Roberto Foà
- Genetics top 2%
- Chronic Lymphocytic Leukemia Research 18
- Hematology top 2%
- Hematopoietic Stem Cell Transplantation 11
- Acute Myeloid Leukemia Research 11
- Chronic Myeloid Leukemia Treatments 7
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- Lymphoma Diagnosis and Treatment 13
- Immunology top 5%
- Immunodeficiency and Autoimmune Disorders 8
- Immune Cell Function and Interaction 5
- Oncology top 10%
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- Acute Lymphoblastic Leukemia research 14
- Co-authors
- F. LauriaDaniel CatovskyD CatovskyS TuraNicola MigoneMassimo BrecciaGiovanni De GiudiciGiulia Casorati
- Partner nations
- ItalyUnited KingdomFrance
In The Last Decade
Roberto Foà
60 papers receiving 1.2k citations
Peers
Comparison fields: 5 of 78
- Genetics 479
- Hematology 455
- Pathology and Forensic Medicine 461
- Immunology 497
- Oncology 269
Countries citing papers authored by Roberto Foà
This map shows the geographic impact of Roberto Foà's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Roberto Foà with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Roberto Foà more than expected).
Fields of papers citing papers by Roberto Foà
This network shows the impact of papers produced by Roberto Foà. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Roberto Foà. The network helps show where Roberto Foà may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Roberto Foà, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2019 | 10 | |
| 2 | 2019 | 58 | |
| 3 | 2017 | 17 | |
| 4 | 2015 | 27 | |
| 5 | 2014 | 10 | |
| 6 | 2014 | 10 | |
| 7 | 2013 | 20 | |
| 8 | 2013 | 3 | |
| 9 | 2012 | 3 | |
| 10 | 2011 | 13 | |
| 11 | 2011 | 81 | |
| 12 | 2008 | 45 | |
| 13 | 2006 | 33 | |
| 14 | 2004 | 5 | |
| 15 | 2000 | 9 | |
| 16 | 1991 | 25 | |
| 17 | 1991 | 4 | |
| 18 | 1988 | 14 | |
| 19 | Reduced T lymphocyte colonies in B chronic lymphocytic leukaemia. III. Evidence of a proliferative abnormality of the T helper cell population. | 1982 | 6 |
| 20 | 1979 | 17 |
About Roberto Foà
Roberto Foà is a scholar working on Hematology, Genetics and Immunology, having authored 60 papers that have together received 1.3k indexed citations. Recurring topics across this work include Chronic Lymphocytic Leukemia Research (18 papers), Acute Lymphoblastic Leukemia research (14 papers), Lymphoma Diagnosis and Treatment (13 papers), Hematopoietic Stem Cell Transplantation (11 papers), Acute Myeloid Leukemia Research (11 papers), Immunodeficiency and Autoimmune Disorders (8 papers), Chronic Myeloid Leukemia Treatments (7 papers) and Immune Cell Function and Interaction (5 papers). The work is most often cited by research in Genetics (479 citations), Hematology (455 citations) and Pathology and Forensic Medicine (461 citations). Roberto Foà has collaborated with scholars based in Italy, United Kingdom and France. Frequent co-authors include F. Lauria, Daniel Catovsky, D Catovsky, S Tura, Nicola Migone, Massimo Breccia, Giovanni De Giudici, Giulia Casorati, Paola Francia di Celle and F Gavosto. Their work appears in journals such as Blood, British Journal of Haematology, Haematologica, European Journal Of Haematology and Cancer.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.