Robert H. Schloemer

1.1k total citations
27 papers, 879 citations indexed

About

Robert H. Schloemer is a scholar working on Molecular Biology, Epidemiology and Infectious Diseases. According to data from OpenAlex, Robert H. Schloemer has authored 27 papers receiving a total of 879 indexed citations (citations by other indexed papers that have themselves been cited), including 12 papers in Molecular Biology, 11 papers in Epidemiology and 7 papers in Infectious Diseases. Recurrent topics in Robert H. Schloemer's work include Hepatitis B Virus Studies (7 papers), Virus-based gene therapy research (6 papers) and Bacteriophages and microbial interactions (4 papers). Robert H. Schloemer is often cited by papers focused on Hepatitis B Virus Studies (7 papers), Virus-based gene therapy research (6 papers) and Bacteriophages and microbial interactions (4 papers). Robert H. Schloemer collaborates with scholars based in United States. Robert H. Schloemer's co-authors include J S Twu, R R Wagner, Reginald H. Garrett, Charles E. Wilde, Robert R. Wagner, C H Lee, Alyssa M. Kaiser, Fred Heffron, Chao‐Hung Lee and Karen K. Gerlach and has published in prestigious journals such as Proceedings of the National Academy of Sciences, PLANT PHYSIOLOGY and Journal of Virology.

In The Last Decade

Robert H. Schloemer

27 papers receiving 783 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Robert H. Schloemer United States 15 503 293 205 165 145 27 879
Robert Z. Maigetter United States 13 348 0.7× 397 1.4× 117 0.6× 135 0.8× 201 1.4× 26 977
Nobuya Ohtomo Japan 10 498 1.0× 324 1.1× 198 1.0× 128 0.8× 195 1.3× 26 859
Poul Andersson Denmark 10 297 0.6× 322 1.1× 256 1.2× 106 0.6× 58 0.4× 13 691
Burton Beames United States 16 515 1.0× 431 1.5× 468 2.3× 76 0.5× 74 0.5× 17 1.1k
Heinrich G. Köchel Germany 17 565 1.1× 478 1.6× 124 0.6× 80 0.5× 125 0.9× 23 1.1k
Biswendu B. Goswami United States 15 180 0.4× 201 0.7× 96 0.5× 84 0.5× 90 0.6× 32 603
Takashi Shimoike Japan 17 362 0.7× 391 1.3× 449 2.2× 222 1.3× 100 0.7× 24 1.1k
G. Kaluza Germany 13 227 0.5× 231 0.8× 45 0.2× 130 0.8× 85 0.6× 22 677
Byung Yoon Ahn South Korea 14 276 0.5× 212 0.7× 105 0.5× 113 0.7× 105 0.7× 18 713
Véronique Lambert France 15 210 0.4× 103 0.4× 118 0.6× 74 0.4× 33 0.2× 23 464

Countries citing papers authored by Robert H. Schloemer

Since Specialization
Citations

This map shows the geographic impact of Robert H. Schloemer's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Robert H. Schloemer with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Robert H. Schloemer more than expected).

Fields of papers citing papers by Robert H. Schloemer

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Robert H. Schloemer. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Robert H. Schloemer. The network helps show where Robert H. Schloemer may publish in the future.

Co-authorship network of co-authors of Robert H. Schloemer

This figure shows the co-authorship network connecting the top 25 collaborators of Robert H. Schloemer. A scholar is included among the top collaborators of Robert H. Schloemer based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Robert H. Schloemer. Robert H. Schloemer is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Hobbs, Jacqueline A., et al.. (2001). Caspase-3-like proteases are activated by infection but are not required for replication of vesicular stomatitis virus. Virus Research. 80(1-2). 53–65. 17 indexed citations
2.
Goebel, W. Scott, Robert H. Schloemer, & Zacharie Brahmi. (1996). Target cell-induced perforin mRNA turnover in NK3.3 cells is mediated by multiple elements within the mRNA coding region. Molecular Immunology. 33(4-5). 341–349. 2 indexed citations
3.
Gerlach, Karen K. & Robert H. Schloemer. (1992). Hepatitis B virus C gene promoter is under negative regulation. Virology. 189(1). 59–66. 14 indexed citations
4.
Schloemer, Robert H., et al.. (1991). An internal domain of the hepatitis B virus X antigen is necessary for transactivating activity. Virology. 182(2). 841–845. 35 indexed citations
5.
Schloemer, Robert H., et al.. (1991). Identification of the hepatitis B virus factor that inhibits expression of the beta interferon gene. Journal of Virology. 65(9). 4699–4704. 63 indexed citations
6.
Schloemer, Robert H., et al.. (1989). Accumulation of chlamydial lipopolysaccharide antigen in the plasma membranes of infected cells. Infection and Immunity. 57(6). 1780–1785. 49 indexed citations
7.
Schloemer, Robert H., et al.. (1988). Identification and characterization of Clostridium difficile promoter element that is functional in Escherichia coli. Gene. 70(2). 343–350. 5 indexed citations
8.
Schloemer, Robert H., et al.. (1988). Cloning and expression of secreted antigens of Clostridium difficile in Escherichia coli. Infection and Immunity. 56(6). 1655–1657. 3 indexed citations
9.
Twu, J S, et al.. (1988). Hepatitis B virus suppresses expression of human beta-interferon.. Proceedings of the National Academy of Sciences. 85(1). 252–256. 54 indexed citations
10.
Kaiser, Alyssa M., et al.. (1987). Factors influencing the phagocytosis of Clostridium difficile by human polymorphonuclear leukocytes. Infection and Immunity. 55(7). 1541–1546. 29 indexed citations
11.
Twu, J S & Robert H. Schloemer. (1987). Transcriptional trans-activating function of hepatitis B virus. Journal of Virology. 61(11). 3448–3453. 235 indexed citations
12.
Schloemer, Robert H., et al.. (1986). Inhibition of Banzi Viral RNA Synthesis in BHK-21 Cells by Banzi Virion Matrix Protein. Intervirology. 25(1). 38–47. 1 indexed citations
13.
Heffron, Fred, et al.. (1986). Analysis of Tn3 sequences required for transposition and immunity. Gene. 41(1). 23–31. 35 indexed citations
15.
Schloemer, Robert H., et al.. (1976). Sialylatin of glycoproteins of murine mammary tumor virus, murine leukemia virus, and Mason-Pfizer monkey virus. Journal of Virology. 18(2). 804–808. 14 indexed citations
16.
Schloemer, Robert H. & R R Wagner. (1975). Association of vesicular stomatitis virus glycoprotein with virion membrane: characterization of the lipophilic tail fragment. Journal of Virology. 16(2). 237–240. 70 indexed citations
17.
Schloemer, Robert H. & R R Wagner. (1975). Mosquito cells infected with vesicular stomatitis virus yield unsialylated virions of low infectivity. Journal of Virology. 15(4). 1029–1032. 34 indexed citations
18.
Schloemer, Robert H. & Robert R. Wagner. (1974). Sialoglycoprotein of Vesicular Stomatitis Virus: Role of the Neuraminic Acid in Infection. Journal of Virology. 14(2). 270–281. 32 indexed citations
19.
Schloemer, Robert H. & Reginald H. Garrett. (1974). Nitrate Transport System in Neurospora crassa. Journal of Bacteriology. 118(1). 259–269. 63 indexed citations
20.
Schloemer, Robert H. & Reginald H. Garrett. (1973). Partial Purification of the NADH-Nitrate Reductase Complex from Chlorella pyrenoidosa. PLANT PHYSIOLOGY. 51(3). 591–593. 9 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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