Robert B. Elliott

532 total citations
12 papers, 396 citations indexed

About

Robert B. Elliott is a scholar working on Genetics, Surgery and Endocrinology, Diabetes and Metabolism. According to data from OpenAlex, Robert B. Elliott has authored 12 papers receiving a total of 396 indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Genetics, 9 papers in Surgery and 7 papers in Endocrinology, Diabetes and Metabolism. Recurrent topics in Robert B. Elliott's work include Diabetes and associated disorders (9 papers), Pancreatic function and diabetes (9 papers) and Diabetes Management and Research (5 papers). Robert B. Elliott is often cited by papers focused on Diabetes and associated disorders (9 papers), Pancreatic function and diabetes (9 papers) and Diabetes Management and Research (5 papers). Robert B. Elliott collaborates with scholars based in New Zealand, United States and Australia. Robert B. Elliott's co-authors include H. Peter Chase, C.C. Pilcher, Shiva Reddy, Liping Yu, George S. Eisenbarth, María J. Redondo, Satish K. Garg, Marian Rewers, David Wu and David M. Fergusson and has published in prestigious journals such as Diabetes, Annals of the New York Academy of Sciences and Diabetologia.

In The Last Decade

Robert B. Elliott

12 papers receiving 381 citations

Peers

Robert B. Elliott
R. Fußgänger Germany
Hans-Joachim Partke Germany
Yukikazu Kaino Japan
P Heyma Australia
Tanja Arndt Germany
Christiaan Van Schravendijk Belgium
Louise Ambye Denmark
Xunjun Xiao United States
Robert J. Leonard United States
Valérie Picard Canada
R. Fußgänger Germany
Robert B. Elliott
Citations per year, relative to Robert B. Elliott Robert B. Elliott (= 1×) peers R. Fußgänger

Countries citing papers authored by Robert B. Elliott

Since Specialization
Citations

This map shows the geographic impact of Robert B. Elliott's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Robert B. Elliott with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Robert B. Elliott more than expected).

Fields of papers citing papers by Robert B. Elliott

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Robert B. Elliott. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Robert B. Elliott. The network helps show where Robert B. Elliott may publish in the future.

Co-authorship network of co-authors of Robert B. Elliott

This figure shows the co-authorship network connecting the top 25 collaborators of Robert B. Elliott. A scholar is included among the top collaborators of Robert B. Elliott based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Robert B. Elliott. Robert B. Elliott is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

12 of 12 papers shown
1.
Rojas, M. Guadalupe, Robert B. Elliott, & Juan A. Morales-Ramos. (2018). Mortality of Solenopsis invicta Workers (Hymenoptera: Formicidae) After Indirect Exposure to Spores of Three Entomopathogenic Fungi. Journal of Insect Science. 18(3). 6 indexed citations
2.
3.
Reddy, Shiva, Robert B. Elliott, Mike Chang, et al.. (2008). Newly Weaned Nonobese Diabetic Mice Show Heightened Early Diabetes Sensitivity to Multiple Low Doses of Streptozotocin Than Nondiabetes-Prone CD-1 Mice. Pancreas. 37(1). e8–e19. 2 indexed citations
4.
Redondo, María J., Marian Rewers, Liping Yu, et al.. (1999). Genetic determination of islet cell autoimmunity in monozygotic twin, dizygotic twin, and non-twin siblings of patients with type 1 diabetes: prospective twin study. BMJ. 318(7185). 698–702. 106 indexed citations
5.
Reddy, Shiva, David Wu, & Robert B. Elliott. (1995). Low Dose Streptozotocin Causes Diabetes in Severe Combined Immunodeficient (Scid) Mice Without Immune Cell Infiltration of the Pancreatic Islets. Autoimmunity. 20(2). 83–92. 35 indexed citations
6.
7.
Leslie, Gerrie A., et al.. (1994). Perspectives in Diabetes Early Environmental Events as a Cause of IDDM Evidence and Implications. 6 indexed citations
8.
Elliott, Robert B., et al.. (1993). The Use of Nicotinamide in the Prevention of Type 1 Diabetes. Annals of the New York Academy of Sciences. 696(1). 333–341. 41 indexed citations
9.
Reddy, Shiva, et al.. (1992). First phase insulin release in the non-obese diabetic mouse: correlation with insulitis, beta cell number and autoantibodies. Diabetes Research and Clinical Practice. 17(1). 17–25. 16 indexed citations
10.
Elliott, Robert B. & H. Peter Chase. (1991). Prevention or delay of Type 1 (insulin-dependent) diabetes mellitus in children using nicotinamide. Diabetologia. 34(5). 362–365. 144 indexed citations
11.
Crossley, Jeanette R. & Robert B. Elliott. (1975). Insulin-like Insulinase-resistant Material, Distinguishable from Normal Insulin, in Juvenile Diabetes. Diabetes. 24(7). 609–617. 1 indexed citations
12.
Elliott, Robert B., et al.. (1954). Energy values for some deuteron reactions with 10B, 11B and 12C. Proceedings of the Royal Society of London A Mathematical and Physical Sciences. 224(1156). 129–133. 4 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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