Robert M. Bare

720 total citations
15 papers, 601 citations indexed

About

Robert M. Bare is a scholar working on Nutrition and Dietetics, Health, Toxicology and Mutagenesis and Molecular Biology. According to data from OpenAlex, Robert M. Bare has authored 15 papers receiving a total of 601 indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Nutrition and Dietetics, 9 papers in Health, Toxicology and Mutagenesis and 4 papers in Molecular Biology. Recurrent topics in Robert M. Bare's work include Trace Elements in Health (10 papers), Heavy Metal Exposure and Toxicity (9 papers) and Viral gastroenteritis research and epidemiology (2 papers). Robert M. Bare is often cited by papers focused on Trace Elements in Health (10 papers), Heavy Metal Exposure and Toxicity (9 papers) and Viral gastroenteritis research and epidemiology (2 papers). Robert M. Bare collaborates with scholars based in United States, Poland and Italy. Robert M. Bare's co-authors include Michael P. Waalkes, Timothy P. Coogan, John R. Henneman, Martin L. Wenk, Sabine Rehm, Lionel A. Poirier, Charles W. Riggs, Deborah E. Devor, Kazimierz S. Kasprzak and Bhalchandra A. Diwan and has published in prestigious journals such as JNCI Journal of the National Cancer Institute, International Journal of Cancer and Carcinogenesis.

In The Last Decade

Robert M. Bare

15 papers receiving 573 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Robert M. Bare United States 12 397 276 124 79 69 15 601
Goyer Ra 15 651 1.6× 459 1.7× 134 1.1× 67 0.8× 106 1.5× 22 975
Stephen B. Waters United States 13 513 1.3× 207 0.8× 402 3.2× 39 0.5× 61 0.9× 16 953
Shugene Lynn Taiwan 9 263 0.7× 167 0.6× 349 2.8× 130 1.6× 35 0.5× 12 718
Shailaja D. Divekar United States 9 467 1.2× 159 0.6× 103 0.8× 69 0.9× 137 2.0× 10 770
Max Costa United States 16 275 0.7× 132 0.5× 346 2.8× 133 1.7× 39 0.6× 25 735
Xianglin Shi United States 8 289 0.7× 109 0.4× 150 1.2× 67 0.8× 75 1.1× 8 620
Max Costa United States 11 248 0.6× 165 0.6× 332 2.7× 218 2.8× 48 0.7× 15 721
Yu Mei Hsueh Taiwan 6 450 1.1× 146 0.5× 194 1.6× 50 0.6× 81 1.2× 9 742
H. J. Wiegand Germany 12 347 0.9× 86 0.3× 100 0.8× 87 1.1× 40 0.6× 22 533
Nicole V. Soucy United States 10 217 0.5× 109 0.4× 262 2.1× 67 0.8× 26 0.4× 15 670

Countries citing papers authored by Robert M. Bare

Since Specialization
Citations

This map shows the geographic impact of Robert M. Bare's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Robert M. Bare with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Robert M. Bare more than expected).

Fields of papers citing papers by Robert M. Bare

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Robert M. Bare. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Robert M. Bare. The network helps show where Robert M. Bare may publish in the future.

Co-authorship network of co-authors of Robert M. Bare

This figure shows the co-authorship network connecting the top 25 collaborators of Robert M. Bare. A scholar is included among the top collaborators of Robert M. Bare based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Robert M. Bare. Robert M. Bare is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

15 of 15 papers shown
1.
Cheng, Robert Y.S., Ailian Zhao, W. Gregory Alvord, et al.. (2003). Gene expression dose-response changes in microarrays after exposure of human peripheral lung epithelial cells to nickel(II). Toxicology and Applied Pharmacology. 191(1). 22–39. 33 indexed citations
2.
Salnikow, Konstantin, et al.. (2003). Reduced Fhit protein expression in nickel-transformed mouse cells and in nickel-induced murine sarcomas. Molecular and Cellular Biochemistry. 255(1-2). 195–202. 20 indexed citations
3.
Karaczyn, Aldona, Wojciech Bal, Susan L. North, et al.. (2003). The Octapeptidic End of the C-Terminal Tail of Histone H2A Is Cleaved Off in Cells Exposed to Carcinogenic Nickel(II). Chemical Research in Toxicology. 16(12). 1555–1559. 40 indexed citations
4.
Shimada, Hideaki, Robert M. Bare, James F. Hochadel, & Michael P. Waalkes. (1997). Testosterone pretreatment mitigates cadmium toxicity in male C57 mice but not in C3H mice. Toxicology. 116(1-3). 183–191. 12 indexed citations
5.
Bare, Robert M., et al.. (1996). Metallothionein gene expression in testicular interstitial cells and liver of rats treated with cadmium. Toxicology. 107(2). 121–130. 35 indexed citations
6.
Coogan, Timothy P., et al.. (1994). Enhanced metallothionein gene expression is associated with protection from cadmium‐induced genotoxicity in cultured rat liver cells. Journal of Toxicology and Environmental Health. 41(2). 233–245. 42 indexed citations
7.
Coogan, Timothy P., Robert M. Bare, & Michael P. Waalkes. (1992). Cadmium-induced DNA strand damage in cultured liver cells: Reduction in cadmium genotoxicity following zinc pretreatment. Toxicology and Applied Pharmacology. 113(2). 227–233. 105 indexed citations
8.
Martelletti, Paolo, A. Andréoli, Guido F. Sasso, et al.. (1991). Bioelectrical Impedance Assay (BIA) of Total Body Composition in Alcohol‐Induced Migraine Patients. Preliminary Report.. Headache The Journal of Head and Face Pain. 31(1). 41–45. 2 indexed citations
9.
Waalkes, Michael P., Bhalchandra A. Diwan, Christopher M. Weghorst, et al.. (1991). Anticarcinogenic effects of cadmium in B6C3F1 mouse liver and lung. Toxicology and Applied Pharmacology. 110(2). 327–335. 30 indexed citations
10.
Kasprzak, Kazimierz S. & Robert M. Bare. (1989). In vitro polymerization of histones by carcinogenic nickel compounds. Carcinogenesis. 10(3). 621–624. 25 indexed citations
11.
Waalkes, Michael P., Sabine Rehm, Charles W. Riggs, et al.. (1989). Cadmium carcinogenesis in male Wistar [Crl:(WI)BR] rats: dose-response analysis of effects of zinc on tumor induction in the prostate, in the testes, and at the injection site.. PubMed. 49(15). 4282–8. 89 indexed citations
12.
Waalkes, Michael P., et al.. (1988). Increased metallothionein gene expression in 5-AZA-2′-deoxycytidine-induced resistance to cadmium cytotoxicity. Chemico-Biological Interactions. 66(3-4). 189–204. 26 indexed citations
13.
Waalkes, Michael P., Sabine Rehm, Charles W. Riggs, et al.. (1988). Cadmium carcinogenesis in male Wistar [Crl:(WI)BR] rats: dose-response analysis of tumor induction in the prostate and testes and at the injection site.. PubMed. 48(16). 4656–63. 139 indexed citations
14.
Bare, Robert M., et al.. (1979). Prevention of spontaneous leukemia in mice by oncornavirus immunization. International Journal of Cancer. 23(2). 269–273. 2 indexed citations
15.
Gilden, Raymond V., et al.. (1979). Prevention of Spontaneous Leukemia in AKR Mice by Passive Immunization and Type Specificity of This Protection<xref ref-type="fn" rid="FN2">2</xref>. JNCI Journal of the National Cancer Institute. 62(4). 943–5. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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