolo Monti with:
Paolo Monti Italy
Lina Lü United States
Владимир Субботин United States
Malin Flodström‐Tullberg Sweden
Agnes M. Azimzadeh United States
Elena Urcelay Spain
Philippe Druet France
Jan Ehrchen Germany
Linrong Lu China
Ronald G. Gill relative to Paolo Monti Italy Paolo Monti's profile →
Citations per field
00.5×1.5×2.4×
Paolo Monti · 1×
Citations per year

Countries citing papers authored by Ronald G. Gill

Since Specialization
Citations

This map shows the geographic impact of Ronald G. Gill's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ronald G. Gill with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ronald G. Gill more than expected).

Fields of papers citing papers by Ronald G. Gill

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ronald G. Gill. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ronald G. Gill. The network helps show where Ronald G. Gill may publish in the future.

Co-authorship network of co-authors of Ronald G. Gill

This figure shows the co-authorship network connecting the top 25 collaborators of Ronald G. Gill. A scholar is included among the top collaborators of Ronald G. Gill based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ronald G. Gill. Ronald G. Gill is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
#WorkIndexed citations
1 9
2 80
3 2
4 23
5 54
6 8
7 21
8 35
9 211
10 88
11 35
12 32
13 74
14 32
15 4
16
Immunologically mediated endocrine diseases
9
17 77
18 28
19 2
20 11

About Ronald G. Gill

Ronald G. Gill is a scholar working on Transplantation, Immunology and Genetics, having authored 121 papers that have together received 3.9k indexed citations. Recurring topics across this work include Pancreatic function and diabetes (61 papers), Diabetes and associated disorders (48 papers) and Immune Cell Function and Interaction (39 papers). The work is most often cited by research in Transplantation (384 citations), Immunology (1.5k citations) and Genetics (1.6k citations). Ronald G. Gill has collaborated with scholars based in United States, Canada and Australia. Frequent co-authors include Marilyne Coulombe, Joshua Beilke, Kevin J. Lafferty, Magdalene So, Mark R. Nicolls, Stanley Falkow, Dylan Daniel, Dale R. Wegmann, Nanette C. Schloot and Todd J. Grazia. Their work appears in journals such as Proceedings of the National Academy of Sciences, Journal of Clinical Investigation and Nature Medicine.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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