Robert J. Mark

4.2k total citations · 1 hit paper
33 papers, 3.6k citations indexed

About

Robert J. Mark is a scholar working on Physiology, Molecular Biology and Cellular and Molecular Neuroscience. According to data from OpenAlex, Robert J. Mark has authored 33 papers receiving a total of 3.6k indexed citations (citations by other indexed papers that have themselves been cited), including 15 papers in Physiology, 13 papers in Molecular Biology and 7 papers in Cellular and Molecular Neuroscience. Recurrent topics in Robert J. Mark's work include Alzheimer's disease research and treatments (12 papers), Neuroscience and Neuropharmacology Research (5 papers) and Mitochondrial Function and Pathology (4 papers). Robert J. Mark is often cited by papers focused on Alzheimer's disease research and treatments (12 papers), Neuroscience and Neuropharmacology Research (5 papers) and Mitochondrial Function and Pathology (4 papers). Robert J. Mark collaborates with scholars based in United States, Japan and Austria. Robert J. Mark's co-authors include Mark P. Mattson, Kôji Uchida, William R. Markesbery, James G. Begley, Mark A. Lovell, Emmanuelle Blanc, Steven W. Barger, James W. Geddes, Zhen Pang and Katsutoshi Furukawa and has published in prestigious journals such as Journal of Neuroscience, The Journal of Cell Biology and Molecular and Cellular Biology.

In The Last Decade

Robert J. Mark

32 papers receiving 3.5k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

A Role for 4‐Hydroxynonenal, an Aldehydic Product of Lipid Peroxidation, in Disruption of Ion Homeostasis and Neuronal Death Induced by Amyloid β‐Peptide

1997 647 citations Robert J. Mark, Mark A. Lovell et al. Journal of Neurochemistry profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Robert J. Mark United States	24	1.6k	1.6k	857	506	412	33	3.6k
K. Hensley United States	16	1.3k 0.8×	1.8k 1.1×	570 0.7×	477 0.9×	567 1.4×	17	3.3k
H. Fai Poon United States	28	2.2k 1.3×	2.0k 1.2×	642 0.7×	562 1.1×	499 1.2×	43	4.1k
Ward A. Pedersen United States	25	2.1k 1.3×	2.0k 1.2×	778 0.9×	587 1.2×	501 1.2×	37	4.6k
Elena Tamagno Italy	34	1.3k 0.8×	1.8k 1.1×	447 0.5×	427 0.8×	642 1.6×	64	3.7k
Usha MacGarvey United States	15	2.2k 1.4×	1.1k 0.7×	1.1k 1.3×	362 0.7×	297 0.7×	16	3.9k
S. Prasad Gabbita United States	18	1.7k 1.0×	1.3k 0.8×	272 0.3×	376 0.7×	339 0.8×	21	3.5k
Wan Sung Choi South Korea	37	2.2k 1.3×	1.0k 0.6×	886 1.0×	646 1.3×	405 1.0×	163	4.9k
Joanna B. Strosznajder Poland	36	2.4k 1.5×	1.9k 1.2×	1.0k 1.2×	721 1.4×	566 1.4×	165	4.8k
James G. Begley United States	15	1.2k 0.8×	1.3k 0.8×	772 0.9×	284 0.6×	303 0.7×	19	2.6k

Countries citing papers authored by Robert J. Mark

Since Specialization

Citations

This map shows the geographic impact of Robert J. Mark's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Robert J. Mark with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Robert J. Mark more than expected).

Fields of papers citing papers by Robert J. Mark

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Robert J. Mark. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Robert J. Mark. The network helps show where Robert J. Mark may publish in the future.

Co-authorship network of co-authors of Robert J. Mark

This figure shows the co-authorship network connecting the top 25 collaborators of Robert J. Mark. A scholar is included among the top collaborators of Robert J. Mark based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Robert J. Mark. Robert J. Mark is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Wang, Weijia, Vladimir Turzhitsky, Robert J. Mark, et al.. (2024). Economic Impact of Postoperative Urinary Retention in the US Hospital Setting. Journal of health economics and outcomes research. 29–34. 2 indexed citations

Bash, Lori D., Vladimir Turzhitsky, Robert J. Mark, Ira Hofer, & Toby N. Weingarten. (2023). Post-operative urinary retention is impacted by neuromuscular block reversal agent choice: A retrospective cohort study in US hospital setting. Journal of Clinical Anesthesia. 93. 111344–111344. 8 indexed citations

Parmentier‐Batteur, Sophie, Hemaka A. Rajapakse, John M. Sanders, et al.. (2011). Attenuation of scratch‐induced reactive astrogliosis by novel EphA4 kinase inhibitors. Journal of Neurochemistry. 118(6). 1016–1031. 25 indexed citations

Beyer, Chad E., Jason M. Dwyer, Brian J. Platt, et al.. (2010). Angiotensin IV elevates oxytocin levels in the rat amygdala and produces anxiolytic-like activity through subsequent oxytocin receptor activation. Psychopharmacology. 209(4). 303–311. 23 indexed citations

Pong, Kevin, Cathleen Gonzales, Yi Chen, et al.. (2009). Neuroprotective Profile of Novel Src Kinase Inhibitors in Rodent Models of Cerebral Ischemia. Journal of Pharmacology and Experimental Therapeutics. 331(3). 827–835. 32 indexed citations

Cheng, Aiwu, Thiruma V. Arumugam, Dong Liu, et al.. (2007). Pancortin-2 Interacts with WAVE1 and Bcl-xL in a Mitochondria-Associated Protein Complex That Mediates Ischemic Neuronal Death. Journal of Neuroscience. 27(7). 1519–1528. 46 indexed citations

Wood, Michael W., et al.. (2000). Inflammatory Cytokines Enhance Muscarinic‐Mediated Arachidonic Acid Release Through p38 Mitogen‐Activated Protein Kinase in A2058 Cells. Journal of Neurochemistry. 74(5). 2033–2040. 8 indexed citations

Mark, Robert J., et al.. (1999). Fibroblast growth factor-8 protects cultured rat hippocampal neurons from oxidative insult. Brain Research. 830(1). 88–93. 22 indexed citations

Mark, Robert J., Kimberly S. Fuson, & Patrick C. May. (1999). Characterization of 8‐Epiprostaglandin F_2α as aMarker of Amyloid β‐Peptide‐Induced Oxidative Damage. Journal of Neurochemistry. 72(3). 1146–1153. 39 indexed citations

10.

Mark, Robert J.. (1999). Common mechanisms of oxidative damage in chronic neurodegenerative conditions: potential points of therapeutic intervention. Expert Opinion on Therapeutic Patents. 9(10). 1339–1346. 4 indexed citations

11.

Holtsberg, Frederick W., Marion R. Steiner, Jeffrey N. Keller, et al.. (1998). Lysophosphatidic Acid Induces Necrosis and Apoptosis in Hippocampal Neurons. Journal of Neurochemistry. 70(1). 66–76. 93 indexed citations

12.

Mark, Robert J., Jeffrey N. Keller, Inna I. Kruman, & Mark P. Mattson. (1997). Basic FGF attenuates amyloid β-peptide-induced oxidative stress, mitochondrial dysfunction, and impairment of Na+/K+-ATPase activity in hippocampal neurons. Brain Research. 756(1-2). 205–214. 141 indexed citations

13.

Keller, Jeffrey N., Robert J. Mark, Annadora J. Bruce, et al.. (1997). 4-Hydroxynonenal, an aldehydic product of membrane lipid peroxidation, impairs glutamate transport and mitochondrial function in synaptosomes. Neuroscience. 80(3). 685–696. 286 indexed citations

14.

Mattson, Mark P., Steven W. Barger, Katsutoshi Furukawa, et al.. (1997). Cellular signaling roles of TGFβ, TNFα and βAPP in brain injury responses and Alzheimer's disease. Brain Research Reviews. 23(1-2). 47–61. 214 indexed citations

15.

Mark, Robert J., Mark A. Lovell, William R. Markesbery, Kôji Uchida, & Mark P. Mattson. (1997). A Role for 4‐Hydroxynonenal, an Aldehydic Product of Lipid Peroxidation, in Disruption of Ion Homeostasis and Neuronal Death Induced by Amyloid β‐Peptide. Journal of Neurochemistry. 68(1). 255–264. 647 indexed citations breakdown →

16.

Mattson, Mark P., James G. Begley, Robert J. Mark, & Katsutoshi Furukawa. (1997). Aβ25–35 induces rapid lysis of red blood cells: contrast with Aβ1–42 and examination of underlying mechanisms. Brain Research. 771(1). 147–153. 119 indexed citations

17.

Springer, Joe E., Robert D. Azbill, Robert J. Mark, et al.. (1997). 4‐Hydroxynonenal, a Lipid Peroxidation Product, Rapidly Accumulates Following Traumatic Spinal Cord Injury and Inhibits Glutamate Uptake. Journal of Neurochemistry. 68(6). 2469–2476. 155 indexed citations

18.

Hensley, K., Nathan C. Hall, Ram Subramaniam, et al.. (1996). Reactive Oxygen Species as Causal Agents in the Neurotoxicity of the Alzheimer's Disease‐Associated Amyloid Beta Peptidea. Annals of the New York Academy of Sciences. 786(1). 120–134. 99 indexed citations

19.

Mark, Robert J., Emmanuelle Blanc, & Mark P. Mattson. (1996). Amyloid β-peptide and oxidative cellular injury in Alzheimer’s disease. Molecular Neurobiology. 12(3). 211–224. 119 indexed citations

20.

Mark, Robert J., J. Wesson Ashford, Yadong Goodman, & Mark P. Mattson. (1995). Anticonvulsants attenuate amyloid β-peptide neurotoxicity, Ca2+ deregulation, and cytoskeletal pathology. Neurobiology of Aging. 16(2). 187–198. 97 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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