Robert Clarke

64.9k citations
492 papers · 27.8k indexed · 3 hit papers · h-index 74

Robert Clarke

480 papers receiving 26.9k citations

Hit Papers

Autosis is a Na +,K + -ATPase–regulated form o...45320022026201020182.0k4.0k6.0k

Peers

Robert Clarke
Comparison fields: 5 of 209
  • Endocrinology 1.3k
  • Cardiology and Cardiovascular Medicine 5.3k
  • Cancer Research 2.6k
  • Oncology 4.5k
  • Nutrition and Dietetics 2.3k
Replace Greg S. Martin with:
Greg S. Martin United States
Timothy R. Billiar United States
Michael Bauer Germany
Xiaodong Wang China
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Robert Clarke relative to Greg S. Martin United States Greg S. Martin's profile →
Citations per field
00.5×3.8×
Greg S. Martin · 1×
Citations per year

Countries citing papers authored by Robert Clarke

Since Specialization
Citations

This map shows the geographic impact of Robert Clarke's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Robert Clarke with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Robert Clarke more than expected).

Fields of papers citing papers by Robert Clarke

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Robert Clarke. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Robert Clarke. The network helps show where Robert Clarke may publish in the future.

Co-authorship network

The 25 scholars most cited alongside Robert Clarke, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Robert Clarke Line = papers co-authored together Robert Clarke links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown
#Work
1 20250
2 202511
3 202419
4 20242
5 202218
6 20219
7 201915
8 201751
9 201688
10 201529
11 201547
12 2014170
13 201467
14 201411
15 201343
16 2012132
17 201015
18 201058
19 200891
20
Multiple Types of Programmed Cell Death and their Relevance to Perinatal Brain Damage
20082

About Robert Clarke

Robert Clarke is a scholar working on Endocrinology, Cancer Research and Molecular Biology, having authored 492 papers that have together received 27.8k indexed citations. Recurring topics across this work include Gene expression and cancer classification (83 papers), Estrogen and related hormone effects (82 papers), Bioinformatics and Genomic Networks (64 papers), Autophagy in Disease and Therapy (37 papers), Neuroscience and Neuropharmacology Research (35 papers), Gene Regulatory Network Analysis (35 papers), Endoplasmic Reticulum Stress and Disease (33 papers) and Retinal Development and Disorders (29 papers). The work is most often cited by research in Endocrinology (1.3k citations), Cardiology and Cardiovascular Medicine (5.3k citations) and Cancer Research (2.6k citations). Robert Clarke has collaborated with scholars based in United States, Switzerland and Canada. Frequent co-authors include Rory Collins, Nawab Qizilbash, Sarah Lewington, Richárd Pető, Leena Hilakivi‐Clarke, Fabio Leonessa, Julien Puyal, Bruce J. Trock, Ayesha N. Shajahan‐Haq and Katherine L. Cook. Their work appears in journals such as Cancer Research, Bioinformatics, The Journal of Comparative Neurology, Breast Cancer Research and Treatment and Endocrine Related Cancer.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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