Robert J. Mark

4.2k citations

33 papers · 3.6k indexed · 1 hit paper · h-index 24

Impact in

Physiology top 1%
- Alzheimer's disease research and treatments
- Biochemical effects in animals
Neurology top 2%
- Neuroinflammation and Neurodegeneration Mechanisms

Papers in ⓘ

Neurology 5
- Neurological Disease Mechanisms and Treatments 3
Physiology 15
- Alzheimer's disease research and treatments 12
- Biochemical effects in animals 4

Co-authors: Mark P. Mattson (18 shared papers)Kôji Uchida (3 shared papers)William R. Markesbery (2 shared papers)James G. Begley (3 shared papers)Mark A. Lovell (1 shared paper)Emmanuelle Blanc (4 shared papers)Steven W. Barger (2 shared papers)Katsutoshi Furukawa (4 shared papers)
Journals: Journal of Neurochemistry (9 papers)Journal of Neuroscience (4 papers)Brain Research (4 papers)The Journal of Cell Biology (1 paper)Journal of Pharmacology and Experimental Therapeutics (1 paper)
Partner nations: United States Japan Austria

In The Last Decade

Robert J. Mark

32 papers receiving 3.5k citations

Hit Papers

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A Role for 4‐Hydroxynonenal, an Aldehydic Product of Lipid Peroxidation, in Disruption of Ion Homeostasis and Neuronal Death Induced by Amyloid β‐Peptide 1997 · 647 citations

Peers

Countries citing papers authored by Robert J. Mark

Since Specialization

Citations

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Fields of papers citing papers by Robert J. Mark

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Robert J. Mark. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Robert J. Mark. The network helps show where Robert J. Mark may publish in the future.

Co-authors

The 25 scholars most cited alongside Robert J. Mark, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Robert J. Mark Line = papers co-authored together Robert J. Mark links everyone, so they are left out of the graph.

All Works

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20 of 20 papers shown

Showing the 20 most-cited of 33 papers — load more, or switch the sort, to bring in the rest.

#	Work
1	A Role for 4‐Hydroxynonenal, an Aldehydic Product of Lipid Peroxidation, in Disruption of Ion Homeostasis and Neuronal Death Induced by Amyloid β‐Peptide Journal of Neurochemistry ·Robert J. Mark, Mark A. Lovell, William R. Markesbery, Kôji Uchida, Mark P. Mattson Hit paper breakdown →	1997	647
2	Amyloid β-Peptide Impairs Glucose Transport in Hippocampal and Cortical Neurons: Involvement of Membrane Lipid Peroxidation Journal of Neuroscience ·Robert J. Mark, Zhen Pang, James W. Geddes, Kôji Uchida, Mark P. Mattson	1997	462
3	Chapter 10 Calcium, Free Radicals, and Excitotoxic Neuronal Death in Primary Cell Culture Methods in cell biology ·Mark P. Mattson, Steven W. Barger, James G. Begley, Robert J. Mark	1995	309
4	4-Hydroxynonenal, an aldehydic product of membrane lipid peroxidation, impairs glutamate transport and mitochondrial function in synaptosomes Neuroscience ·Jeffrey N. Keller, Robert J. Mark, Annadora J. Bruce, Emmanuelle Blanc, J. D. Rothstein, Kôji Uchida, G. Waeg, Mark P. Mattson	1997	286
5	Cellular signaling roles of TGFβ, TNFα and βAPP in brain injury responses and Alzheimer's disease Brain Research Reviews ·Mark P. Mattson, Steven W. Barger, Katsutoshi Furukawa, Annadora J. Bruce, Tony Wyss‐Coray, Robert J. Mark, Lennart Mucke	1997	214
6	4‐Hydroxynonenal, a Lipid Peroxidation Product, Rapidly Accumulates Following Traumatic Spinal Cord Injury and Inhibits Glutamate Uptake Journal of Neurochemistry ·Joe E. Springer, Robert D. Azbill, Robert J. Mark, James G. Begley, Georg Waeg, Mark P. Mattson	1997	155
7	Amyloid β‐Peptide Induces Cell Monolayer Albumin Permeability, Impairs Glucose Transport, and Induces Apoptosis in Vascular Endothelial Cells Journal of Neurochemistry ·Emmanuelle Blanc, Michał Toborek, Robert J. Mark, Bernhard Hennig, Mark P. Mattson	1997	148
8	Basic FGF attenuates amyloid β-peptide-induced oxidative stress, mitochondrial dysfunction, and impairment of Na+/K+-ATPase activity in hippocampal neurons Brain Research ·Robert J. Mark, Jeffrey N. Keller, Inna I. Kruman, Mark P. Mattson	1997	141
9	Aβ25–35 induces rapid lysis of red blood cells: contrast with Aβ1–42 and examination of underlying mechanisms Brain Research ·Mark P. Mattson, James G. Begley, Robert J. Mark, Katsutoshi Furukawa	1997	119
10	Amyloid β-peptide and oxidative cellular injury in Alzheimer’s disease Molecular Neurobiology ·Robert J. Mark, Emmanuelle Blanc, Mark P. Mattson	1996	119
11	4‐Hydroxynonenal, an Aldehydic Product of Lipid Peroxidation, Impairs Signal Transduction Associated with Muscarinic Acetylcholine and Metabotropic Glutamate Receptors: Possible Action on Gα_q/11 Journal of Neurochemistry ·Emmanuelle Blanc, Jeremiah F. Kelly, Robert J. Mark, Georg Waeg, Mark P. Mattson	1997	115
12	Neuroprotective Action of Cycloheximide Involves Induction of Bcl-2 and Antioxidant Pathways The Journal of Cell Biology ·Katsutoshi Furukawa, Steven Estus, Weiming Fu, Robert J. Mark, Mark P. Mattson	1997	109
13	Reactive Oxygen Species as Causal Agents in the Neurotoxicity of the Alzheimer's Disease‐Associated Amyloid Beta Peptidea Annals of the New York Academy of Sciences ·K. Hensley, D. A. BUTTERFIELDLD, Nathan C. Hall, Paige L. Cole, Ram Subramaniam, Robert J. Mark, Mark P. Mattson, William R. Markesbery, Marni E. Harris, Michael Y. Aksenov, Marina Aksenova, Jiin‐Sheng Wu, John M. Carney	1996	99
14	Anticonvulsants attenuate amyloid β-peptide neurotoxicity, Ca2+ deregulation, and cytoskeletal pathology Neurobiology of Aging ·Robert J. Mark, J. Wesson Ashford, Yadong Goodman, Mark P. Mattson	1995	97
15	Lysophosphatidic Acid Induces Necrosis and Apoptosis in Hippocampal Neurons Journal of Neurochemistry ·Frederick W. Holtsberg, Marion R. Steiner, Jeffrey N. Keller, Robert J. Mark, Mark P. Mattson, Sheldon M. Steiner	1998	93
16	Characterization of the WAVE1 Knock-Out Mouse: Implications for CNS Development Journal of Neuroscience ·John P. Dahl, Jeanne Wang-Dunlop, Cathleen Gonzales, Mary E.P. Goad, Robert J. Mark, Seung Kwak	2003	83
17	Heparin-Binding Epidermal Growth Factor-Like Growth Factor in Hippocampus: Modulation of Expression by Seizures and Anti-Excitotoxic Action Journal of Neuroscience ·Lisa A. Opanashuk, Robert J. Mark, Julie A. Porter, Deborah Damm, Mark P. Mattson, Kim B. Seroogy	1999	63
18	Pancortin-2 Interacts with WAVE1 and Bcl-xL in a Mitochondria-Associated Protein Complex That Mediates Ischemic Neuronal Death Journal of Neuroscience ·Aiwu Cheng, Thiruma V. Arumugam, Dong Liu, Rina Khatri, Khadija Mustafa, Seung Kwak, Huai-Ping Ling, Cathleen Gonzales, Xin Ouyang, Dong‐Gyu Jo, Zhihong Guo, Robert J. Mark, Mark P. Mattson	2007	46
19	Disruption of Brain Cell Ion Homeostasis in Alzheimer's Disease by Oxy Radicals, and Signaling Pathways That Protect Therefrom Chemical Research in Toxicology ·Mark P. Mattson, Robert J. Mark, Katsutoshi Furukawa, Annadora J. Bruce	1997	40
20	Characterization of 8‐Epiprostaglandin F_2α as aMarker of Amyloid β‐Peptide‐Induced Oxidative Damage Journal of Neurochemistry ·Robert J. Mark, Kimberly S. Fuson, Patrick C. May	1999	39

About Robert J. Mark

Robert J. Mark is a scholar working on Neurology, Physiology, Biochemistry, Cellular and Molecular Neuroscience and Biochemistry, having authored 33 papers that have together received 3.6k indexed citations. Recurring topics across this work include Alzheimer's disease research and treatments (12 papers), Neuroscience and Neuropharmacology Research (5 papers), Biochemical effects in animals (4 papers), Mitochondrial Function and Pathology (4 papers), Neurological Disease Mechanisms and Treatments (3 papers), Cholinesterase and Neurodegenerative Diseases (3 papers), Receptor Mechanisms and Signaling (2 papers) and S100 Proteins and Annexins (2 papers). The work is most often cited by research in Physiology (1.6k citations), Neurology (506 citations), Biological Psychiatry (136 citations), Cellular and Molecular Neuroscience (857 citations) and Developmental Neuroscience (186 citations). Robert J. Mark has collaborated with scholars based in United States, Japan and Austria. Frequent co-authors include Mark P. Mattson, Kôji Uchida, William R. Markesbery, James G. Begley, Mark A. Lovell, Emmanuelle Blanc, Steven W. Barger, Katsutoshi Furukawa, Zhen Pang and James W. Geddes. Their work appears in journals such as Journal of Neurochemistry, Journal of Neuroscience, Brain Research, The Journal of Cell Biology and Journal of Pharmacology and Experimental Therapeutics.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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