Romina Inès Cervigni

419 total citations
8 papers, 323 citations indexed

About

Romina Inès Cervigni is a scholar working on Cell Biology, Molecular Biology and Surgery. According to data from OpenAlex, Romina Inès Cervigni has authored 8 papers receiving a total of 323 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Cell Biology, 6 papers in Molecular Biology and 1 paper in Surgery. Recurrent topics in Romina Inès Cervigni's work include Cellular transport and secretion (8 papers), Microtubule and mitosis dynamics (6 papers) and Retinal Development and Disorders (3 papers). Romina Inès Cervigni is often cited by papers focused on Cellular transport and secretion (8 papers), Microtubule and mitosis dynamics (6 papers) and Retinal Development and Disorders (3 papers). Romina Inès Cervigni collaborates with scholars based in Italy, United States and Japan. Romina Inès Cervigni's co-authors include Maria Luisa Barretta, Antonino Colanzi, Daniela Corda, Daniela Spano, Flavia Valtorta, Fabio Benfenati, Pierluigi Valente, Anna Corradi, Fabrizia Claudia Guarnieri and Pia Rossi and has published in prestigious journals such as Nature Communications, Journal of Cell Science and FEBS Letters.

In The Last Decade

Romina Inès Cervigni

8 papers receiving 323 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Romina Inès Cervigni Italy	8	196	181	55	51	44	8	323
Ivan Litvinenko Bulgaria	9	67 0.3×	155 0.9×	68 1.2×	87 1.7×	13 0.3×	21	282
Levi Barse United States	8	50 0.3×	87 0.5×	41 0.7×	73 1.4×	36 0.8×	14	253
M. Natalia Zanetti Argentina	10	146 0.7×	217 1.2×	32 0.6×	56 1.1×	16 0.4×	11	382
Yujuan Hong China	7	110 0.6×	168 0.9×	78 1.4×	42 0.8×	10 0.2×	9	322
Ippei Okada Japan	4	149 0.8×	210 1.2×	255 4.6×	120 2.4×	122 2.8×	5	444
Emily Tuttle United States	7	62 0.3×	211 1.2×	113 2.1×	42 0.8×	32 0.7×	10	314
Antonella Sferra Italy	8	78 0.4×	151 0.8×	43 0.8×	47 0.9×	10 0.2×	12	246
Nicola Vanni Italy	5	66 0.3×	145 0.8×	122 2.2×	33 0.6×	59 1.3×	6	278
Giulia Massaro United Kingdom	10	80 0.4×	189 1.0×	86 1.6×	83 1.6×	29 0.7×	17	391
Sébastien Moutton France	12	45 0.2×	212 1.2×	211 3.8×	64 1.3×	24 0.5×	17	367

Countries citing papers authored by Romina Inès Cervigni

Since Specialization

Citations

This map shows the geographic impact of Romina Inès Cervigni's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Romina Inès Cervigni with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Romina Inès Cervigni more than expected).

Fields of papers citing papers by Romina Inès Cervigni

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Romina Inès Cervigni. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Romina Inès Cervigni. The network helps show where Romina Inès Cervigni may publish in the future.

Co-authorship network of co-authors of Romina Inès Cervigni

This figure shows the co-authorship network connecting the top 25 collaborators of Romina Inès Cervigni. A scholar is included among the top collaborators of Romina Inès Cervigni based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Romina Inès Cervigni. Romina Inès Cervigni is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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8 of 8 papers shown

1.

Cervigni, Romina Inès, Pierluigi Valente, Anna Corradi, et al.. (2020). Proline-rich transmembrane protein 2 (PRRT2) regulates the actin cytoskeleton during synaptogenesis. Cell Death and Disease. 11(10). 856–856. 9 indexed citations

2.

Barretta, Maria Luisa, Daniela Spano, Chiara DˈAmbrosio, et al.. (2016). Aurora-A recruitment and centrosomal maturation are regulated by a Golgi-activated pool of Src during G2. Nature Communications. 7(1). 11727–11727. 33 indexed citations

3.

Valente, Pierluigi, Enrico Castroflorio, Pia Rossi, et al.. (2016). PRRT2 Is a Key Component of the Ca2+-Dependent Neurotransmitter Release Machinery. Cell Reports. 15(1). 117–131. 109 indexed citations

4.

Cervigni, Romina Inès, Maria Luisa Barretta, Daniela Spano, et al.. (2015). JNK2 controls fragmentation of the Golgi complex and the G2/M transition through phosphorylation of GRASP65. Journal of Cell Science. 128(12). 2249–2260. 50 indexed citations

5.

Corda, Daniela, Maria Luisa Barretta, Romina Inès Cervigni, & Antonino Colanzi. (2012). Golgi complex fragmentation in G2/M transition: An organelle‐based cell‐cycle checkpoint. IUBMB Life. 64(8). 661–670. 45 indexed citations

6.

Cervigni, Romina Inès, et al.. (2011). The role of Aurora-A kinase in the Golgi-dependent control of mitotic entry. PubMed. 1(2). 61–65. 11 indexed citations

7.

Cervigni, Romina Inès, et al.. (2010). Golgi Partitioning Controls Mitotic Entry through Aurora-A Kinase. Molecular Biology of the Cell. 21(21). 3708–3721. 37 indexed citations

8.

Cervigni, Romina Inès, et al.. (2009). Mitotic inheritance of the Golgi complex. FEBS Letters. 583(23). 3857–3862. 29 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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