Robert G. Liteplo

888 total citations
36 papers, 631 citations indexed

About

Robert G. Liteplo is a scholar working on Molecular Biology, Cancer Research and Health, Toxicology and Mutagenesis. According to data from OpenAlex, Robert G. Liteplo has authored 36 papers receiving a total of 631 indexed citations (citations by other indexed papers that have themselves been cited), including 18 papers in Molecular Biology, 14 papers in Cancer Research and 8 papers in Health, Toxicology and Mutagenesis. Recurrent topics in Robert G. Liteplo's work include Epigenetics and DNA Methylation (10 papers), Carcinogens and Genotoxicity Assessment (8 papers) and Cancer Research and Treatments (7 papers). Robert G. Liteplo is often cited by papers focused on Epigenetics and DNA Methylation (10 papers), Carcinogens and Genotoxicity Assessment (8 papers) and Cancer Research and Treatments (7 papers). Robert G. Liteplo collaborates with scholars based in Canada and United States. Robert G. Liteplo's co-authors include M.E. Meek, Robert S. Kerbel, Philip Frost, R. Newhook, B E Elliott, Douglas A. Carlow, T. P. Donaghue, Michael Sribney, David S. Rosenblatt and P.K.L. Chan and has published in prestigious journals such as The Journal of Experimental Medicine, The Journal of Immunology and Biochemical and Biophysical Research Communications.

In The Last Decade

Robert G. Liteplo

34 papers receiving 581 citations

Peers

Robert G. Liteplo
Comparison fields: 5 of 111
  • Molecular Biology 259
  • Cancer Research 192
  • Health, Toxicology and Mutagenesis 153
  • Oncology 113
  • Immunology 64
Replace Rong Yao with:
Rong Yao China
Seung-Hee Chang South Korea
Hélène Stridh Sweden
Zhongtian Bai China
Hua Fung United States
Eric R. Gauthier Canada
Michael V. Templin United States
Lorraine Tilbury United States
Yi Qi China
Rong Yao China View profile →
Citations per field, relative to Robert G. Liteplo
Robert G. Liteplo · 1×
Citations per year, relative to Robert G. Liteplo
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Countries citing papers authored by Robert G. Liteplo

Since Specialization
Citations

This map shows the geographic impact of Robert G. Liteplo's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Robert G. Liteplo with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Robert G. Liteplo more than expected).

Fields of papers citing papers by Robert G. Liteplo

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Robert G. Liteplo. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Robert G. Liteplo. The network helps show where Robert G. Liteplo may publish in the future.

Co-authorship network of co-authors of Robert G. Liteplo

This figure shows the co-authorship network connecting the top 25 collaborators of Robert G. Liteplo. A scholar is included among the top collaborators of Robert G. Liteplo based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Robert G. Liteplo. Robert G. Liteplo is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
# Work Indexed citations
1 13
2 81
3
Ethylene glycol : human health aspects
16
4 13
5 26
6 75
7 2
8 32
9 2
10 6
11 3
12 12
13 5
14 19
15 3
16
Periodate-oxidized adenosine induction of murine thymidine kinase: role of DNA methylation in the generation of tumor cell heterogeneity.
11
17
Induction of thymidine kinase activity in a spontaneously enzyme-deficient murine tumor cell line by exposure in vivo to the DNA-hypomethylating agent 5-aza-2'-deoxycytidine: implications for mechanisms of tumor progression.
7
18 71
19 103
20 5

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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