Robert W. McLachlan

523 total citations
7 papers, 439 citations indexed

About

Robert W. McLachlan is a scholar working on Cell Biology, Molecular Biology and Physiology. According to data from OpenAlex, Robert W. McLachlan has authored 7 papers receiving a total of 439 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Cell Biology, 6 papers in Molecular Biology and 1 paper in Physiology. Recurrent topics in Robert W. McLachlan's work include Wnt/β-catenin signaling in development and cancer (6 papers), Cellular Mechanics and Interactions (5 papers) and Hippo pathway signaling and YAP/TAZ (4 papers). Robert W. McLachlan is often cited by papers focused on Wnt/β-catenin signaling in development and cancer (6 papers), Cellular Mechanics and Interactions (5 papers) and Hippo pathway signaling and YAP/TAZ (4 papers). Robert W. McLachlan collaborates with scholars based in Australia, United Kingdom and France. Robert W. McLachlan's co-authors include Alpha S. Yap, Guillermo A. Gómez, Falak Helwani, Astrid Kraemer, Éva Kovács, Suzie Verma, Gang Ren, Scott A. Weed, Robert G. Parton and Katharina Gaus and has published in prestigious journals such as Journal of Biological Chemistry, Trends in Cell Biology and Molecular Biology of the Cell.

In The Last Decade

Robert W. McLachlan

7 papers receiving 435 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Robert W. McLachlan Australia 7 306 257 98 38 33 7 439
Eeva Kaisa Rajakylä Finland 9 298 1.0× 387 1.5× 29 0.3× 21 0.6× 20 0.6× 11 571
Elisa Vitiello France 9 263 0.9× 245 1.0× 32 0.3× 55 1.4× 15 0.5× 9 424
Tom H. Millard United Kingdom 9 250 0.8× 223 0.9× 54 0.6× 25 0.7× 72 2.2× 11 430
Masaru Mitsushima Japan 12 298 1.0× 344 1.3× 69 0.7× 14 0.4× 34 1.0× 13 504
Alexis J. Lomakin Russia 8 244 0.8× 224 0.9× 23 0.2× 52 1.4× 23 0.7× 12 402
Ivar Noordstra Australia 10 288 0.9× 259 1.0× 20 0.2× 32 0.8× 26 0.8× 15 433
Yulia Artemenko United States 9 217 0.7× 182 0.7× 26 0.3× 64 1.7× 31 0.9× 12 374
Petr Folk Czechia 14 166 0.5× 362 1.4× 45 0.5× 52 1.4× 17 0.5× 28 526
Raluca Gagescu United States 7 287 0.9× 345 1.3× 32 0.3× 10 0.3× 34 1.0× 19 497
Ron D. M. Soede Netherlands 11 228 0.7× 223 0.9× 125 1.3× 46 1.2× 141 4.3× 12 523

Countries citing papers authored by Robert W. McLachlan

Since Specialization
Citations

This map shows the geographic impact of Robert W. McLachlan's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Robert W. McLachlan with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Robert W. McLachlan more than expected).

Fields of papers citing papers by Robert W. McLachlan

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Robert W. McLachlan. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Robert W. McLachlan. The network helps show where Robert W. McLachlan may publish in the future.

Co-authorship network of co-authors of Robert W. McLachlan

This figure shows the co-authorship network connecting the top 25 collaborators of Robert W. McLachlan. A scholar is included among the top collaborators of Robert W. McLachlan based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Robert W. McLachlan. Robert W. McLachlan is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

7 of 7 papers shown
1.
Teo, Jessica L., Vanesa M. Tomatis, Anne K. Lagendijk, et al.. (2020). Src kinases relax adherens junctions between the neighbors of apoptotic cells to permit apical extrusion. Molecular Biology of the Cell. 31(23). 2557–2569. 18 indexed citations
2.
Gómez, Guillermo A., Robert W. McLachlan, Selwin K. Wu, et al.. (2015). An RPTPα/Src family kinase/Rap1 signaling module recruits myosin IIB to support contractile tension at apical E-cadherin junctions. Molecular Biology of the Cell. 26(7). 1249–1262. 38 indexed citations
3.
Gómez, Guillermo A., Robert W. McLachlan, & Alpha S. Yap. (2011). Productive tension: force-sensing and homeostasis of cell–cell junctions. Trends in Cell Biology. 21(9). 499–505. 118 indexed citations
4.
McLachlan, Robert W. & Alpha S. Yap. (2010). Protein tyrosine phosphatase activity is necessary for E‐cadherin‐activated Src signaling. Cytoskeleton. 68(1). 32–43. 29 indexed citations
5.
Ren, Gang, Falak Helwani, Suzie Verma, et al.. (2009). Cortactin Is a Functional Target of E-cadherin-activated Src Family Kinases in MCF7 Epithelial Monolayers. Journal of Biological Chemistry. 284(28). 18913–18922. 40 indexed citations
6.
McLachlan, Robert W. & Alpha S. Yap. (2007). Not so simple: the complexity of phosphotyrosine signaling at cadherin adhesive contacts. Journal of Molecular Medicine. 85(6). 545–554. 72 indexed citations
7.
McLachlan, Robert W., Astrid Kraemer, Falak Helwani, Éva Kovács, & Alpha S. Yap. (2007). E-Cadherin Adhesion Activates c-Src Signaling at Cell–Cell Contacts. Molecular Biology of the Cell. 18(8). 3214–3223. 124 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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2026