Robert J. Waldbillig

1.3k total citations
41 papers, 1.1k citations indexed

About

Robert J. Waldbillig is a scholar working on Endocrinology, Diabetes and Metabolism, Molecular Biology and Cellular and Molecular Neuroscience. According to data from OpenAlex, Robert J. Waldbillig has authored 41 papers receiving a total of 1.1k indexed citations (citations by other indexed papers that have themselves been cited), including 15 papers in Endocrinology, Diabetes and Metabolism, 13 papers in Molecular Biology and 8 papers in Cellular and Molecular Neuroscience. Recurrent topics in Robert J. Waldbillig's work include Growth Hormone and Insulin-like Growth Factors (14 papers), Regulation of Appetite and Obesity (8 papers) and Retinal Development and Disorders (8 papers). Robert J. Waldbillig is often cited by papers focused on Growth Hormone and Insulin-like Growth Factors (14 papers), Regulation of Appetite and Obesity (8 papers) and Retinal Development and Disorders (8 papers). Robert J. Waldbillig collaborates with scholars based in United States and Kuwait. Robert J. Waldbillig's co-authors include Gerald J. Chader, Timothy J. Bartness, Timothy J. Schoen, R. Theodore Fletcher, Derek LeRoith, Dagmar Arnold, B. Glenn Stanley, Michael O’Callaghan, Brenda E. Jones and David C. Beebe and has published in prestigious journals such as Brain Research, Biochemical and Biophysical Research Communications and Endocrinology.

In The Last Decade

Robert J. Waldbillig

41 papers receiving 1.1k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Robert J. Waldbillig United States 22 441 297 276 206 191 41 1.1k
Koichi Takatsuji Japan 18 369 0.8× 449 1.5× 22 0.1× 240 1.2× 49 0.3× 47 937
A-M O'Carroll United States 9 483 1.1× 469 1.6× 175 0.6× 165 0.8× 9 0.0× 11 1.4k
Dong Won Kim United States 22 577 1.3× 215 0.7× 53 0.2× 267 1.3× 33 0.2× 72 1.5k
Ch. Pilgrim Germany 21 510 1.2× 491 1.7× 43 0.2× 194 0.9× 10 0.1× 59 1.5k
Shami Kanekar United States 15 887 2.0× 449 1.5× 18 0.1× 93 0.5× 86 0.5× 20 1.4k
Ana L. Miranda‐Angulo Colombia 7 474 1.1× 162 0.5× 78 0.3× 336 1.6× 12 0.1× 9 1.0k
Xavier Bonnefont France 20 362 0.8× 273 0.9× 266 1.0× 506 2.5× 8 0.0× 31 1.3k
Koji Ohno Japan 23 791 1.8× 1.1k 3.7× 15 0.1× 118 0.6× 62 0.3× 58 1.8k
Shimpei Higo Japan 20 220 0.5× 399 1.3× 378 1.4× 161 0.8× 10 0.1× 36 1.3k
Lynne Shanley United Kingdom 13 311 0.7× 281 0.9× 26 0.1× 323 1.6× 13 0.1× 13 918

Countries citing papers authored by Robert J. Waldbillig

Since Specialization
Citations

This map shows the geographic impact of Robert J. Waldbillig's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Robert J. Waldbillig with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Robert J. Waldbillig more than expected).

Fields of papers citing papers by Robert J. Waldbillig

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Robert J. Waldbillig. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Robert J. Waldbillig. The network helps show where Robert J. Waldbillig may publish in the future.

Co-authorship network of co-authors of Robert J. Waldbillig

This figure shows the co-authorship network connecting the top 25 collaborators of Robert J. Waldbillig. A scholar is included among the top collaborators of Robert J. Waldbillig based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Robert J. Waldbillig. Robert J. Waldbillig is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Bitar, Milad S., C.W.T. Pilcher, Islam Ullah Khan, & Robert J. Waldbillig. (1997). Diabetes-induced suppression of IGF-1 and its receptor mRNA levels in rat superior cervical ganglia. Diabetes Research and Clinical Practice. 38(2). 73–80. 37 indexed citations
2.
McMurtry, John P., et al.. (1996). Developmental Changes in Serum Insulin-Like Growth Factor-I and Insulin-Like Growth Factor Binding Proteins in the Turkey Embryo. Poultry Science. 75(4). 563–569. 12 indexed citations
3.
Schoen, Timothy J., et al.. (1995). Vitreous and aqueous humors contain a latent proteinase activity that abolishes IGF binding to specific IGF binding proteins. Current Eye Research. 14(7). 555–561. 6 indexed citations
4.
Schoen, Timothy J., Krzysztof Mazuruk, Robert J. Waldbillig, et al.. (1995). Cloning and characterization of a chick embryo cDNA and gene for IGF-binding protein-2. Journal of Molecular Endocrinology. 15(1). 49–59. 48 indexed citations
5.
Ikeda, Tsunehiko, Robert J. Waldbillig, & Donald G. Puro. (1995). Truncation of IGF-I yields two mitogens for retinal Müller glial cells. Brain Research. 686(1). 87–92. 40 indexed citations
6.
7.
Waldbillig, Robert J., Brenda E. Jones, Timothy J. Schoen, et al.. (1994). Vitreal insulin-like growth factor binding proteins (IGFBPs) are increased in human and animal diabetics. Current Eye Research. 13(7). 539–546. 29 indexed citations
8.
9.
Waldbillig, Robert J., Timothy J. Schoen, Gerald J. Chader, & Bruce A. Pfeffer. (1992). Monkey retinal pigment epithelial cells in vitro synthesize, secrete, and degrade insulin‐like growth factor binding proteins. Journal of Cellular Physiology. 150(1). 76–83. 15 indexed citations
10.
11.
Waldbillig, Robert J., Dagmar Arnold, R. Theodore Fletcher, & Gerald J. Chader. (1991). Insulin and IGF-I binding in developing chick neural retina and pigment epithelium: A characterization of binding and structural differences. Experimental Eye Research. 53(1). 13–22. 41 indexed citations
12.
Waldbillig, Robert J., Bruce A. Pfeffer, Timothy J. Schoen, et al.. (1991). Evidence for an Insulin‐Like Growth Factor Autocrine‐Paracrine System in the Retinal Photoreceptor‐Pigment Epithelial Cell Complex. Journal of Neurochemistry. 57(5). 1522–1533. 75 indexed citations
13.
Waldbillig, Robert J., et al.. (1988). Retinal insulin receptors: localization using a polyclonal anti-insulin receptor antibody. Brain Research. 443(1-2). 389–394. 48 indexed citations
14.
Waldbillig, Robert J. & Gerald J. Chader. (1988). Anomalous insulin-binding activity in the bovine neural retina: A possible mechanism for regulation of receptor binding specificity. Biochemical and Biophysical Research Communications. 151(3). 1105–1112. 14 indexed citations
15.
Waldbillig, Robert J., R. Theodore Fletcher, Robert L. Somers, & Gerald J. Chader. (1988). IGF-I receptors in the bovine neural retina: Structure, kinase activity and comparison with retinal insulin receptors. Experimental Eye Research. 47(4). 587–607. 48 indexed citations
16.
Waldbillig, Robert J., et al.. (1987). Retinal insulin receptors. 1. Structural heterogeneity and functional characterization. Experimental Eye Research. 45(6). 823–835. 30 indexed citations
17.
Waldbillig, Robert J., et al.. (1987). Retinal insulin receptors. 2. Characterization and insulininduced tyrosine kinase activity in bovine retinal rod outer segments. Experimental Eye Research. 45(6). 837–844. 35 indexed citations
18.
Waldbillig, Robert J. & Timothy J. Bartness. (1987). Insulin-induced drinking: an analysis of the involvement of renal angiotensin II and insulin-induced changes in plasma volume. Pharmacology Biochemistry and Behavior. 28(4). 447–452. 2 indexed citations
19.
Bartness, Timothy J. & Robert J. Waldbillig. (1984). Dietary self-selection in intact, ovariectomized, and estradiol-treated female rats.. Behavioral Neuroscience. 98(1). 125–137. 19 indexed citations
20.
Bartness, Timothy J. & Robert J. Waldbillig. (1984). Cholecystokinin-induced suppression of feeding: An evaluation of the generality of gustatory-cholecystokinin interactions. Physiology & Behavior. 32(3). 409–415. 19 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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