Ronald B. Saulnier

759 total citations
8 papers, 646 citations indexed

About

Ronald B. Saulnier is a scholar working on Molecular Biology, Immunology and Allergy and Immunology. According to data from OpenAlex, Ronald B. Saulnier has authored 8 papers receiving a total of 646 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Molecular Biology, 3 papers in Immunology and Allergy and 2 papers in Immunology. Recurrent topics in Ronald B. Saulnier's work include Protein Kinase Regulation and GTPase Signaling (4 papers), Cell Adhesion Molecules Research (3 papers) and Signaling Pathways in Disease (2 papers). Ronald B. Saulnier is often cited by papers focused on Protein Kinase Regulation and GTPase Signaling (4 papers), Cell Adhesion Molecules Research (3 papers) and Signaling Pathways in Disease (2 papers). Ronald B. Saulnier collaborates with scholars based in Canada, United States and Ukraine. Ronald B. Saulnier's co-authors include Shoukat Dedhar, David W. Litchfield, Greg Vilk, Raymond B. Nagle, Christopher M. Overall, Cunjie Zhang, Denis G. Bosc, Rebecca St. Pierre, David A. Prober and David A. Canton and has published in prestigious journals such as Journal of Biological Chemistry, The Journal of Cell Biology and Experimental Cell Research.

In The Last Decade

Ronald B. Saulnier

8 papers receiving 637 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Ronald B. Saulnier Canada 8 410 257 170 147 104 8 646
Jinli Chang United States 6 276 0.7× 249 1.0× 156 0.9× 127 0.9× 100 1.0× 8 504
Takahiro Tokuhara Japan 8 225 0.5× 188 0.7× 279 1.6× 89 0.6× 120 1.2× 8 532
Ta-Jen Liu United States 16 566 1.4× 128 0.5× 231 1.4× 87 0.6× 143 1.4× 16 785
Benjamin Caleb United States 7 526 1.3× 121 0.5× 232 1.4× 288 2.0× 149 1.4× 8 827
Kym L. Stanley Australia 9 588 1.4× 134 0.5× 310 1.8× 146 1.0× 123 1.2× 10 893
Annette Bistrup United States 10 619 1.5× 273 1.1× 119 0.7× 383 2.6× 80 0.8× 10 901
Andreas Enns Germany 9 506 1.2× 122 0.5× 289 1.7× 143 1.0× 129 1.2× 9 889
Carl Nyberg United States 6 278 0.7× 297 1.2× 145 0.9× 185 1.3× 86 0.8× 8 521
Sabine Gesierich Germany 7 519 1.3× 114 0.4× 100 0.6× 102 0.7× 207 2.0× 7 650
Rajeshwari R. Valiathan United States 8 313 0.8× 444 1.7× 250 1.5× 186 1.3× 98 0.9× 10 722

Countries citing papers authored by Ronald B. Saulnier

Since Specialization
Citations

This map shows the geographic impact of Ronald B. Saulnier's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ronald B. Saulnier with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ronald B. Saulnier more than expected).

Fields of papers citing papers by Ronald B. Saulnier

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ronald B. Saulnier. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ronald B. Saulnier. The network helps show where Ronald B. Saulnier may publish in the future.

Co-authorship network of co-authors of Ronald B. Saulnier

This figure shows the co-authorship network connecting the top 25 collaborators of Ronald B. Saulnier. A scholar is included among the top collaborators of Ronald B. Saulnier based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ronald B. Saulnier. Ronald B. Saulnier is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

8 of 8 papers shown
1.
Saulnier, Ronald B., et al.. (2002). Interactions between Protein Kinase CK2 and Pin1. Journal of Biological Chemistry. 277(25). 23054–23064. 74 indexed citations
2.
Litchfield, David W., Denis G. Bosc, David A. Canton, et al.. (2001). Functional specialization of CK2 isoforms and characterization of isoform-specific binding partners. PubMed. 227(1-2). 21–29. 33 indexed citations
3.
Litchfield, David W., Denis G. Bosc, David A. Canton, et al.. (2001). Functional specialization of CK2 isoforms and characterization of isoform-specific binding partners. Molecular and Cellular Biochemistry. 227(1-2). 21–29. 42 indexed citations
4.
Bosc, Denis G., Kevin C. Graham, Ronald B. Saulnier, et al.. (2000). Identification and Characterization of CKIP-1, a Novel Pleckstrin Homology Domain-containing Protein That Interacts with Protein Kinase CK2. Journal of Biological Chemistry. 275(19). 14295–14306. 94 indexed citations
5.
Vilk, Greg, Ronald B. Saulnier, Rebecca St. Pierre, & David W. Litchfield. (1999). Inducible Expression of Protein Kinase CK2 in Mammalian Cells. Journal of Biological Chemistry. 274(20). 14406–14414. 89 indexed citations
6.
Dedhar, Shoukat, Ronald B. Saulnier, Raymond B. Nagle, & Christopher M. Overall. (1993). Specific alterations in the expression of ?3?1 and ?6?4 integrins in highly invasive and metastatic variants of human prostate carcinoma cells selected by in vitro invasion through reconstituted basement membrane. Clinical & Experimental Metastasis. 11(5). 391–400. 114 indexed citations
7.
Dedhar, Shoukat, et al.. (1992). Identification and characterization of a novel high-molecular-weight form of the integrin α3 subunit. Experimental Cell Research. 203(1). 270–275. 12 indexed citations
8.
Dedhar, Shoukat & Ronald B. Saulnier. (1990). Alterations in integrin receptor expression on chemically transformed human cells: specific enhancement of laminin and collagen receptor complexes.. The Journal of Cell Biology. 110(2). 481–489. 188 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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