Robert E. Mrak

11.7k total citations · 3 hit papers
143 papers, 9.4k citations indexed

About

Robert E. Mrak is a scholar working on Physiology, Molecular Biology and Neurology. According to data from OpenAlex, Robert E. Mrak has authored 143 papers receiving a total of 9.4k indexed citations (citations by other indexed papers that have themselves been cited), including 55 papers in Physiology, 48 papers in Molecular Biology and 31 papers in Neurology. Recurrent topics in Robert E. Mrak's work include Alzheimer's disease research and treatments (46 papers), Neuroinflammation and Neurodegeneration Mechanisms (29 papers) and S100 Proteins and Annexins (20 papers). Robert E. Mrak is often cited by papers focused on Alzheimer's disease research and treatments (46 papers), Neuroinflammation and Neurodegeneration Mechanisms (29 papers) and S100 Proteins and Annexins (20 papers). Robert E. Mrak collaborates with scholars based in United States, United Kingdom and Japan. Robert E. Mrak's co-authors include W. Sue T. Griffin, Jin G. Sheng, Wolfgang J. Streit, David I. Graham, W. Sue T. Griffin, Craig N. Karson, Steven W. Barger, G.W. Roberts, J.E. McKenzie and M.C. Royston and has published in prestigious journals such as SHILAP Revista de lepidopterología, Neurology and The Journal of Comparative Neurology.

In The Last Decade

Robert E. Mrak

140 papers receiving 9.1k citations

Hit Papers

Microglia and neuroinflammation: a pathological perspective. 1998 2026 2007 2016 2004 1998 2004 250 500 750

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Robert E. Mrak United States 51 3.8k 3.4k 3.1k 1.4k 1.4k 143 9.4k
M. Kerry O’Banion United States 49 3.6k 0.9× 4.5k 1.3× 3.2k 1.0× 1.6k 1.2× 969 0.7× 146 11.8k
Douglas L. Feinstein United States 62 3.5k 0.9× 3.3k 1.0× 4.8k 1.5× 2.5k 1.8× 797 0.6× 211 12.2k
Bruce T. Lamb United States 53 5.6k 1.5× 4.6k 1.4× 3.6k 1.1× 1.6k 1.1× 809 0.6× 130 11.4k
Markus Schwaninger Germany 60 1.8k 0.5× 3.4k 1.0× 4.1k 1.3× 1.7k 1.2× 1.1k 0.8× 217 12.3k
Douglas G. Walker United States 64 6.4k 1.7× 5.1k 1.5× 3.9k 1.2× 2.0k 1.4× 2.9k 2.1× 173 13.7k
Ignacio Lizasoaín Spain 60 2.8k 0.7× 3.6k 1.1× 3.8k 1.2× 1.9k 1.3× 959 0.7× 208 12.4k
John Denis Fryer United States 49 2.9k 0.8× 1.8k 0.5× 3.4k 1.1× 1.4k 1.0× 878 0.6× 133 7.7k
Marı́a A. Moro Spain 63 2.5k 0.7× 3.5k 1.0× 3.9k 1.3× 1.9k 1.4× 861 0.6× 202 12.0k
W. Sue T. Griffin United States 59 6.5k 1.7× 5.9k 1.8× 5.2k 1.7× 2.1k 1.5× 1.8k 1.3× 126 13.8k
Abhay P. Sagare United States 46 7.0k 1.8× 6.7k 2.0× 4.7k 1.5× 1.9k 1.4× 2.5k 1.8× 78 16.0k

Countries citing papers authored by Robert E. Mrak

Since Specialization
Citations

This map shows the geographic impact of Robert E. Mrak's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Robert E. Mrak with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Robert E. Mrak more than expected).

Fields of papers citing papers by Robert E. Mrak

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Robert E. Mrak. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Robert E. Mrak. The network helps show where Robert E. Mrak may publish in the future.

Co-authorship network of co-authors of Robert E. Mrak

This figure shows the co-authorship network connecting the top 25 collaborators of Robert E. Mrak. A scholar is included among the top collaborators of Robert E. Mrak based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Robert E. Mrak. Robert E. Mrak is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Aboud, Orwa, Robert E. Mrak, Frederick A. Boop, & W. Sue T. Griffin. (2013). Epilepsy: neuroinflammation, neurodegeneration, and APOE genotype. Acta Neuropathologica Communications. 1(1). 41–41. 31 indexed citations
2.
Marchetti, Philippe, et al.. (2007). Neurotrophin receptors and heparanase: a functional axis in human medulloblastoma invasion.. PubMed. 26(1). 5–23. 7 indexed citations
3.
Griffin, W. Sue T., et al.. (2006). Interleukin-1 mediates Alzheimer and Lewy body pathologies. Journal of Neuroinflammation. 3(1). 5–5. 207 indexed citations
4.
Romanovsky, Dmitry, Amy E. Moseley, Robert E. Mrak, Michael D. Taylor, & Maxim Dobretsov. (2006). Phylogenetic preservation of α3 Na+,K+‐ATPase distribution in vertebrate peripheral nervous systems. The Journal of Comparative Neurology. 500(6). 1106–1116. 21 indexed citations
5.
Sinnappah-Kang, Neeta Devi, et al.. (2005). Heparanase, TrkC and p75NTR: their functional involvement in human medulloblastoma cell invasion.. PubMed. 27(3). 617–26. 15 indexed citations
6.
Mrak, Robert E. & W. Sue T. Griffin. (2004). Welcome to the Journal of Neuroinflammation!. Journal of Neuroinflammation. 1(1). 1–1. 50 indexed citations
7.
Sheng, Jin G., Robert E. Mrak, Kelly R. Bales, et al.. (2000). Overexpression of the Neuritotrophic Cytokine S100β Precedes the Appearance of Neuritic β‐Amyloid Plaques in APPV717F Mice. Journal of Neurochemistry. 74(1). 295–301. 67 indexed citations
8.
Mrak, Robert E. & W. Sue T. Griffin. (2000). Interleukin-1 and the Immunogenetics of Alzheimer Disease. Journal of Neuropathology & Experimental Neurology. 59(6). 471–476. 88 indexed citations
9.
Sheng, Jin G., et al.. (2000). Interleukin-1 Promotes Expression and Phosphorylation of Neurofilament and tau Proteins in Vivo. Experimental Neurology. 163(2). 388–391. 120 indexed citations
10.
Zhu, Shaojun, Jin G. Sheng, Richard A. Jones, et al.. (1999). Increased lnterleukin-1β Converting Enzyme Expression and Activity in Alzheimer Disease. Journal of Neuropathology & Experimental Neurology. 58(6). 582–587. 40 indexed citations
12.
Graham, David I., Stephen M. Gentleman, James A. R. Nicoll, et al.. (1999). Is There a Genetic Basis for the Deposition of β-Amyloid After Fatal Head Injury?. Cellular and Molecular Neurobiology. 19(1). 19–30. 20 indexed citations
13.
Sheng, Jin G., Robert E. Mrak, & W. Sue T. Griffin. (1998). Progressive Neuronal DNA Damage Associated with Neurofibrillary Tangle Formation in Alzheimer Disease. Journal of Neuropathology & Experimental Neurology. 57(4). 323–328. 44 indexed citations
14.
Sheng, Jin G., Robert E. Mrak, & W. Sue T. Griffin. (1995). Microglial inter leukin‐1α expression in brain regions in Alzheimer's disease: correlation with neuritic plaque distribution. Neuropathology and Applied Neurobiology. 21(4). 290–301. 149 indexed citations
15.
Stanley, Laura, Robert E. Mrak, Robert C. Woody, et al.. (1994). Glial Cytokines as Neuropathogenic Factors in HIV Infection. Journal of Neuropathology & Experimental Neurology. 53(3). 231–238. 122 indexed citations
16.
Griffin, W. Sue T., Jin G. Sheng, Stephen M. Gentleman, et al.. (1994). Microglial interleukin-lα expression in human head injury: Correlations with neuronal and neuritic β-amyloid precursor protein expression. Neuroscience Letters. 176(2). 133–136. 138 indexed citations
17.
Mrak, Robert E.. (1994). Malignant neurocytic tumor. Human Pathology. 25(8). 747–752. 45 indexed citations
18.
Mrak, Robert E., et al.. (1994). Rabies Encephalitis in Humans : Pathology, Pathogenesis and Pathophysiology. Journal of Neuropathology & Experimental Neurology. 53(1). 1–10. 37 indexed citations
19.
Mrak, Robert E. & Glen F. Baker. (1987). Granular cell basal cell carcinoma. Journal of Cutaneous Pathology. 14(1). 37–42. 44 indexed citations
20.
Mrak, Robert E.. (1985). Muscle membranes in diseases of muscle. CRC Press eBooks. 13 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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