Richard D. Press

5.0k total citations
105 papers, 2.8k citations indexed

About

Richard D. Press is a scholar working on Hematology, Genetics and Molecular Biology. According to data from OpenAlex, Richard D. Press has authored 105 papers receiving a total of 2.8k indexed citations (citations by other indexed papers that have themselves been cited), including 74 papers in Hematology, 53 papers in Genetics and 22 papers in Molecular Biology. Recurrent topics in Richard D. Press's work include Chronic Myeloid Leukemia Treatments (28 papers), Chronic Lymphocytic Leukemia Research (27 papers) and Acute Myeloid Leukemia Research (26 papers). Richard D. Press is often cited by papers focused on Chronic Myeloid Leukemia Treatments (28 papers), Chronic Lymphocytic Leukemia Research (27 papers) and Acute Myeloid Leukemia Research (26 papers). Richard D. Press collaborates with scholars based in United States, Canada and Germany. Richard D. Press's co-authors include Brian Druker, Michael W. Deininger, Michael J. Mauro, Jody L. Kujovich, Kenneth A. Bauer, John A. Heit, Fei Yang, Thomas G. DeLoughery, A. Evans and Stephanie G. Willis and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of the American Chemical Society and Circulation.

In The Last Decade

Richard D. Press

102 papers receiving 2.7k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Richard D. Press United States 31 1.9k 1.4k 794 550 211 105 2.8k
Elliott F. Winton United States 23 1.4k 0.7× 1.2k 0.9× 479 0.6× 841 1.5× 208 1.0× 81 2.5k
Peter C. Harpel United States 25 964 0.5× 382 0.3× 537 0.7× 599 1.1× 106 0.5× 46 2.8k
Anne Goodeve United Kingdom 36 3.6k 1.9× 917 0.7× 116 0.1× 1.2k 2.3× 253 1.2× 114 4.3k
Irene Lorand‐Metze Brazil 27 1.2k 0.6× 622 0.4× 184 0.2× 671 1.2× 291 1.4× 205 2.5k
H. Kronenberg Australia 20 832 0.4× 314 0.2× 386 0.5× 285 0.5× 128 0.6× 125 1.9k
H. Jean Khoury United States 30 1.9k 1.0× 620 0.4× 195 0.2× 538 1.0× 525 2.5× 117 3.1k
Paul Browne Ireland 26 1.3k 0.7× 956 0.7× 86 0.1× 858 1.6× 335 1.6× 68 3.7k
N L Esmon United States 26 2.7k 1.4× 582 0.4× 186 0.2× 645 1.2× 98 0.5× 38 3.9k
Daniela Cilloni Italy 33 2.7k 1.4× 1.6k 1.1× 676 0.9× 1.6k 2.9× 783 3.7× 181 4.4k
Dino Veneri Italy 30 1.0k 0.5× 458 0.3× 70 0.1× 232 0.4× 136 0.6× 93 2.2k

Countries citing papers authored by Richard D. Press

Since Specialization
Citations

This map shows the geographic impact of Richard D. Press's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Richard D. Press with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Richard D. Press more than expected).

Fields of papers citing papers by Richard D. Press

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Richard D. Press. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Richard D. Press. The network helps show where Richard D. Press may publish in the future.

Co-authorship network of co-authors of Richard D. Press

This figure shows the co-authorship network connecting the top 25 collaborators of Richard D. Press. A scholar is included among the top collaborators of Richard D. Press based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Richard D. Press. Richard D. Press is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Li, Yonghong, Fei Yang, Nicola Long, et al.. (2023). NGS-defined measurable residual disease (MRD) after initial chemotherapy as a prognostic biomarker for acute myeloid leukemia. Blood Cancer Journal. 13(1). 12 indexed citations
2.
Mohebnasab, Maedeh, Wei Xie, Philipp W. Raess, et al.. (2022). Distinguishing STAT3/STAT5B-mutated large granular lymphocyte leukemia from myeloid neoplasms by genetic profiling. Blood Advances. 7(1). 40–45. 4 indexed citations
3.
Shomali, William, Alisa Damnernsawad, David A. Sampson, et al.. (2021). A novel activating JAK1 mutation in chronic eosinophilic leukemia. Blood Advances. 5(18). 3581–3586. 10 indexed citations
4.
Yang, Fei, et al.. (2019). Clinical Utility of Next-Generation Sequencing in Acute Myeloid Leukemia. Molecular Diagnosis & Therapy. 24(1). 1–13. 33 indexed citations
5.
Spiciarich, David R., Stephen T. Oh, Michael J. Mauro, et al.. (2018). A Novel Germline Variant in CSF3R Reduces N-Glycosylation and Exerts Potent Oncogenic Effects in Leukemia. Cancer Research. 78(24). 6762–6770. 16 indexed citations
6.
7.
Press, Richard D., et al.. (2016). Measurement ofBCR-ABL1transcripts on the International Scale in the United States: current status and best practices. Leukemia & lymphoma. 58(1). 8–16. 13 indexed citations
8.
Zhang, Zhenzhen, Hyun-Jung Lee, Jennifer Dunlap, et al.. (2016). Clonal hematopoiesis as determined by the HUMARA assay is a marker for acquired mutations in epigenetic regulators in older women. Experimental Hematology. 44(9). 857–865.e5. 3 indexed citations
9.
Press, Richard D., et al.. (2013). BCR–ABL PCR testing in chronic myelogenous leukemia: molecular diagnosis for targeted cancer therapy and monitoring. Expert Review of Molecular Diagnostics. 13(7). 749–762. 20 indexed citations
10.
Jones, Dan, Suzanne Kamel‐Reid, David W. Bahler, et al.. (2008). Laboratory Practice Guidelines for Detecting and Reporting BCR-ABL Drug Resistance Mutations in Chronic Myelogenous Leukemia and Acute Lymphoblastic Leukemia. Journal of Molecular Diagnostics. 11(1). 4–11. 45 indexed citations
11.
Sherbenou, Daniel W., Oliver Hantschel, Ines Kaupe, et al.. (2008). Characterization of BCR-ABL deletion mutants from patients with chronic myeloid leukemia. Leukemia. 22(6). 1184–1190. 31 indexed citations
12.
Barton, James C., Ronald T. Acton, Catherine Leiendecker‐Foster, et al.. (2007). HFE C282Y Homozygotes Aged 25–29 Years at HEIRS Study Initial Screening. Genetic Testing. 11(3). 269–275. 6 indexed citations
13.
Rt, Acton, James C. Barton, P C Adams, et al.. (2007). A genome‐wide linkage scan for iron phenotype quantitative trait loci: the HEIRS Family Study. Clinical Genetics. 71(6). 518–529. 12 indexed citations
14.
Müller, Martin, Giuseppe Saglio, Feng Lin, et al.. (2007). An international study to standardize the detection and quantitation of BCR-ABL transcripts from stabilized peripheral blood preparations by quantitative RT-PCR. Haematologica. 92(7). 970–973. 33 indexed citations
15.
Rivers, Charles, James C. Barton, Victor R. Gordeuk, et al.. (2007). Association of ferroportin Q248H polymorphism with elevated levels of serum ferritin in African Americans in the Hemochromatosis and Iron Overload Screening (HEIRS) Study. Blood Cells Molecules and Diseases. 38(3). 247–252. 41 indexed citations
16.
17.
Press, Richard D., Nancy B. Beamer, A. Evans, Thomas G. DeLoughery, & Bruce M. Coull. (1999). Role of a Common Mutation in the Homocysteine Regulatory Enzyme Methylenetetrahydrofolate Reductase in Ischemic Stroke. Diagnostic Molecular Pathology. 8(1). 54–54. 26 indexed citations
18.
Press, Richard D.. (1999). Hemochromatosis: A “Simple” Genetic Trait. Hospital Practice. 34(8). 55–74. 5 indexed citations
19.
Press, Richard D., E. Premkumar Reddy, & Donald L. Ewert. (1994). Overexpression of C-Terminally but Not N-Terminally Truncated Myb Induces Fibrosarcomas: a Novel Nonhematopoietic Target cell for the myb Oncogene. Molecular and Cellular Biology. 14(4). 2278–2290. 5 indexed citations
20.
Press, Richard D., James W. Jacobberger, David Samols, & David A. Goldthwait. (1990). The cell cycle dependence of c‐sis gene expression: artifactual conclusions in cells prepared by chemical but not physical techniques. Cell Proliferation. 23(4). 299–312. 2 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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